Blaschke, Sabine

[["dc.bibliographiccitation.artnumber","076002"],["dc.bibliographiccitation.issue","7"],["dc.bibliographiccitation.journal","Journal of Biomedical Optics"],["dc.bibliographiccitation.volume","18"],["dc.contributor.author","Montejo, Ludguier D."],["dc.contributor.author","Jia, Jingfei"],["dc.contributor.author","Kim, Hyun K."],["dc.contributor.author","Netz, Uwe J."],["dc.contributor.author","Blaschke, Sabine"],["dc.contributor.author","Mueller, Georg Anton"],["dc.contributor.author","Hielscher, Andreas H."],["dc.date.accessioned","2018-11-07T09:22:50Z"],["dc.date.available","2018-11-07T09:22:50Z"],["dc.date.issued","2013"],["dc.description.abstract","This is the second part of a two-part paper on the application of computer-aided diagnosis to diffuse optical tomography (DOT) for diagnosing rheumatoid arthritis (RA). A comprehensive analysis of techniques for the classification of DOT images of proximal interphalangeal joints of subjects with and without RA is presented. A method for extracting heuristic features from DOT images was presented in Part 1. The ability of five classification algorithms to accurately label each DOT image as belonging to a subject with or without RA is analyzed here. The algorithms of interest are the k-nearest-neighbors, linear and quadratic discriminant analysis, self-organizing maps, and support vector machines (SVM). With a polynomial SVM classifier, we achieve 100.0% sensitivity and 97.8% specificity. Lower bounds for these results (at 95.0% confidence level) are 96.4% and 93.8%, respectively. Image features most predictive of RA are from the spatial variation of optical properties and the absolute range in feature values. The optimal classifiers are low-dimensional combinations (<7 features). These results underscore the high potential for DOT to become a clinically useful diagnostic tool and warrant larger prospective clinical trials to conclusively demonstrate the ultimate clinical utility of this approach. (c) 2013 Society of Photo-Optical Instrumentation Engineers (SPIE)"],["dc.identifier.doi","10.1117/1.JBO.18.7.076002"],["dc.identifier.isi","000323030400010"],["dc.identifier.pmid","23856916"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/29438"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Spie-soc Photo-optical Instrumentation Engineers"],["dc.relation.issn","1083-3668"],["dc.title","Computer-aided diagnosis of rheumatoid arthritis with optical tomography, Part 2: image classification"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.artnumber","45"],["dc.bibliographiccitation.journal","Arthritis Research & Therapy"],["dc.bibliographiccitation.volume","17"],["dc.contributor.author","Blaschke, Sabine"],["dc.contributor.author","Rinke, Kathinka"],["dc.contributor.author","Maring, Michael"],["dc.contributor.author","Flad, Thomas"],["dc.contributor.author","Patschan, Susann A."],["dc.contributor.author","Jahn, Olaf"],["dc.contributor.author","Mueller, Claudia A."],["dc.contributor.author","Mueller, Georg Anton"],["dc.contributor.author","Dihazi, Hassan"],["dc.date.accessioned","2018-11-07T09:59:46Z"],["dc.date.available","2018-11-07T09:59:46Z"],["dc.date.issued","2015"],["dc.description.abstract","Introduction: The introduction of tumor necrosis factor-alpha (TNF-alpha) antagonists has substantially improved patient's clinical outcome in rheumatoid arthritis (RA). However, nearly 20% to 40% of RA patients do not respond to anti-TNF-alpha treatment strategies. To identify valid predictors of TNF-alpha antagonist response in RA, serum proteome profiles from responders (R) and non-responders (NR) to etanercept, a soluble recombinant TNF-alpha receptor/IgG Fc fusion protein receptor, were compared in a prospective cohort study. Methods: In this clinical study 50 RA patients with inadequate response to conventional DMARDs were included and treated with etanercept. The primary efficacy endpoint was response according to the European League against Rheumatism (EULAR) improvement criteria. Serum samples collected prior to initiation and after six months of etanercept therapy were cleared of the most abundant major proteins by immunoaffinity chromatography. After separation by two-dimensional differential gel electrophoresis (2D-DIGE) and identification by mass spectrometry (MS) data were validated by Western blot analysis. Results: After six months of etanercept treatment 62% (n = 31) of RA patients achieved response. Haptoglobin-alpha 