Schmidt, Holger

[["dc.bibliographiccitation.firstpage","61"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Journal of Vascular Surgery"],["dc.bibliographiccitation.lastpage","70"],["dc.bibliographiccitation.volume","53"],["dc.contributor.author","Wasser, Katrin"],["dc.contributor.author","Pilgram-Pastor, Sara M."],["dc.contributor.author","Schnaudigel, Sonja"],["dc.contributor.author","Stojanovic, Tomislav"],["dc.contributor.author","Schmidt, Holger"],["dc.contributor.author","Knauf, Jana"],["dc.contributor.author","Groeschel, Klaus"],["dc.contributor.author","Knauth, Michael"],["dc.contributor.author","Hildebrandt, Helmut"],["dc.contributor.author","Kastrup, Andreas"],["dc.date.accessioned","2018-11-07T09:01:37Z"],["dc.date.available","2018-11-07T09:01:37Z"],["dc.date.issued","2011"],["dc.description.abstract","Purpose: Carotid angioplasty and stenting (CAS) is increasingly being used as a treatment alternative to endarterectomy (CEA) for patients with significant carotid stenosis. However, diffusion-weighted imaging (DWI) has indicated that CAS is associated with a significantly higher burden of microemboli. This study evaluated the potential effect on intellectual functions of new DWI lesions after CEA or CAS. Methods: This prospective study analyzed the neuropsychologic outcomes after revascularization in 24 CAS and 31 CEA patients with severe carotid stenosis compared with a control group of 27 healthy individuals. All patients underwent clinical examinations, magnetic resonance imaging scans, and a neuropsychologic test battery that assessed six major cognitive domains performed immediately before CEA or CAS, <= 72 hours after, and at 3 months. Results: New DWI lesions were detected among 15 of 21 (71%) of the CAS patients immediately after treatment but in only 1 of the 28 CEA patients (4%; P < .01). As a group, patients with new DWI lesions showed a decline in their performance in the cognitive domains, attention, and visuoconstructive functions within 72 hours of carotid revascularization. Individually, however, in none of the cognitive domains did the decreases reach a clinically relevant threshold of z < -1.5. Moreover, the cognitive performance was not significantly different between patients with and without new DWI lesions 3 months after treatment. The cognitive performance was similar between CEA and CAS patients at all points. Conclusions: The findings support the assumption that new brain lesions, as detected with DWI after CAS or CEA, do not affect cognitive performance in a manner that is long-lasting or clinically relevant. Despite the higher embolic load detected by DWI, CAS is not associated with a greater cognitive decline than CEA. (J Vasc Surg 2011;53:61-70.)"],["dc.identifier.doi","10.1016/j.jvs.2010.07.061"],["dc.identifier.isi","000286085200009"],["dc.identifier.pmid","20875716"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/24474"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Mosby-elsevier"],["dc.relation.issn","0741-5214"],["dc.title","New brain lesions after carotid revascularization are not associated with cognitive performance"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1041"],["dc.bibliographiccitation.issue","11"],["dc.bibliographiccitation.journal","Journal of Neurology Neurosurgery & Psychiatry"],["dc.bibliographiccitation.lastpage","1047"],["dc.bibliographiccitation.volume","83"],["dc.contributor.author","von Steinbuechel, Nicole"],["dc.contributor.author","Wilson, Lindsay"],["dc.contributor.author","Gibbons, Henning"],["dc.contributor.author","Muehlan, Holger"],["dc.contributor.author","Schmidt, Holger"],["dc.contributor.author","Schmidt, Silke"],["dc.contributor.author","Sasse, Nadine"],["dc.contributor.author","Koskinen, Sanna"],["dc.contributor.author","Sarajuuri, Jaana"],["dc.contributor.author","Hoefer, Stefan"],["dc.contributor.author","Bullinger, Monika"],["dc.contributor.author","Maas, Andrew"],["dc.contributor.author","Neugebauer, Edmund"],["dc.contributor.author","Powell, Jane"],["dc.contributor.author","von Wild, Klaus"],["dc.contributor.author","Zitnay, George"],["dc.contributor.author","Bakx, Wilbert"],["dc.contributor.author","Christensen, Anne-Lise"],["dc.contributor.author","Formisano, Rita"],["dc.contributor.author","Hawthorne, Graeme"],["dc.contributor.author","Truelle, Jean-Luc"],["dc.date.accessioned","2018-11-07T09:04:16Z"],["dc.date.available","2018-11-07T09:04:16Z"],["dc.date.issued","2012"],["dc.description.abstract","Background The Quality of Life after Brain Injury (QOLIBRI) scale is a recently developed instrument