Arnim, Christine A. F. von

[["dc.bibliographiccitation.artnumber","e0266906"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","PLoS One"],["dc.bibliographiccitation.volume","17"],["dc.contributor.author","Yousefzadeh-Nowshahr, Elham"],["dc.contributor.author","Winter, Gordon"],["dc.contributor.author","Bohn, Peter"],["dc.contributor.author","Kneer, Katharina"],["dc.contributor.author","von Arnim, Christine A. F."],["dc.contributor.author","Otto, Markus"],["dc.contributor.author","Solbach, Christoph"],["dc.contributor.author","Anderl-Straub, Sarah"],["dc.contributor.author","Polivka, Dörte"],["dc.contributor.author","Fissler, Patrick"],["dc.contributor.authorgroup","for the Alzheimer’s Disease Neuroimaging Initiative"],["dc.contributor.editor","Su, Yi"],["dc.date.accessioned","2022-05-02T08:09:28Z"],["dc.date.available","2022-05-02T08:09:28Z"],["dc.date.issued","2022"],["dc.description.abstract","Purpose The recent developments of tau-positron emission tomography (tau-PET) enable in vivo assessment of neuropathological tau aggregates. Among the tau-specific tracers, the application of 11 C-pyridinyl-butadienyl-benzothiazole 3 ( 11 C-PBB3) in PET shows high sensitivity to Alzheimer disease (AD)-related tau deposition. The current study investigates the regional tau load in patients within the AD continuum, biomarker-negative individuals (BN) and patients with suspected non-AD pathophysiology (SNAP) using 11 C-PBB3-PET. Materials and methods A total of 23 memory clinic outpatients with recent decline of episodic memory were examined using 11 C-PBB3-PET. Pittsburg compound B ( 11 C-PIB) PET was available for 17, 18 F-flurodeoxyglucose ( 18 F-FDG) PET for 16, and cerebrospinal fluid (CSF) protein levels for 11 patients. CSF biomarkers were considered abnormal based on Aβ 42 (< 600 ng/L) and t-tau (> 450 ng/L). The PET biomarkers were classified as positive or negative using statistical parametric mapping (SPM) analysis and visual assessment. Using the amyloid/tau/neurodegeneration (A/T/N) scheme, patients were grouped as within the AD continuum, SNAP, and BN based on amyloid and neurodegeneration status. The 11 C-PBB3 load detected by PET was compared among the groups using both atlas-based and voxel-wise analyses. Results Seven patients were identified as within the AD continuum, 10 SNAP and 6 BN. In voxel-wise analysis, significantly higher 11 C-PBB3 binding was observed in the AD continuum group compared to the BN patients in the cingulate gyrus, tempo-parieto-occipital junction and frontal lobe. Compared to the SNAP group, patients within the AD continuum had a considerably increased 11 C-PBB3 uptake in the posterior cingulate cortex. There was no significant difference between SNAP and BN groups. The atlas-based analysis supported the outcome of the voxel-wise quantification analysis. Conclusion Our results suggest that 11 C-PBB3-PET can effectively analyze regional tau load and has the potential to differentiate patients in the AD continuum group from the BN and SNAP group."],["dc.description.abstract","Purpose The recent developments of tau-positron emission tomography (tau-PET) enable in vivo assessment of neuropathological tau aggregates. Among the tau-specific tracers, the application of 11 C-pyridinyl-butadienyl-benzothiazole 3 ( 11 C-PBB3) in PET shows high sensitivity to Alzheimer disease (AD)-related tau deposition. The current study investigates the regional tau load in patients within the AD continuum, biomarker-negative individuals (BN) and patients with suspected non-AD pathophysiology (SNAP) using 11 C-PBB3-PET. Materials and methods A total of 23 memory clinic outpatients with recent decline of episodic memory were examined using 11 C-PBB3-PET. Pittsburg compound B ( 11 C-PIB) PET was available for 17, 18 F-flurodeoxyglucose ( 18 F-FDG) PET for 16, and cerebrospinal fluid (CSF) protein levels for 11 patients. CSF biomarkers were considered abnormal based on Aβ 42 (< 600 ng/L) and t-tau (> 450 ng/L). The PET biomarkers were classified as positive or negative using statistical