1 (Hp-alpha 1) and -alpha 2 (Hp-alpha 2) and vitamin D-binding protein (VDBP) were found to be significantly upregulated in responder sera (P <= 0.02) at study entry. In contrast, apolipoprotein C-III (ApoC-III) showed significantly higher levels in non-responders (P = 0.0162). At study end ApoA-II, Hp-alpha 1, Hp-alpha 2 and VDBP were identified to be expressed at significantly higher levels (P < 0.05) in responder sera. Conclusions: By application of clinical proteomics in immunodepleted sera we could identify and validate for the first time Hp-alpha 1, -alpha 2, VDBP and ApoC-III as potential biomarkers for prediction of etanercept drug response in RA."],["dc.description.sponsorship","Pfizer Research Initiative"],["dc.identifier.doi","10.1186/s13075-015-0553-1"],["dc.identifier.isi","000352187400001"],["dc.identifier.pmid","25884688"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/13467"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/37661"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Biomed Central Ltd"],["dc.relation.issn","1478-6362"],["dc.relation.issn","1478-6354"],["dc.rights","CC BY 4.0"],["dc.rights.uri","https://creativecommons.org/licenses/by/4.0"],["dc.title","Haptoglobin-alpha 1, -alpha 2, vitamin D-binding protein and apolipoprotein C-III as predictors of etanercept drug response in rheumatoid arthritis"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.artnumber","076001"],["dc.bibliographiccitation.issue","7"],["dc.bibliographiccitation.journal","Journal of Biomedical Optics"],["dc.bibliographiccitation.volume","18"],["dc.contributor.author","Montejo, Ludguier D."],["dc.contributor.author","Jia, Jingfei"],["dc.contributor.author","Kim, Hyun K."],["dc.contributor.author","Netz, Uwe J."],["dc.contributor.author","Blaschke, Sabine"],["dc.contributor.author","Mueller, Georg Anton"],["dc.contributor.author","Hielscher, Andreas H."],["dc.date.accessioned","2018-11-07T09:22:51Z"],["dc.date.available","2018-11-07T09:22:51Z"],["dc.date.issued","2013"],["dc.description.abstract","This is the first part of a two-part paper on the application of computer-aided diagnosis to diffuse optical tomography (DOT). An approach for extracting heuristic features from DOT images and a method for using these features to diagnose rheumatoid arthritis (RA) are presented. Feature extraction is the focus of Part 1, while the utility of five classification algorithms is evaluated in Part 2. The framework is validated on a set of 219 DOT images of proximal interphalangeal (PIP) joints. Overall, 594 features are extracted from the absorption and scattering images of each joint. Three major findings are deduced. First, DOT images of subjects with RA are statistically different (p < 0.05) from images of subjects without RA for over 90% of the features investigated. Second, DOT images of subjects with RA that do not have detectable effusion, erosion, or synovitis (as determined by MRI and ultrasound) are statistically indistinguishable from DOT images of subjects with RA that do exhibit effusion, erosion, or synovitis. Thus, this subset of subjects may be diagnosed with RA from DOT images while they would go undetected by reviews of MRI or ultrasound images. Third, scattering coefficient images yield better one-dimensional classifiers. A total of three features yield a Youden index greater than 0.8. These findings suggest that DOT may be capable of distinguishing between PIP joints that are healthy and those affected by RA with or without effusion, erosion, or synovitis. (c) 2013 Society of Photo-Optical Instrumentation Engineers (SPIE)"],["dc.identifier.doi","10.1117/1.JBO.18.7.076001"],["dc.identifier.isi","000323030400009"],["dc.identifier.pmid","23856915"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/29439"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Spie-soc Photo-optical Instrumentation Engineers"],["dc.relation.issn","1083-3668"],["dc.title","Computer-aided diagnosis of rheumatoid arthritis with optical tomography, Part 1: feature extraction"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.issue","10"],["dc.bibliographiccitation.journal","Arthritis & Rheumatism"],["dc.bibliographiccitation.volume","64"],["dc.contributor.author","Blaschke, Sabine"],["dc.contributor.author","Trautmann, Sandra"],["dc.contributor.author","Beham, Alexander W."],["dc.contributor.author","Mai, Burkhard"],["dc.contributor.author","Koelling, Sebastian"],["dc.contributor.author","Breysach, Caroline"],["dc.contributor.author","Wolf, Gabriele"],["dc.contributor.author","Mueller, Georg