Belle, Gijsbert Jacob Christiaan van

[["dc.bibliographiccitation.artnumber","jcs223230"],["dc.bibliographiccitation.issue","5"],["dc.bibliographiccitation.journal","Journal of Cell Science"],["dc.bibliographiccitation.volume","132"],["dc.contributor.author","Leinhos, Lisa"],["dc.contributor.author","Peters, Johannes"],["dc.contributor.author","Krull, Sabine"],["dc.contributor.author","Helbig, Lena"],["dc.contributor.author","Vogler, Melanie"],["dc.contributor.author","Levay, Magdolna"],["dc.contributor.author","van Belle, Gijsbert J."],["dc.contributor.author","Ridley, Anne J."],["dc.contributor.author","Lutz, Susanne"],["dc.contributor.author","Katschinski, Dörthe M."],["dc.contributor.author","Zieseniss, Anke"],["dc.date.accessioned","2020-12-10T18:41:53Z"],["dc.date.available","2020-12-10T18:41:53Z"],["dc.date.issued","2019"],["dc.identifier.doi","10.1242/jcs.223230"],["dc.identifier.eissn","1477-9137"],["dc.identifier.issn","0021-9533"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/77715"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","Hypoxia suppresses myofibroblast differentiation by changing RhoA activity"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.issue","12"],["dc.bibliographiccitation.journal","Progress of Theoretical and Experimental Physics"],["dc.bibliographiccitation.volume","2019"],["dc.contributor.author","Kou, E"],["dc.contributor.author","Urquijo, P"],["dc.contributor.author","Altmannshofer, W"],["dc.contributor.author","Beaujean, F"],["dc.contributor.author","Bell, G"],["dc.contributor.author","Beneke, M"],["dc.contributor.author","Bigi, I I"],["dc.contributor.author","Bishara, F"],["dc.contributor.author","Blanke, M"],["dc.contributor.author","Bobeth, C"],["dc.contributor.author","Zupanc, A"],["dc.date.accessioned","2020-12-10T18:19:40Z"],["dc.date.available","2020-12-10T18:19:40Z"],["dc.date.issued","2019"],["dc.identifier.doi","10.1093/ptep/ptz106"],["dc.identifier.eissn","2050-3911"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/75330"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","The Belle II Physics Book"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","101071"],["dc.bibliographiccitation.journal","Redox Biology"],["dc.bibliographiccitation.volume","21"],["dc.contributor.author","Shokhina, Arina G."],["dc.contributor.author","Kostyuk, Alexander I."],["dc.contributor.author","Ermakova, Yulia G."],["dc.contributor.author","Panova, Anastasiya S."],["dc.contributor.author","Staroverov, Dmitry B."],["dc.contributor.author","Egorov, Evgeny S."],["dc.contributor.author","Baranov, Mikhail S."],["dc.contributor.author","van Belle, Gijsbert J."],["dc.contributor.author","Katschinski, Dörthe M."],["dc.contributor.author","Belousov, Vsevolod V."],["dc.contributor.author","Bilan, Dmitry S."],["dc.date.accessioned","2020-12-10T15:21:03Z"],["dc.date.available","2020-12-10T15:21:03Z"],["dc.date.issued","2019"],["dc.identifier.doi","10.1016/j.redox.2018.101071"],["dc.identifier.issn","2213-2317"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/72897"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","Red fluorescent redox-sensitive biosensor Grx1-roCherry"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","716"],["dc.bibliographiccitation.issue","7"],["dc.bibliographiccitation.journal","Journal of Neurology Neurosurgery & Psychiatry"],["dc.bibliographiccitation.lastpage","718"],["dc.bibliographiccitation.volume","81"],["dc.contributor.author","Bell, G. S."],["dc.contributor.author","Sinha, S."],["dc.contributor.author","de Tisi, J."],["dc.contributor.author","Stephani, Caspar"],["dc.contributor.author","Scott, Catherine"],["dc.contributor.author","Harkness, W. F."],["dc.contributor.author","McEvoy, A. W."],["dc.contributor.author","Peacock, J. L."],["dc.contributor.author","Walker, M. C."],["dc.contributor.author","Smith, S. J."],["dc.contributor.author","Duncan, John S."],["dc.contributor.author","Sander, Josemir W."],["dc.date.accessioned","2018-11-07T08:41:48Z"],["dc.date.available","2018-11-07T08:41:48Z"],["dc.date.issued","2010"],["dc.description.abstract","Background Epilepsy carries an increased risk of premature death. For some people with intractable focal epilepsy, surgery offers hope for a seizure-free life. The authors aimed to see whether epilepsy surgery influenced mortality in people with intractable epilepsy. Methods The authors audited survival status in two cohorts (those who had surgery and those who