parametric mapping (SPM) analysis and visual assessment. Using the amyloid/tau/neurodegeneration (A/T/N) scheme, patients were grouped as within the AD continuum, SNAP, and BN based on amyloid and neurodegeneration status. The 11 C-PBB3 load detected by PET was compared among the groups using both atlas-based and voxel-wise analyses. Results Seven patients were identified as within the AD continuum, 10 SNAP and 6 BN. In voxel-wise analysis, significantly higher 11 C-PBB3 binding was observed in the AD continuum group compared to the BN patients in the cingulate gyrus, tempo-parieto-occipital junction and frontal lobe. Compared to the SNAP group, patients within the AD continuum had a considerably increased 11 C-PBB3 uptake in the posterior cingulate cortex. There was no significant difference between SNAP and BN groups. The atlas-based analysis supported the outcome of the voxel-wise quantification analysis. Conclusion Our results suggest that 11 C-PBB3-PET can effectively analyze regional tau load and has the potential to differentiate patients in the AD continuum group from the BN and SNAP group."],["dc.identifier.doi","10.1371/journal.pone.0266906"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/107385"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-561"],["dc.relation.eissn","1932-6203"],["dc.rights.uri","http://creativecommons.org/licenses/by/4.0/"],["dc.title","Quantitative analysis of regional distribution of tau pathology with 11C-PBB3-PET in a clinical setting"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","157"],["dc.bibliographiccitation.issue","04"],["dc.bibliographiccitation.journal","DMW - Deutsche Medizinische Wochenschrift"],["dc.bibliographiccitation.lastpage","164"],["dc.bibliographiccitation.volume","147"],["dc.contributor.author","Kirsch, Mareike"],["dc.contributor.author","von Arnim, Christine"],["dc.date.accessioned","2022-04-01T10:00:42Z"],["dc.date.available","2022-04-01T10:00:42Z"],["dc.date.issued","2022"],["dc.identifier.doi","10.1055/a-1593-8205"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/105490"],["dc.language.iso","de"],["dc.notes.intern","DOI-Import GROB-530"],["dc.relation.eissn","1439-4413"],["dc.relation.issn","0012-0472"],["dc.title","Medikamentöse Therapie der Demenz"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.artnumber","alz.12794"],["dc.bibliographiccitation.journal","Alzheimer's & Dementia"],["dc.contributor.author","Schaeverbeke, Jolien"],["dc.contributor.author","Tomé, Sandra O."],["dc.contributor.author","Ronisz, Alicja"],["dc.contributor.author","Ospitalieri, Simona"],["dc.contributor.author","von Arnim, Christine A. F."],["dc.contributor.author","Otto, Markus"],["dc.contributor.author","Vandenberghe, Rik"],["dc.contributor.author","Thal, Dietmar Rudolf"],["dc.date.accessioned","2022-10-04T10:21:57Z"],["dc.date.available","2022-10-04T10:21:57Z"],["dc.date.issued","2022"],["dc.identifier.doi","10.1002/alz.12794"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/114547"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-600"],["dc.relation.eissn","1552-5279"],["dc.relation.issn","1552-5260"],["dc.title","Neuronal loss of the nucleus basalis of Meynert in primary progressive aphasia is associated with Alzheimer's disease neuropathological changes"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Alzheimer's Research & Therapy"],["dc.bibliographiccitation.volume","14"],["dc.contributor.author","Halbgebauer, Steffen"],["dc.contributor.author","Steinacker, Petra"],["dc.contributor.author","Riedel, Daniel"],["dc.contributor.author","Oeckl, Patrick"],["dc.contributor.author","Anderl-Straub, Sarah"],["dc.contributor.author","Lombardi, Jolina"],["dc.contributor.author","von Arnim, Christine A. F."],["dc.contributor.author","Nagl, Magdalena"],["dc.contributor.author","Giese, Armin"],["dc.contributor.author","Ludolph, Albert C."],["dc.contributor.author","Otto, Markus"],["dc.date.accessioned","2022-12-01T08:31:24Z"],["dc.date.available","2022-12-01T08:31:24Z"],["dc.date.issued","2022"],["dc.description.abstract","Abstract\n \n Background\n Visinin-like protein 1 (VILIP-1) belongs to the group of emerging biomarkers with the potential to support the early diagnosis of Alzheimer’s disease (AD). However, studies investigating the differential diagnostic potential in cerebrospinal fluid (CSF) are rare and are not available for blood.