Papiol, Sergi

[["dc.bibliographiccitation.journal","Molecular Psychiatry"],["dc.contributor.author","Amare, Azmeraw T."],["dc.contributor.author","Schubert, Klaus Oliver"],["dc.contributor.author","Hou, Liping"],["dc.contributor.author","Clark, Scott R."],["dc.contributor.author","Papiol, Sergi"],["dc.contributor.author","Cearns, Micah"],["dc.contributor.author","Heilbronner, Urs"],["dc.contributor.author","Degenhardt, Franziska"],["dc.contributor.author","Tekola-Ayele, Fasil"],["dc.contributor.author","Hsu, Yi-Hsiang"],["dc.contributor.author","Baune, Bernhard T."],["dc.date.accessioned","2020-12-10T18:09:37Z"],["dc.date.available","2020-12-10T18:09:37Z"],["dc.date.issued","2020"],["dc.identifier.doi","10.1038/s41380-020-0689-5"],["dc.identifier.eissn","1476-5578"],["dc.identifier.issn","1359-4184"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/73707"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","Association of polygenic score for major depression with response to lithium in patients with bipolar disorder"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","4076"],["dc.bibliographiccitation.issue","20"],["dc.bibliographiccitation.journal","Human Molecular Genetics"],["dc.bibliographiccitation.lastpage","4081"],["dc.bibliographiccitation.volume","20"],["dc.contributor.author","Steinberg, Stacy"],["dc.contributor.author","de Jong, Simone"],["dc.contributor.author","Andreassen, Ole A."],["dc.contributor.author","Werge, Thomas"],["dc.contributor.author","Børglum, Anders D."],["dc.contributor.author","Mors, Ole"],["dc.contributor.author","Mortensen, Preben B."],["dc.contributor.author","Gustafsson, Omar"],["dc.contributor.author","Costas, Javier"],["dc.contributor.author","Pietiläinen, Olli P. H."],["dc.contributor.author","Demontis, Ditte"],["dc.contributor.author","Papiol, Sergi"],["dc.contributor.author","Huttenlocher, Johanna"],["dc.contributor.author","Mattheisen, Manuel"],["dc.contributor.author","Breuer, René"],["dc.contributor.author","Vassos, Evangelos"],["dc.contributor.author","Giegling, Ina"],["dc.contributor.author","Fraser, Gillian"],["dc.contributor.author","Walker, Nicholas"],["dc.contributor.author","Tuulio-Henriksson, Annamari"],["dc.contributor.author","Suvisaari, Jaana"],["dc.contributor.author","Lönnqvist, Jouko"],["dc.contributor.author","Paunio, Tiina"],["dc.contributor.author","Agartz, Ingrid"],["dc.contributor.author","Melle, Ingrid"],["dc.contributor.author","Djurovic, Srdjan"],["dc.contributor.author","Strengman, Eric"],["dc.contributor.author","Jürgens, Gesche"],["dc.contributor.author","Glenthøj, Birte"],["dc.contributor.author","Terenius, Lars"],["dc.contributor.author","Hougaard, David M."],["dc.contributor.author","Ørntoft, Torben"],["dc.contributor.author","Wiuf, Carsten"],["dc.contributor.author","Didriksen, Michael"],["dc.contributor.author","Hollegaard, Mads V."],["dc.contributor.author","Nordentoft, Merete"],["dc.contributor.author","Winkel, Ruud van"],["dc.contributor.author","Kenis, Gunter"],["dc.contributor.author","Abramova, Lilia"],["dc.contributor.author","Kaleda, Vasily"],["dc.contributor.author","Arrojo, Manuel"],["dc.contributor.author","Sanjuán, Julio"],["dc.contributor.author","Arango, Celso"],["dc.contributor.author","Sperling, Swetlana"],["dc.contributor.author","Rossner, Moritz J."],["dc.contributor.author","Ribolsi, Michele"],["dc.contributor.author","Magni, Valentina"],["dc.contributor.author","Siracusano, Alberto"],["dc.contributor.author","Christiansen, Claus"],["dc.contributor.author","Kiemeney, Lambertus A."],["dc.contributor.author","Veldink, Jan"],["dc.contributor.author","Van den Berg, Leonard"],["dc.contributor.author","Ingason, Andres"],["dc.contributor.author","Muglia, Pierandrea"],["dc.contributor.author","Murray, Robin M."],["dc.contributor.author","Nöthen, Markus