had presurgical assessment but did not have surgery). Results There were 40 known deaths in the non-surgical group (3365 person years of follow-up) and 19 in the surgical group (3905 person-years of follow-up). Non-operated patients were 2.4 times (95% CI 1.4 to 4.2) as likely to die as those who had surgery. They were 4.5 times (95% CI 1.9 to 10.9) as likely to die a probable epilepsy-related death. In the surgical group, those with ongoing seizures 1 year after surgery were 4.0 (95% CI 1.2 to 13.7) times as likely to die as those who were seizure-free or who had only simple partial seizures. Time-dependent Cox analysis showed that the yearly outcome group did not significantly affect mortality (HR 1.3, 95% CI 0.9 to 1.8). Conclusion Successful epilepsy surgery was associated with a reduced risk of premature mortality, compared with those with refractory focal epilepsy who did not have surgical treatment. To some extent, the reduced mortality is likely to be conferred by inducing freedom from seizures. It is not certain whether better survival is attributable only to surgery, as treatment decisions were not randomised, and there may be inherent differences between the groups."],["dc.identifier.doi","10.1136/jnnp.2008.170837"],["dc.identifier.isi","000280040800005"],["dc.identifier.pmid","20478848"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/19551"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","B M J Publishing Group"],["dc.relation.issn","0022-3050"],["dc.title","Premature mortality in refractory partial epilepsy: does surgical treatment make a difference?"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","Progress of Theoretical and Experimental Physics"],["dc.bibliographiccitation.volume","2020"],["dc.contributor.author","Kou, E"],["dc.contributor.author","Urquijo, P"],["dc.contributor.author","Altmannshofer, W"],["dc.contributor.author","Beaujean, F"],["dc.contributor.author","Bell, G"],["dc.contributor.author","Beneke, M"],["dc.contributor.author","Bigi, I I"],["dc.contributor.author","Bishara, F"],["dc.contributor.author","Blanke, M"],["dc.contributor.author","Bobeth, C"],["dc.contributor.author","Zupanc, A"],["dc.date.accessioned","2020-12-10T18:19:40Z"],["dc.date.available","2020-12-10T18:19:40Z"],["dc.date.issued","2020"],["dc.identifier.doi","10.1093/ptep/ptaa008"],["dc.identifier.eissn","2050-3911"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/75329"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","The Belle II Physics Book"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","59"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Acta physiologica"],["dc.bibliographiccitation.lastpage","73"],["dc.bibliographiccitation.volume","221"],["dc.contributor.author","Wottawa, M."],["dc.contributor.author","Naas, S."],["dc.contributor.author","Böttger, J."],["dc.contributor.author","van Belle, G. J."],["dc.contributor.author","Möbius, W."],["dc.contributor.author","Revelo, Natalia H."],["dc.contributor.author","Heidenreich, D."],["dc.contributor.author","von Ahlen, M."],["dc.contributor.author","Zieseniss, A."],["dc.contributor.author","Kröhnert, K."],["dc.contributor.author","Lutz, S."],["dc.contributor.author","Lenz, C."],["dc.contributor.author","Urlaub, H."],["dc.contributor.author","Rizzoli, S. O."],["dc.contributor.author","Katschinski, D. M."],["dc.date.accessioned","2018-01-09T16:05:27Z"],["dc.date.available","2018-01-09T16:05:27Z"],["dc.date.issued","2017"],["dc.description.abstract","Traffic between the plasma membrane and the endomembrane compartments is an essential feature of eukaryotic cells. The secretory pathway sends cargoes from biosynthetic compartments to the plasma membrane. This is counterbalanced by a retrograde endocytic route and is essential for cell homoeostasis. Cells need to adapt rapidly to environmental challenges such as the reduction of pO2 which, however, has not been analysed in relation to membrane trafficking in detail. Therefore, we determined changes in the plasma membrane trafficking in normoxia, hypoxia, and after reoxygenation."],["dc.identifier.doi","10.1111/apha.12859"],["dc.identifier.pmid","28218996"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/11620"],["dc.language.iso","en"],["dc.notes.status","final"],["dc.relation.eissn","1748-1716"],["dc.title","Hypoxia-stimulated membrane trafficking requires T-plastin"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dspace.entity.type","Publication"]]