\n \n \n Methods\n We set up a novel, sensitive single molecule array (Simoa) assay for the detection of VILIP-1 in CSF and serum. In total, paired CSF and serum samples from 234 patients were investigated: 73 AD, 18 behavioral variant frontotemporal dementia (bvFTD), 26 parkinsonian syndromes, 20 amyotrophic lateral sclerosis (ALS), 22 Creutzfeldt-Jakob disease (CJD), and 75 non-neurodegenerative control (Con) patients. The differential diagnostic potential of CSF and serum VILIP-1 was assessed using the receiver operating characteristic curve analysis and findings were compared to core AD biomarkers.\n \n \n Results\n \n CSF and serum VILIP-1 levels correlated weakly (\n r\n =0.32 (CI: 0.20–0.43),\n p\n <0.0001). VILIP-1 concentrations in CSF and serum were elevated in AD compared to Con (\n p\n <0.0001 and\n p\n <0.01) and CJD (\n p\n <0.0001 for CSF and serum), and an increase in CSF was observed already in early AD stages (\n p\n <0.0001). In the discrimination of AD versus Con, we could demonstrate a strong diagnostic potential for CSF VILIP-1 alone (area under the curve (AUC): 0.87), CSF VILIP-1/CSF Abeta 1-42 (AUC: 0.98), and serum VILIP-1/CSF Abeta 1-42 ratio (AUC: 0.89).\n \n \n \n Conclusions\n We here report on the successful establishment of a novel Simoa assay for VILIP-1 and illustrate the potential of CSF and serum VILIP-1 in the differential diagnosis of AD with highest levels in CJD."],["dc.description.sponsorship","Intramural funding University of Ulm"],["dc.description.sponsorship","EU Joint Programme-Neurodegenerative Diseases networks Genfi-Prox"],["dc.description.sponsorship","German Federal Ministry of Education and Research"],["dc.description.sponsorship","EU (Moodmarker) program"],["dc.description.sponsorship","German Research Foundation/DFG"],["dc.description.sponsorship","Foundation of the state Baden-Württemberg"],["dc.description.sponsorship","Boehringer Ingelheim Ulm University BioCenter"],["dc.description.sponsorship","Thierry Latran Foundation"],["dc.description.sponsorship","Martin-Luther-Universität Halle-Wittenberg"],["dc.identifier.doi","10.1186/s13195-022-01122-4"],["dc.identifier.pii","1122"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/118163"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-621"],["dc.relation.eissn","1758-9193"],["dc.rights.uri","https://creativecommons.org/licenses/by/4.0"],["dc.title","Visinin-like protein 1 levels in blood and CSF as emerging markers for Alzheimer’s and other neurodegenerative diseases"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","340"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","Zeitschrift für Gerontologie und Geriatrie"],["dc.bibliographiccitation.lastpage","346"],["dc.bibliographiccitation.volume","53"],["dc.contributor.author","Jacobs, Andreas H."],["dc.contributor.author","Emmert, Kirsten"],["dc.contributor.author","Baron, Ralf"],["dc.contributor.author","Bartsch, Thorsten"],["dc.contributor.author","Bauer, Juergen"],["dc.contributor.author","Becker, Clemens"],["dc.contributor.author","Berg, Daniela"],["dc.contributor.author","Bergmann, Philipp"],["dc.contributor.author","Boetzel, Kai"],["dc.contributor.author","Bollheimer, Cornelius"],["dc.contributor.author","Deuschl, Guenther"],["dc.contributor.author","Djukic, Marija"],["dc.contributor.author","Drey, Michael"],["dc.contributor.author","Durwen, Herbert"],["dc.contributor.author","Ebersbach, Georg"],["dc.contributor.author","Elshehabi, Morad"],["dc.contributor.author","Geritz, Johanna"],["dc.contributor.author","Gisinger, Christoph"],["dc.contributor.author","Guennewig, Thomas"],["dc.contributor.author","Hauptmann, Bjoern"],["dc.contributor.author","Heppner, Hans-Juergen"],["dc.contributor.author","Hobert, Markus