M."],["dc.contributor.author","Sigurdsson, Engilbert"],["dc.contributor.author","Petursson, Hannes"],["dc.contributor.author","Thorsteinsdottir, Unnur"],["dc.contributor.author","Kong, Augustine"],["dc.contributor.author","Rubino, I. Alex"],["dc.contributor.author","Hert, Marc de"],["dc.contributor.author","Réthelyi, János M."],["dc.contributor.author","Bitter, István"],["dc.contributor.author","Jönsson, Erik G."],["dc.contributor.author","Golimbet, Vera"],["dc.contributor.author","Carracedo, Angel"],["dc.contributor.author","Ehrenreich, Hannelore"],["dc.contributor.author","Craddock, Nick"],["dc.contributor.author","Owen, Michael J."],["dc.contributor.author","O’Donovan, Michael C."],["dc.contributor.author","Ruggeri, Mirella"],["dc.contributor.author","Tosato, Sarah"],["dc.contributor.author","Peltonen, Leena"],["dc.contributor.author","Ophoff, Roel A."],["dc.contributor.author","Collier, David A."],["dc.contributor.author","St Clair, David"],["dc.contributor.author","Rietschel, Marcella"],["dc.contributor.author","Cichon, Sven"],["dc.contributor.author","Stefansson, Hreinn"],["dc.contributor.author","Rujescu, Dan"],["dc.contributor.author","Stefansson, Kari"],["dc.date.accessioned","2017-09-07T11:46:19Z"],["dc.date.available","2017-09-07T11:46:19Z"],["dc.date.issued","2011"],["dc.description.abstract","Common sequence variants have recently joined rare structural polymorphisms as genetic factors with strong evidence for association with schizophrenia. Here we extend our previous genome-wide association study and meta-analysis (totalling 7 946 cases and 19 036 controls) by examining an expanded set of variants using an enlarged follow-up sample (up to 10 260 cases and 23 500 controls). In addition to previously reported alleles in the major histocompatibility complex region, near neurogranin (NRGN) and in an intron of transcription factor 4 (TCF4), we find two novel variants showing genome-wide significant association: rs2312147[C], upstream of vaccinia-related kinase 2 (VRK2) [odds ratio (OR) = 1.09, P = 1.9 × 10(-9)] and rs4309482[A], between coiled-coiled domain containing 68 (CCDC68) and TCF4, about 400 kb from the previously described risk allele, but not accounted for by its association (OR = 1.09, P = 7.8 × 10(-9))."],["dc.identifier.doi","10.1093/hmg/ddr325"],["dc.identifier.gro","3150487"],["dc.identifier.pmid","21791550"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/7257"],["dc.language.iso","en"],["dc.notes.status","final"],["dc.title","Common variants at VRK2 and TCF4 conferring risk of schizophrenia"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dc.type.peerReviewed","no"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","29"],["dc.bibliographiccitation.journal","Schizophrenia Research"],["dc.bibliographiccitation.lastpage","38"],["dc.bibliographiccitation.volume","244"],["dc.contributor.author","Schulte, Eva C."],["dc.contributor.author","Kondofersky, Ivan"],["dc.contributor.author","Budde, Monika"],["dc.contributor.author","Papiol, Sergi"],["dc.contributor.author","Senner, Fanny"],["dc.contributor.author","Schaupp, Sabrina K."],["dc.contributor.author","Reich-Erkelenz, Daniela"],["dc.contributor.author","Klöhn-Saghatolislam, Farahnaz"],["dc.contributor.author","Kalman, Janos L."],["dc.contributor.author","Gade, Katrin"],["dc.contributor.author","Schulze, Thomas G."],["dc.date.accessioned","2022-06-01T09:38:59Z"],["dc.date.available","2022-06-01T09:38:59Z"],["dc.date.issued","2022"],["dc.identifier.doi","10.1016/j.schres.2022.05.001"],["dc.identifier.pii","S0920996422001633"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/108361"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-572"],["dc.relation.issn","0920-9964"],["dc.rights.uri","https://www.elsevier.com/tdm/userlicense/1.0/"],["dc.title","A novel longitudinal clustering approach to psychopathology across diagnostic entities in