that provides a profile of health-related quality of life (HRQoL) in domains typically affected by brain injury. However, for global assessment it is desirable to have a brief summary measure. This study examined a 6-item QOLIBRI Overall Scale (QOLIBRI-OS), and considered whether it could provide an index of HRQoL after traumatic brain injury (TBI). Methods The properties of the QOLIBRI-OS were studied in a sample of 792 participants with TBI recruited from centres in nine countries covering six languages. An examination of construct validity was undertaken on a subsample of 153 participants recruited in Germany who had been assessed on two relevant brief quality of life measures, the Satisfaction With Life Scale and the Quality of Life Visual Analogue Scale. Results The reliability of the QOLIBRI-OS was good (Cronbach's a 0.86, test-retest reliability =0.81) and similar in participants with higher and lower cognitive performance. Factor analysis indicated that the scale is unidimensional. Rasch analysis also showed a satisfactory fit with this model. The QOLIBRI-OS correlates highly with the total score from the full QOLIBRI scale (r=0.87). Moderate to strong relationships were found among the QOLIBRI-OS and the Extended Glasgow Outcome Scale, Short-Form-36, and Hospital Anxiety and Depression scale (r=0.54 to -0.76). The QOLIBRI-OS showed good construct validity in the TBI group. Conclusions The QOLIBRI-OS assesses a similar construct to the QOLIBRI total score and can be used as a brief index of HRQoL for TBI."],["dc.identifier.doi","10.1136/jnnp-2012-302361"],["dc.identifier.isi","000309813600005"],["dc.identifier.pmid","22851609"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/25081"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Bmj Publishing Group"],["dc.relation.issn","0022-3050"],["dc.title","QOLIBRI Overall Scale: a brief index of health-related quality of life after traumatic brain injury"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","30"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Journal of Neuroimmunology"],["dc.bibliographiccitation.lastpage","39"],["dc.bibliographiccitation.volume","113"],["dc.contributor.author","Schmidt, H"],["dc.contributor.author","Tlustochowska, A."],["dc.contributor.author","Stuertz, K."],["dc.contributor.author","Djukic, M."],["dc.contributor.author","Gerber, Joachim"],["dc.contributor.author","Schutz, Ekkehard"],["dc.contributor.author","Kuhnt, U."],["dc.contributor.author","Nau, R."],["dc.date.accessioned","2021-06-01T10:50:09Z"],["dc.date.available","2021-06-01T10:50:09Z"],["dc.date.issued","2001"],["dc.description.abstract","Hippocampal slices of newborn rats were exposed to either heat-inactivated Streptococcus pneumoniae R6 (hiR6) equivalent to 10(6) and 10(8) CFU/ml, lipoteichoic acid (LTA) (0.3 mug/ml and 30 mug/ml), peptidoglycans (PG) (0.3, 30, 50 and 100 mug/ml), pneumococcal DNA (pDNA) (0.3 and 30 mug/ml) or medium only (control). Cell injury was examined by Nissl staining, Annexin V and NeuN immunohistochemistry, and quantified by propidium iodide (PI) uptake and by determining neuron-specific enolase (NSE) concentration in the culture medium. Necrotic and apoptotic cell damage occurred in all treatment groups. Overall damage (Nissl and PI staining) was most prominent after hiR6 (10(8) CFU/ml), followed by LTA (30 mug/ml), pDNA (30 mug/ml), and not detectable after PG (30 mug/ml) exposure. PG (100 mug/ml) induced severe damage. Apoptotic cells were most frequent after exposure to LTA and hiR6. Damage in the neuronal cell layers (NeuN, NSE) was most severe after treatment with hiR6 (10(8) CFU/ml), followed by PG (100 mug/ml), pDNA (30 mug/ml), and LTA (30 mug/ml). (C) 2001 Elsevier Science B.V. All rights reserved."],["dc.identifier.doi","10.1016/S0165-5728(00)00402-1"],["dc.identifier.isi","000166575000004"],["dc.identifier.pmid","11137574"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/86547"],["dc.notes.intern","DOI-Import GROB-425"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Elsevier Science Bv"],["dc.relation.issn","0165-5728"],["dc.title","Organotypic hippocampal cultures A model of brain tissue damage in Streptococcus pneumoniae meningitis"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","152"],["dc.bibliographiccitation.issue","1-2"],["dc.bibliographiccitation.journal","Journal of the Neurological