Anton"],["dc.contributor.author","Miosge, Nicolai"],["dc.date.accessioned","2018-11-07T09:04:58Z"],["dc.date.available","2018-11-07T09:04:58Z"],["dc.date.issued","2012"],["dc.format.extent","S517"],["dc.identifier.isi","000309748302284"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/25220"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Wiley-blackwell"],["dc.publisher.place","Hoboken"],["dc.relation.conference","Annual Scientific Meeting of the American-College-of-Rheumatology (ACR) and Association-of-Rheumatology-Health-Professionals (ARHP)"],["dc.relation.eventlocation","Washington, DC"],["dc.relation.issn","0004-3591"],["dc.title","Functional Role of Chondrogenic Progenitor Cells in Rheumatoid Arthritis."],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1312"],["dc.bibliographiccitation.issue","7"],["dc.bibliographiccitation.journal","Intensive Care Medicine"],["dc.bibliographiccitation.lastpage","1313"],["dc.bibliographiccitation.volume","35"],["dc.contributor.author","Hagenah, Gerrit C."],["dc.contributor.author","Klinger, Michael"],["dc.contributor.author","Nagorsnik, Ulf"],["dc.contributor.author","Puls, Miriam"],["dc.contributor.author","Schweyer, Stefan"],["dc.contributor.author","Mueller, Georg Anton"],["dc.contributor.author","Blaschke, Sabine"],["dc.date.accessioned","2018-11-07T08:28:11Z"],["dc.date.available","2018-11-07T08:28:11Z"],["dc.date.issued","2009"],["dc.identifier.doi","10.1007/s00134-009-1398-3"],["dc.identifier.isi","000267220800027"],["dc.identifier.pmid","19169669"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?goescholar/3478"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/16365"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.relation.issn","0342-4642"],["dc.relation.orgunit","Wirtschaftswissenschaftliche Fakultät"],["dc.rights","Goescholar"],["dc.rights.uri","https://goescholar.uni-goettingen.de/licenses"],["dc.title","Acute renal failure due to severe rhabdomyolysis: a rare clinical manifestation of atrial myxoma"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.subtype","letter_note"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1725"],["dc.bibliographiccitation.issue","10"],["dc.bibliographiccitation.journal","IEEE Transactions on Medical Imaging"],["dc.bibliographiccitation.lastpage","1736"],["dc.bibliographiccitation.volume","30"],["dc.contributor.author","Hielscher, Andreas H."],["dc.contributor.author","Kim, Hyun Keol"],["dc.contributor.author","Montejo, Ludguier D."],["dc.contributor.author","Blaschke, Sabine"],["dc.contributor.author","Netz, Uwe J."],["dc.contributor.author","Zwaka, Paul Anton"],["dc.contributor.author","Illing, Gerd"],["dc.contributor.author","Mueller, Georg Anton"],["dc.contributor.author","Beuthan, Juergen"],["dc.date.accessioned","2018-11-07T08:51:16Z"],["dc.date.available","2018-11-07T08:51:16Z"],["dc.date.issued","2011"],["dc.description.abstract","We are presenting data from the largest clinical trial on optical tomographic imaging of finger joints to date. Overall we evaluated 99 fingers of patients affected by rheumatoid arthritis (RA) and 120 fingers from healthy volunteers. Using frequency-domain imaging techniques we show that sensitivities and specificities of 0.85 and higher can be achieved in detecting RA. This is accomplished by deriving multiple optical parameters from the optical tomographic images and combining them for the statistical analysis. Parameters derived from the scattering coefficient perform slightly better than absorption derived parameters. Furthermore we found that data obtained at 600 MHz leads to better classification results than data obtained at 0 or 300 MHz."],["dc.identifier.doi","10.1109/TMI.2011.2135374"],["dc.identifier.isi","000295511400001"],["dc.identifier.pmid","21964730"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/21890"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Ieee-inst Electrical Electronics Engineers Inc"],["dc.relation.issn","0278-0062"],["dc.title","Frequency-Domain Optical Tomographic Imaging of Arthritic Finger Joints"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1558"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","Nephrology Dialysis Transplantation"],["dc.bibliographiccitation.lastpage","1566"],["dc.bibliographiccitation.volume","22"],["dc.contributor.author","Markovic-Lipkovski, Jasmina"],["dc.contributor.author","Mueller, Claudia