the hospital-based PsyCourse study"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","S968"],["dc.bibliographiccitation.journal","European Neuropsychopharmacology"],["dc.bibliographiccitation.volume","29"],["dc.contributor.author","Gade, Katrin"],["dc.contributor.author","Papiol, Sergi"],["dc.contributor.author","Heilbronner, Urs"],["dc.contributor.author","Budde, Monika"],["dc.contributor.author","Adorjan, Kristina"],["dc.contributor.author","Anderson-Schmidt, Heike"],["dc.contributor.author","Kalman, Janos"],["dc.contributor.author","Aldinger, Fanny"],["dc.contributor.author","Andlauer, Till F.M."],["dc.contributor.author","Schulze, Thomas"],["dc.date.accessioned","2020-12-10T14:23:55Z"],["dc.date.available","2020-12-10T14:23:55Z"],["dc.date.issued","2019"],["dc.identifier.doi","10.1016/j.euroneuro.2017.08.333"],["dc.identifier.issn","0924-977X"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/72075"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","THE IMPACT OF BIPOLAR AND SCHIZOPHRENIA POLYGENIC RISK SCORES ON FAMILIAL FEATURES OF BIPOLAR DISORDER"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","340"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","American Journal of Medical Genetics Part B: Neuropsychiatric Genetics"],["dc.bibliographiccitation.lastpage","345"],["dc.bibliographiccitation.volume","156B"],["dc.contributor.author","Papiol, Sergi"],["dc.contributor.author","Begemann, Martin"],["dc.contributor.author","Rosenberger, Albert"],["dc.contributor.author","Friedrichs, Heidi"],["dc.contributor.author","Ribbe, Katja"],["dc.contributor.author","Grube, Sabrina"],["dc.contributor.author","Schwab, Markus H."],["dc.contributor.author","Jahn, Henriette"],["dc.contributor.author","Gunkel, Stefan"],["dc.contributor.author","Benseler, Fritz"],["dc.contributor.author","Nave, Klaus-Armin"],["dc.contributor.author","Ehrenreich, Hannelore"],["dc.date.accessioned","2017-09-07T11:46:36Z"],["dc.date.available","2017-09-07T11:46:36Z"],["dc.date.issued","2011"],["dc.description.abstract","By pure endpoint diagnosis of the disease, the risk of developing schizophrenia has been repeatedly associated with specific variants of the neuregulin1 (NRG1) gene. However, the role of NRG1 in the etiology of schizophrenia has remained unclear. Since Nrg1 serves vital functions in early brain development of mice, we hypothesized that human NRG1 alleles codetermine developmentally influenced readouts of the disease: age of onset and positive symptom severity. We analyzed 1,071 comprehensively phenotyped schizophrenic/schizoaffective patients, diagnosed according to DSM-IV-TR, from the GRAS (Göttingen Research Association for Schizophrenia) Data Collection for genetic variability in the Icelandic region of risk in the NRG1 gene. For the case-control analysis part of the study, we included 1,056 healthy individuals with comparable ethnicity. The phenotype-based genetic association study (PGAS) was performed on the GRAS sample. Instead of a risk constellation, we detected that several haplotypic variants of NRG1 were, unexpectedly, less frequent in the schizophrenic than in the control sample (mean OR=0.78, range between 0.68 and 0.85). In the PGAS we found that these \"protective\" NRG1 variants are specifically underrepresented in subgroups of schizophrenic subjects with early age of onset and high positive symptom load. The GRAS Data Collection as a prerequisite for PGAS has enabled us to associate protective NRG1 genotypes with later onset and milder course of schizophrenia."],["dc.identifier.doi","10.1002/ajmg.b.31168"],["dc.identifier.gro","3150547"],["dc.identifier.pmid","21234898"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/7321"],["dc.language.iso","en"],["dc.notes.status","final"],["dc.title","A phenotype-based genetic association study reveals the contribution of