Sciences"],["dc.bibliographiccitation.lastpage","154"],["dc.bibliographiccitation.volume","331"],["dc.contributor.author","Schmidt, Holger"],["dc.contributor.author","Kretzschmar, Benedikt"],["dc.contributor.author","Lingor, Paul"],["dc.contributor.author","Pauli, Silke"],["dc.contributor.author","Schramm, Peter"],["dc.contributor.author","Otto, Markus"],["dc.contributor.author","Ohlenbusch, Andreas"],["dc.contributor.author","Brockmann, Knut"],["dc.date.accessioned","2018-11-07T09:21:15Z"],["dc.date.available","2018-11-07T09:21:15Z"],["dc.date.issued","2013"],["dc.description.abstract","Adult-onset Alexander disease (AOAD) is a rare leukoencephalopathy affecting predominantly the brainstem and cervical cord with insidious onset of clinical features. Acute onset is very rare and has yet been described only twice, to our knowledge. We report a 32-year-old hitherto healthy male who, after excessive consumption of alcohol, presented with stroke-like onset of symptoms including rigidospasticity, loss of consciousness, and bulbar dysfunction. MRI features comprised bilateral T2-hyperintensities of frontal white matter and basal ganglia as well as atrophy of medulla oblongata with a peculiar \"tadpole\" appearance, a pattern characteristic of AOAD. Mutation analysis of the GFAP gene revealed a heterozygous de novo 9-bp microduplication in exon 1. Adult Alexander disease may present with stroke-like features. MRI patterns of chronic neurodegenerative conditions may be recognizable even in acute neurological emergencies. (C) 2013 Elsevier B.V. All rights reserved."],["dc.identifier.doi","10.1016/j.jns.2013.05.006"],["dc.identifier.isi","000322415000030"],["dc.identifier.pmid","23706596"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/29072"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Elsevier Science Bv"],["dc.relation.issn","0022-510X"],["dc.title","Acute onset of adult Alexander disease"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","464"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","Journal of Head Trauma Rehabilitation"],["dc.bibliographiccitation.lastpage","472"],["dc.bibliographiccitation.volume","28"],["dc.contributor.author","Sasse, Nadine"],["dc.contributor.author","Gibbons, Henning"],["dc.contributor.author","Wilson, Lindsay"],["dc.contributor.author","Martinez-Olivera, Ramon"],["dc.contributor.author","Schmidt, Holger"],["dc.contributor.author","Hasselhorn, Marcus"],["dc.contributor.author","von Wild, Klaus"],["dc.contributor.author","von Steinbuechel, Nicole"],["dc.date.accessioned","2018-11-07T09:17:44Z"],["dc.date.available","2018-11-07T09:17:44Z"],["dc.date.issued","2013"],["dc.description.abstract","Objective: To investigate the relations among self-awareness (SA), impaired SA, and health-related quality of life (HRQOL) after traumatic brain injury (TBI). Participants: One hundred forty-one adults hospitalized with TBI and their significant others from a cross-sectional multicenter study. Using Glasgow Coma Scale classification, 32 participants had severe injuries, 29 moderate, 44 mild, and 25 complicated mild TBI. Measures: Patient Competency Rating Scale for Neurorehabilitation; Short Form-36 Health Survey; Cognitive Quality of Life; Quality Of Life after Brain Injury; Hospital Anxiety and Depression Scale; Profile of Mood States; Glasgow Outcome Scale Extended. Method: Patient Competency Rating Scale for Neurorehabilitation ratings made by participants and their significant others were used to assess SA and discrepancies between the 2 ratings were used to define impaired SA. Results: Significant associations were identified between SA and HRQOL, anxiety, depression, and severity of injury. Participants with and without impaired SA differed in cognitive HRQOL and leisure activities. Using multiple regression, no direct predictors of SA were identified, although interaction effects were observed. Conclusion: After TBI, lower SA is associated with higher estimates of HRQOL, particularly in the cognitive domain. Although the associations are modest, the assessment of SA should play a role in the interpretation of reported HRQOL after TBI."],["dc.description.sponsorship","ZNS-Hannelore Kohl Foundation, Germany [2008014]"],["dc.identifier.doi","10.1097/HTR.0b013e318263977d"],["dc.identifier.isi","000330365200011"],["dc.identifier.pmid","22935572"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/28239"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Lippincott Williams & Wilkins"],["dc.relation.issn","1550-509X"],["dc.relation.issn","0885-9701"],["dc.title","Self-Awareness and Health-Related Quality of Life After Traumatic Brain Injury"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.artnumber","e1002380"],["dc.bibliographiccitation.issue","11"],["dc.bibliographiccitation.journal","PLOS