A."],["dc.contributor.author","Hofmann, Werner"],["dc.contributor.author","Huellemann, Philipp"],["dc.contributor.author","Jahn, Klaus"],["dc.contributor.author","Klucken, Jochen"],["dc.contributor.author","Kurth, Roland"],["dc.contributor.author","Lindner, Reinhard"],["dc.contributor.author","Lingor, Paul"],["dc.contributor.author","Lukas, Albert"],["dc.contributor.author","Maetzold, Sara"],["dc.contributor.author","Mokrusch, Thomas"],["dc.contributor.author","Mollenhauer, Brit"],["dc.contributor.author","Nau, Roland"],["dc.contributor.author","Plate, Annika"],["dc.contributor.author","Polidori, Maria Cristina"],["dc.contributor.author","Prell, Tino"],["dc.contributor.author","Schellinger, Peter"],["dc.contributor.author","Spira, Dominik"],["dc.contributor.author","Stephani, Ulrich"],["dc.contributor.author","Studt, Simone"],["dc.contributor.author","Trenkwalder, Claudia"],["dc.contributor.author","Unger, Heinz L."],["dc.contributor.author","Urban, Peter"],["dc.contributor.author","von Arnim, Christine A. F."],["dc.contributor.author","Warnecke, Tobias"],["dc.contributor.author","Weiss, Michael"],["dc.contributor.author","Wiedemann, Andreas"],["dc.contributor.author","Wirth, Rainer"],["dc.contributor.author","Witt, Karsten"],["dc.contributor.author","Dodel, Richard"],["dc.contributor.author","Maetzler, Walter"],["dc.date.accessioned","2020-12-10T14:10:21Z"],["dc.date.available","2020-12-10T14:10:21Z"],["dc.date.issued","2020"],["dc.identifier.doi","10.1007/s00391-020-01734-1"],["dc.identifier.eissn","1435-1269"],["dc.identifier.issn","0948-6704"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/70733"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","Neurogeriatrie – eine Vision für die verbesserte Versorgung und Forschung für geriatrische Patienten mit führend neurologischen Erkrankungen"],["dc.title.alternative","Neurogeriatrics—a vision for improved care and research for geriatric patients with predominating neurological disabilities"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","e216370"],["dc.bibliographiccitation.journal","JAMA Surgery"],["dc.contributor.author","Deeken, Friederike"],["dc.contributor.author","Sánchez, Alba"],["dc.contributor.author","Rapp, Michael A."],["dc.contributor.author","Denkinger, Michael"],["dc.contributor.author","Brefka, Simone"],["dc.contributor.author","Spank, Juliane"],["dc.contributor.author","Bruns, Carola"],["dc.contributor.author","von Arnim, Christine A. F."],["dc.contributor.author","Küster, Olivia C."],["dc.contributor.author","Conzelmann, Lars O."],["dc.contributor.author","Fichtner-Feigl, Stefan"],["dc.contributor.authorgroup","PAWEL Study Group"],["dc.date.accessioned","2022-01-11T14:07:59Z"],["dc.date.available","2022-01-11T14:07:59Z"],["dc.date.issued","2021"],["dc.identifier.doi","10.1001/jamasurg.2021.6370"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/97905"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-507"],["dc.relation.issn","2168-6254"],["dc.title","Outcomes of a Delirium Prevention Program in Older Persons After Elective Surgery"],["dc.title.alternative","A Stepped-Wedge Cluster Randomized Clinical Trial"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.journal","Frontiers in Aging Neuroscience"],["dc.bibliographiccitation.volume","13"],["dc.contributor.author","Laptinskaya, Daria"],["dc.contributor.author","Küster, Olivia Caroline"],["dc.contributor.author","Fissler, Patrick"],["dc.contributor.author","Thurm, Franka"],["dc.contributor.author","Von Arnim, Christine A. F."],["dc.contributor.author","Kolassa, Iris-Tatjana"],["dc.date.accessioned","2021-06-01T09:42:25Z"],["dc.date.available","2021-06-01T09:42:25Z"],["dc.date.issued","2021"],["dc.description.abstract","An active lifestyle as well as cognitive and physical training (PT) may benefit cognition by increasing cognitive reserve, but the underlying neurobiological mechanisms of this reserve capacity are not well understood. To investigate these mechanisms of cognitive reserve, we focused on electrophysiological correlates of cognitive