A."],["dc.contributor.author","Klein, Gerd"],["dc.contributor.author","Flad, Thomas"],["dc.contributor.author","Klatt, Tatjana"],["dc.contributor.author","Blaschke, Sabine"],["dc.contributor.author","Wessels, Johannes Theodor"],["dc.contributor.author","Mueller, Georg Anton"],["dc.date.accessioned","2018-11-07T11:02:05Z"],["dc.date.available","2018-11-07T11:02:05Z"],["dc.date.issued","2007"],["dc.description.abstract","Background. At early stages of kidney development, the neural cell adhesion molecule (NCAM) is highly expressed on cells of the metanephrogenic mesenchyme. During maturation of the fetal kidney, NCAM gradually disappears. So far, it has been widely accepted that NCAM in the adult kidney is only expressed by nerves, and not by other cell types. Methods. NCAM expression was analysed in human adult healthy and diseased kidneys by immunohistochemistry and western blot analysis. NCAM+ renal interstitial cells were further characterized by double immunofluorescent staining using antibodies against neurofilaments, a smooth muscle actin, vimentin, alpha 5 beta 1 integrin, CD68, CD11c, HLA-DR and the potential progenitor cell markers CD34, CD117, CD133, CD24, nestin and cadherin-11. Results. In adult human kidneys, NCAM expression is restricted to rare interstitial cells with dendritic morphology, which are neurofilament-negative and predominantly localized on the corticomedullary junction. They are also negative for fibroblast cell markers, but co-express the haematopoietic stem cell markers CD34 and CD133. The number of NCAM+ interstitial cells increased in the initial phases of interstitial fibrosis. Western blot analysis of renal tissues with incipient interstitial fibrosis tissues showed the expression of the 140 kDa NCAM isoform. Conclusions. These data indicate that a rare subpopulation of NCAM+ interstitial cells could represent renal progenitors, and that NCAM+ interstitial cells can participate in the initial phase of interstitial fibrosis."],["dc.identifier.doi","10.1093/ndt/gfm006"],["dc.identifier.isi","000247474200012"],["dc.identifier.pmid","17337466"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/51294"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Oxford Univ Press"],["dc.publisher.place","Oxford"],["dc.relation.conference","19th European Congress of Pathology"],["dc.relation.eventlocation","Ljubljana, SLOVENIA"],["dc.relation.issn","0931-0509"],["dc.title","Neural cell adhesion molecule expression on renal interstitial cells"],["dc.type","conference_paper"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","447"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","HISTOLOGY AND HISTOPATHOLOGY"],["dc.bibliographiccitation.lastpage","455"],["dc.bibliographiccitation.volume","24"],["dc.contributor.author","Zopf, Steffen"],["dc.contributor.author","Flaemig, Jakob"],["dc.contributor.author","Schmid, Heide"],["dc.contributor.author","Miosge, Nicolai"],["dc.contributor.author","Blaschke, Sabine"],["dc.contributor.author","Hahn, Eckhart G."],["dc.contributor.author","Mueller, Georg Anton"],["dc.contributor.author","Grunewald, Rolf W."],["dc.date.accessioned","2018-11-07T08:31:07Z"],["dc.date.available","2018-11-07T08:31:07Z"],["dc.date.issued","2009"],["dc.description.abstract","Sorbitol plays an important role in the osmotic regulation of the mammalian kidney. Sorbitol synthesis is regulated by the enzyme aldose reductase (AR) and its degradation to fructose is catalyzed by the enzyme sorbitol dehydrogenase (SDH). Various data exist on the polyol pathway on the rat kidney, but little is known about the distribution of the polyol pathway enzymes in the human kidney. Determination of enzyme activities and a semiquantitative determination of mRNA expression, immunohistochemistry and in-situ hybridisation in healthy human kidney tissue was carried out. The enzyme activity of AR showed a fourfold increase from cortex to papilla, while SDH-activity dropped from cortex to papilla by a factor of four. Corresponding data was obtained at the mRNA level from the semiquantitative polymerase chain reaction (PCR). Additional differentiation at the cellular level reveals both enzymes in cells of the proximal and distal tubules, thick ascending loop, thin loop and collecting duct. Studies of enzyme activity and expression by immunohistochemistry, PCR and in-situ hybridization presented corresponding results with respect to the localization of the enzymes, which