Pathogens"],["dc.bibliographiccitation.volume","7"],["dc.contributor.author","Cordeiro, Tiago N."],["dc.contributor.author","Schmidt, Holger"],["dc.contributor.author","Madrid, Cristina"],["dc.contributor.author","Juarez, Antonio"],["dc.contributor.author","Bernado, Pau"],["dc.contributor.author","Griesinger, Christian"],["dc.contributor.author","Garcia, Jesus"],["dc.contributor.author","Pons, Miquel"],["dc.date.accessioned","2017-09-07T11:43:18Z"],["dc.date.available","2017-09-07T11:43:18Z"],["dc.date.issued","2011"],["dc.description.abstract","Ler, a member of the H-NS protein family, is the master regulator of the LEE pathogenicity island in virulent Escherichia coli strains. Here, we determined the structure of a complex between the DNA-binding domain of Ler (CT-Ler) and a 15-mer DNA duplex. CT-Ler recognizes a preexisting structural pattern in the DNA minor groove formed by two consecutive regions which are narrower and wider, respectively, compared with standard B-DNA. The compressed region, associated with an AT-tract, is sensed by the side chain of Arg90, whose mutation abolishes the capacity of Ler to bind DNA. The expanded groove allows the approach of the loop in which Arg90 is located. This is the first report of an experimental structure of a DNA complex that includes a protein belonging to the H-NS family. The indirect readout mechanism not only explains the capacity of H-NS and other H-NS family members to modulate the expression of a large number of genes but also the origin of the specificity displayed by Ler. Our results point to a general mechanism by which horizontally acquired genes may be specifically recognized by members of the H-NS family."],["dc.identifier.doi","10.1371/journal.ppat.1002380"],["dc.identifier.gro","3142634"],["dc.identifier.isi","000297337300040"],["dc.identifier.pmid","22114557"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/60"],["dc.notes.intern","WoS Import 2017-03-10"],["dc.notes.status","final"],["dc.notes.submitter","PUB_WoS_Import"],["dc.publisher","Public Library Science"],["dc.relation.issn","1553-7374"],["dc.title","Indirect DNA Readout by an H-NS Related Protein: Structure of the DNA Complex of the C-Terminal Domain of Ler"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.subtype","original"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","238"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","Journal of Clinical Laboratory Analysis"],["dc.bibliographiccitation.lastpage","245"],["dc.bibliographiccitation.volume","26"],["dc.contributor.author","Bibl, Mirko"],["dc.contributor.author","Welge, Volker"],["dc.contributor.author","Schmidt, Holger"],["dc.contributor.author","Esselmann, Hermann"],["dc.contributor.author","Mollenhauer, Brit"],["dc.contributor.author","Lewczuk, Piotr"],["dc.contributor.author","Otto, Markus"],["dc.contributor.author","Kornhuber, Johannes"],["dc.contributor.author","Wiltfang, Jens"],["dc.date.accessioned","2018-11-07T09:15:09Z"],["dc.date.available","2018-11-07T09:15:09Z"],["dc.date.issued","2012"],["dc.description.abstract","Background Blood-based tests for a rapid and valid diagnosis as well as outcome prognosis of acute stroke are desirable. Recently, plasma A beta 40 was suggested as an independent cerebrovascular risk factor candidate. Methods We investigated eight plasma samples of patients with clinical signs of acute cerebral ischemia for derangements of plasma amyloid-beta (A beta) peptide patterns as compared to 13 patients with other neuropsychiatric diseases. For the analysis of plasma, we used immunoprecipitation followed by the quantitative A beta-SDS-PAGE/immunoblot. Results The major outcome was a striking decrease of A beta 140 in plasma paralleled by an increase in the ratio of A beta 138/A beta 140 in two patients with acute stroke. Interestingly, these patients had an onset of symptoms within only 24 hr before venous puncture and there was a strong correlation of A beta 138/A beta 140 levels with the time span between onset of symptoms and venous puncture. Conclusion From these results, we suggest the ratio of plasma A beta 138/A beta 140 as a possible biomarker for the early diagnosis of acute stroke. J. Clin. Lab. Anal. 26:238-245, 2012. (c) 2012 Wiley Periodicals, Inc."],["dc.identifier.doi","10.1002/jcla.21511"],["dc.identifier.isi","000306511500004"],["dc.identifier.pmid","22811355"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/27608"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Wiley-blackwell"],["dc.relation.issn","0887-8013"],["dc.title","Plasma