neuregulin1 gene variants to age of onset and positive symptom severity in schizophrenia"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dc.type.peerReviewed","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","S1300"],["dc.bibliographiccitation.journal","European Neuropsychopharmacology"],["dc.bibliographiccitation.lastpage","S1301"],["dc.bibliographiccitation.volume","29"],["dc.contributor.author","Comes, Ashley"],["dc.contributor.author","Adorjan, Kristina"],["dc.contributor.author","Andlauer, Till"],["dc.contributor.author","Budde, Monika"],["dc.contributor.author","Degenhardt, Franziska"],["dc.contributor.author","Forstner, Andreas J."],["dc.contributor.author","Gade, Katrin"],["dc.contributor.author","Heilbronner, Urs"],["dc.contributor.author","Kalman, Janos"],["dc.contributor.author","Kondofersky, Ivan"],["dc.contributor.author","Papiol, Sergi"],["dc.contributor.author","Senner, Fanny"],["dc.contributor.author","Sivalingam, Sugirthan"],["dc.contributor.author","Falkai, Peter"],["dc.contributor.author","Schulze, Thomas"],["dc.date.accessioned","2020-12-10T14:23:56Z"],["dc.date.available","2020-12-10T14:23:56Z"],["dc.date.issued","2019"],["dc.identifier.doi","10.1016/j.euroneuro.2018.08.426"],["dc.identifier.issn","0924-977X"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/72083"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","SU62THE ROLE OF ENVIRONMENTAL STRESS AND DNA METHYLATION IN THE LONGITUDINAL COURSE OF BIPOLAR DISORDER"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.journal","JAMA Psychiatry"],["dc.contributor.author","Amare, Azmeraw T."],["dc.contributor.author","Schubert, Klaus Oliver"],["dc.contributor.author","Hou, Liping"],["dc.contributor.author","Clark, Scott R."],["dc.contributor.author","Papiol, Sergi"],["dc.contributor.author","Heilbronner, Urs"],["dc.contributor.author","Degenhardt, Franziska"],["dc.contributor.author","Tekola-Ayele, Fasil"],["dc.contributor.author","Hsu, Yi-Hsiang"],["dc.contributor.author","Shekhtman, Tatyana"],["dc.contributor.author","Baune, Bernhard T."],["dc.date.accessioned","2020-12-10T14:05:46Z"],["dc.date.available","2020-12-10T14:05:46Z"],["dc.date.issued","2017"],["dc.identifier.doi","10.1001/jamapsychiatry.2017.3433"],["dc.identifier.issn","2168-622X"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/69650"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","Association of Polygenic Score for Schizophrenia and HLA Antigen and Inflammation Genes With Response to Lithium in Bipolar Affective Disorder"],["dc.title.alternative","A Genome-Wide Association Study"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.artnumber","17823"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Scientific Reports"],["dc.bibliographiccitation.volume","11"],["dc.contributor.author","Le Clerc, Sigrid"],["dc.contributor.author","Lombardi, Laura"],["dc.contributor.author","Baune, Bernhard T."],["dc.contributor.author","Amare, Azmeraw T."],["dc.contributor.author","Schubert, Klaus Oliver"],["dc.contributor.author","Hou, Liping"],["dc.contributor.author","Clark, Scott R."],["dc.contributor.author","Papiol, Sergi"],["dc.contributor.author","Cearns, Micah"],["dc.contributor.author","Heilbronner, Urs"],["dc.contributor.author","Tamouza, Ryad"],["dc.date.accessioned","2021-10-01T09:57:45Z"],["dc.date.available","2021-10-01T09:57:45Z"],["dc.date.issued","2021"],["dc.description.abstract","Abstract Bipolar affective disorder (BD) is a severe psychiatric illness, for which lithium (Li) is the gold standard for acute and maintenance therapies. The therapeutic response to Li in BD is heterogeneous and reliable biomarkers allowing patients stratification are still needed. A GWAS performed by the International Consortium on Lithium Genetics (ConLiGen) has recently identified genetic markers associated with treatment responses to