match the experimental data obtained from rats very well. Thus, the established rat model might well represent the situation in the human kidney, too."],["dc.identifier.isi","000263512800006"],["dc.identifier.pmid","19224447"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/17049"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","F Hernandez"],["dc.relation.issn","0213-3911"],["dc.title","Localization of the polyol pathway in the human kidney"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","513"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","CLINICAL AND EXPERIMENTAL RHEUMATOLOGY"],["dc.bibliographiccitation.lastpage","520"],["dc.bibliographiccitation.volume","34"],["dc.contributor.author","Vasko, Radovan"],["dc.contributor.author","Streich, J.-H."],["dc.contributor.author","Blaschke, S."],["dc.contributor.author","Mueller, Georg Anton"],["dc.contributor.author","Mai, Burkhard"],["dc.contributor.author","Kostrzewa, Markus"],["dc.contributor.author","Sparbier, Katrin"],["dc.contributor.author","Korsten, Peter"],["dc.contributor.author","Bohr, Stefan"],["dc.contributor.author","Dihazi, Hassan"],["dc.date.accessioned","2018-11-07T10:14:47Z"],["dc.date.available","2018-11-07T10:14:47Z"],["dc.date.issued","2016"],["dc.description.abstract","Objective To study the protein expression differences between primary fibroblasts explanted from synovial membranes of patients with rheumatoid arthritis (RA) and osteoarthritis (OA). Methods Fibroblast cultures were obtained from 10 patients with RA and 5 patients with OA. After two-dimensional gel electrophoresis, proteins were excised and identified using peptide mass fingerprint. Expression of selected proteins was subsequently examined by immunoblot. Furthermore, we examined the cellular lysates for the presence of citrullinated proteins. Results The study was designed to compare expression changes of the common proteins detected in all studied fibroblast cultures (i.e. detected in all patients samples). We totally identified 191 shared proteins between RA and OA fibroblasts. A significant difference was defined as at least 2-fold upregulation or 0.6-fold downregulation of protein expression. The most obvious alteration observed in RA was the appearance of several vimentin fragments not present in OA. We did not detect citrullinated proteins in lysates from RA fibroblasts. This corroborates the current assumption that fibroblasts are not able to citrullinate proteins by themselves and that invading macrophages play a central role in this process. Conclusion We demonstrated that fibroblasts from patients with RA, despite being grown under identical conditions, preserve a particular feature and generate vimentin fragments not present in fibroblasts from OA. Elevated levels of different vimentin fragments have been recently reported in several rheumatic conditions. Further studies are needed to elucidate the pathogenic mechanisms induced by vimentin fragments in RA."],["dc.identifier.isi","000376977900019"],["dc.identifier.pmid","27049516"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/40684"],["dc.language.iso","en"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.relation.issn","1593-098X"],["dc.relation.issn","0392-856X"],["dc.title","Vimentin fragments are potential markers of rheumatoid synovial fibroblasts"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.issue","9"],["dc.bibliographiccitation.journal","Arthritis & Rheumatism"],["dc.bibliographiccitation.volume","58"],["dc.contributor.author","Blaschke, Sabine"],["dc.contributor.author","Brandt, Philip"],["dc.contributor.author","Wessels, Johannes"],["dc.contributor.author","Mueller, Georg Anton"],["dc.date.accessioned","2018-11-07T11:11:04Z"],["dc.date.available","2018-11-07T11:11:04Z"],["dc.date.issued","2008"],["dc.format.extent","S663"],["dc.identifier.isi","000259244201586"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/53349"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Wiley-liss"],["dc.publisher.place","Hoboken"],["dc.relation.conference","72nd Annual Scientific Meeting of the American-College-of-Rheumatology/43rd Annual Scientific Meeting of the Association-of-Rheumatology-Health-Professionals"],["dc.relation.eventlocation","San Francisco, CA"],["dc.relation.issn","0004-3591"],["dc.title","Expression and functional role of the C-class chemokine lymphotactin (XCL1) in Wegener's granulomatosis"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]