Amyloid-BetaPeptides in Acute Cerebral Ischemia: A Pilot Study"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.journal","Quality of Life Research"],["dc.bibliographiccitation.volume","24"],["dc.contributor.author","von Steinbuechel, Nicole"],["dc.contributor.author","Groeneveld, Annette"],["dc.contributor.author","Schmidt, Holger"],["dc.contributor.author","von Wild, Klaus"],["dc.contributor.author","Covic, Amra"],["dc.contributor.author","Backhaus, Joy C."],["dc.date.accessioned","2018-11-07T09:51:05Z"],["dc.date.available","2018-11-07T09:51:05Z"],["dc.date.issued","2015"],["dc.format.extent","14"],["dc.identifier.isi","000361991100040"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/35840"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.publisher.place","Dordrecht"],["dc.relation.issn","1573-2649"],["dc.relation.issn","0962-9343"],["dc.title","Patient-reported and performance-based outcomes in persons after mild traumatic brain"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","354"],["dc.bibliographiccitation.journal","Epilepsy & Behavior"],["dc.bibliographiccitation.lastpage","359"],["dc.bibliographiccitation.volume","80"],["dc.contributor.author","Nau, Anne L."],["dc.contributor.author","Mwape, Kabemba E."],["dc.contributor.author","Wiefek, Jasmin"],["dc.contributor.author","Schmidt, Kathie"],["dc.contributor.author","Abatih, Emmanuel"],["dc.contributor.author","Dorny, Pierre"],["dc.contributor.author","Praet, Nicolas"],["dc.contributor.author","Chiluba, Clarance"],["dc.contributor.author","Schmidt, Holger"],["dc.contributor.author","Phiri, Isaac K."],["dc.contributor.author","Winkler, Andrea S."],["dc.contributor.author","Gabriël, Sarah"],["dc.contributor.author","Blocher, Joachim"],["dc.date.accessioned","2020-12-10T15:21:46Z"],["dc.date.available","2020-12-10T15:21:46Z"],["dc.date.issued","2018"],["dc.identifier.doi","10.1016/j.yebeh.2017.10.042"],["dc.identifier.issn","1525-5050"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/73156"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","Cognitive impairment and quality of life of people with epilepsy and neurocysticercosis in Zambia"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","269-277"],["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","Oecologia"],["dc.bibliographiccitation.lastpage","277"],["dc.bibliographiccitation.volume","113"],["dc.contributor.author","Denys, Christine"],["dc.contributor.author","Schmidt, Holger"],["dc.date.accessioned","2020-10-07T04:58:16Z"],["dc.date.available","2020-10-07T04:58:16Z"],["dc.date.issued","1998-01"],["dc.description.abstract","We studied the ability of insect herbivores and their natural enemies to colonize exposed, potted mugwort plants (Artemisia vulgaris L.) along a rural-urban gradient in 1994 in Hamburg (northern Germany). Ectophagous insects, leafmines and galls were monitored weekly from mid-May to mid-September. Endophagous insects were counted by harvesting and dissecting the stems at the end of the growing season. The rural-urban gradient was characterized by a gradient of vegetation-free areas and increasing proportion of ground covered in concrete, tarmac, paving and other impermeable surfaces surrounding the Artemisia plots, i.e. six different zones of increasing isolation. Numbers of insect species (herbivores, parasitoids and predators) decreased along the gradient from 43 to 12. Monophagous herbivores were not more affected than polyphagous herbivores, but parasitoids, especially rare species, were more strongly affected by isolation than predators. Some dominant herbivorous species were very successful colonizers and occurred in inner city sites devoid of all natural vegetation. Sometimes their abundance increased in the inner city to significantly higher densities than in the urban fringe. Isolation appeared to be the main reason for the observed patterns, since area and soil conditions were held constant in the experiment. Microclimate and pollution were considered to play a minor role."],["dc.identifier.doi","10.1007/s004420050378"],["dc.identifier.pmid","28308207"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/68006"],["dc.language.iso","en"],["dc.relation.eissn","1432-1939"],["dc.relation.issn","0029-8549"],["dc.subject.gro","Urban ecology Isolation Species richness Mugwort Insect community"],["dc.title","Insect communities on experimental mugwort (Artemisia vulgaris L.) plots along an urban gradient"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.subtype","original_ja"],["dspace.entity.type","Publication"]]