Li in the human leukocyte antigens (HLA) region. To better understand the molecular mechanisms underlying this association, we have genetically imputed the classical alleles of the HLA region in the European patients of the ConLiGen cohort. We found our best signal for amino-acid variants belonging to the HLA-DRB1 11:01 classical allele, associated with a better response to Li ( p  < 1 × 10 −3 ; FDR < 0.09 in the recessive model). Alanine or Leucine at position 74 of the HLA-DRB1 heavy chain was associated with a good response while Arginine or Glutamic acid with a poor response. As these variants have been implicated in common inflammatory/autoimmune processes, our findings strongly suggest that HLA-mediated low inflammatory background may contribute to the efficient response to Li in BD patients, while an inflammatory status overriding Li anti-inflammatory properties would favor a weak response."],["dc.description.abstract","Abstract Bipolar affective disorder (BD) is a severe psychiatric illness, for which lithium (Li) is the gold standard for acute and maintenance therapies. The therapeutic response to Li in BD is heterogeneous and reliable biomarkers allowing patients stratification are still needed. A GWAS performed by the International Consortium on Lithium Genetics (ConLiGen) has recently identified genetic markers associated with treatment responses to Li in the human leukocyte antigens (HLA) region. To better understand the molecular mechanisms underlying this association, we have genetically imputed the classical alleles of the HLA region in the European patients of the ConLiGen cohort. We found our best signal for amino-acid variants belonging to the HLA-DRB1 11:01 classical allele, associated with a better response to Li ( p  < 1 × 10 −3 ; FDR < 0.09 in the recessive model). Alanine or Leucine at position 74 of the HLA-DRB1 heavy chain was associated with a good response while Arginine or Glutamic acid with a poor response. As these variants have been implicated in common inflammatory/autoimmune processes, our findings strongly suggest that HLA-mediated low inflammatory background may contribute to the efficient response to Li in BD patients, while an inflammatory status overriding Li anti-inflammatory properties would favor a weak response."],["dc.identifier.doi","10.1038/s41598-021-97140-7"],["dc.identifier.pii","97140"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/89908"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-469"],["dc.relation.eissn","2045-2322"],["dc.title","HLA-DRB1 and HLA-DQB1 genetic diversity modulates response to lithium in bipolar affective disorders"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1143"],["dc.bibliographiccitation.issue","10"],["dc.bibliographiccitation.journal","Molecular Psychiatry"],["dc.bibliographiccitation.lastpage","1149"],["dc.bibliographiccitation.volume","19"],["dc.contributor.author","Hammer, Christian"],["dc.contributor.author","Stepniak, Beata"],["dc.contributor.author","Schneider, Anja"],["dc.contributor.author","Papiol, Sergi"],["dc.contributor.author","Tantra, Martesa"],["dc.contributor.author","Begemann, Martin"],["dc.contributor.author","Sirén, Anna-Leena"],["dc.contributor.author","Pardo, Luis A."],["dc.contributor.author","Sperling, Swetlana"],["dc.contributor.author","Mohd Jofrry, Sue"],["dc.contributor.author","Gurvich, Artem"],["dc.contributor.author","Jensen, Niels"],["dc.contributor.author","Ostmeier, Katrin"],["dc.contributor.author","Lühder, F."],["dc.contributor.author","Probst, Christian"],["dc.contributor.author","Martens, Henrik"],["dc.contributor.author","Gillis, M."],["dc.contributor.author","Saher, Gesine"],["dc.contributor.author","Assogna, F."],["dc.contributor.author","Spalletta, Gianfranco"],["dc.contributor.author","Stöcker, W."],["dc.contributor.author","Schulz, Thomas F."],["dc.contributor.author","Nave, Klaus-Armin"],["dc.contributor.author","Ehrenreich, Hannelore"],["dc.date.accessioned","2017-09-07T11:46:37Z"],["dc.date.available","2017-09-07T11:46:37Z"],["dc.date.issued","2014"],["dc.description.abstract","In 2007, a multifaceted syndrome, associated with anti-NMDA receptor autoantibodies (NMDAR-AB) of immunoglobulin-G isotype, has been described, which variably consists of psychosis, epilepsy, cognitive decline and extrapyramidal symptoms. Prevalence and significance of NMDAR-AB in complex neuropsychiatric disease versus health, however, have remained unclear. We tested sera of 2817 subjects (1325 healthy, 1081 schizophrenic, 263 Parkinson and 148 affective-disorder subjects) for presence of NMDAR-AB, conducted a genome-wide genetic association study, comparing AB carriers versus non-carriers, and assessed their influenza AB status. For mechanistic insight and documentation of AB functionality, in vivo experiments involving mice with deficient blood-brain barrier (ApoE(-/-)) and in vitro endocytosis assays in primary cortical neurons were performed. In 10.5% of subjects, NMDAR-AB (NR1 subunit) of any immunoglobulin isotype were detected, with no difference in seroprevalence, titer or in vitro functionality between patients and healthy controls. Administration of extracted human serum to mice influenced basal and MK-801-induced activity in the open field only in ApoE(-/-) mice injected with NMDAR-AB-positive serum but not in respective controls. Seropositive schizophrenic patients with a history of neurotrauma or birth complications, indicating an at least temporarily compromised blood-brain barrier, had more neurological abnormalities than seronegative patients with comparable history. A common genetic variant (rs524991, P=6.15E-08) as well as past influenza A (P=0.024) or B (P=0.006) infection were identified as predisposing factors for NMDAR-AB seropositivity. The >10% overall seroprevalence of NMDAR-AB of both healthy individuals and patients is unexpectedly high. Clinical significance, however, apparently depends on association with past or present perturbations of blood-brain barrier function."],["dc.identifier.doi","10.1038/mp.2013.110"],["dc.identifier.gro","3150565"],["dc.identifier.pmid","23999527"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/7339"],["dc.language.iso","en"],["dc.notes.status","final"],["dc.title","Neuropsychiatric disease relevance of circulating anti-NMDA receptor autoantibodies depends on blood-brain barrier integrity"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dc.type.peerReviewed","no"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","S1295"],["dc.bibliographiccitation.journal","European Neuropsychopharmacology"],["dc.bibliographiccitation.lastpage","S1296"],["dc.bibliographiccitation.volume","29"],["dc.contributor.author","Heilbronner, Urs"],["dc.contributor.author","Wendel, Bernadette"],["dc.contributor.author","Budde, Monika"],["dc.contributor.author","Gade, Katrin"],["dc.contributor.author","Adorjan, Kristina"],["dc.contributor.author","Kalman, Janos"],["dc.contributor.author","Senner, Fanny"],["dc.contributor.author","Andlauer, Till"],["dc.contributor.author","Comes, Ashley"],["dc.contributor.author","Schulte, Eva"],["dc.contributor.author","Papiol, Sergi"],["dc.contributor.author","Bickeböller, Heike"],["dc.contributor.author","Schulze, Thomas"],["dc.date.accessioned","2020-12-10T14:23:56Z"],["dc.date.available","2020-12-10T14:23:56Z"],["dc.date.issued","2019"],["dc.identifier.doi","10.1016/j.euroneuro.2018.08.417"],["dc.identifier.issn","0924-977X"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/72082"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","SU53A CROSS-DIAGNOSTIC GWAS OF LONGITUDINAL EXECUTIVE FUNCTION PROFILE SCORES"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]