performance, namely on an event-related measure of auditory memory and on a measure of global coherence. Both measures have shown to be sensitive markers for cognition and might therefore be suitable to investigate potential training- and lifestyle-related changes. Here, we report on the results of an electrophysiological sub-study that correspond to previously published behavioral findings. Altogether, 65 older adults with subjective or objective cognitive impairment and aged 60–88 years were assigned to a 10-week cognitive ( n = 19) or a 10-week PT ( n = 21) or to a passive control group ( n = 25). In addition, self-reported lifestyle was assessed at baseline. We did not find an effect of both training groups on electroencephalography (EEG) measures of auditory memory decay or global coherence ( p s ≥ 0.29) and a more active lifestyle was not associated with improved global coherence ( p = 0.38). Results suggest that a 10-week unimodal cognitive or PT and an active lifestyle in older adults at risk for dementia are not strongly related to improvements in electrophysiological correlates of cognition."],["dc.identifier.doi","10.3389/fnagi.2021.610839"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/85246"],["dc.notes.intern","DOI-Import GROB-425"],["dc.relation.eissn","1663-4365"],["dc.title","No Evidence That Cognitive and Physical Activities Are Related to Changes in EEG Markers of Cognition in Older Adults at Risk of Dementia"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.journal","Zeitschrift für Gerontologie und Geriatrie"],["dc.contributor.author","Frölich, Lutz"],["dc.contributor.author","von Arnim, Christine"],["dc.contributor.author","Bohlken, Jens"],["dc.contributor.author","Pantel, Johannes"],["dc.contributor.author","Peters, Oliver"],["dc.contributor.author","Förstl, Hans"],["dc.date.accessioned","2022-10-04T10:22:03Z"],["dc.date.available","2022-10-04T10:22:03Z"],["dc.date.issued","2022"],["dc.identifier.doi","10.1007/s00391-022-02114-7"],["dc.identifier.pii","2114"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/114575"],["dc.language.iso","de"],["dc.notes.intern","DOI-Import GROB-600"],["dc.relation.eissn","1435-1269"],["dc.relation.iserratumof","/handle/2/113982"],["dc.relation.issn","0948-6704"],["dc.rights.uri","https://www.springer.com/tdm"],["dc.title","Erratum zu: Leichte kognitive Beeinträchtigung (MCI) in der altersmedizinischen Praxis: Patientenorientierung, Diagnostik, Behandlung und Ethik"],["dc.title.translated","Erratum to: Mild cognitive impairment in geriatric practice: patient orientation, diagnostics, treatment and ethics"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.subtype","erratum_ja"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","173"],["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","Acta Neuropathologica"],["dc.bibliographiccitation.lastpage","192"],["dc.bibliographiccitation.volume","141"],["dc.contributor.author","Aragão Gomes, Luis"],["dc.contributor.author","Uytterhoeven, Valerie"],["dc.contributor.author","Lopez-Sanmartin, Diego"],["dc.contributor.author","Tomé, Sandra O."],["dc.contributor.author","Tousseyn, Thomas"],["dc.contributor.author","Vandenberghe, Rik"],["dc.contributor.author","Vandenbulcke, Mathieu"],["dc.contributor.author","von Arnim, Christine A. F."],["dc.contributor.author","Verstreken, Patrik"],["dc.contributor.author","Thal, Dietmar Rudolf"],["dc.date.accessioned","2021-04-14T08:30:40Z"],["dc.date.available","2021-04-14T08:30:40Z"],["dc.date.issued","2021"],["dc.identifier.doi","10.1007/s00401-020-02251-6"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/83332"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-399"],["dc.relation.eissn","1432-0533"],["dc.relation.issn","0001-6322"],["dc.title","Maturation of neuronal AD-tau pathology involves site-specific phosphorylation of cytoplasmic and synaptic tau preceding conformational change and fibril formation"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.journal","Journal of Neurochemistry"],["dc.bibliographiccitation.volume","138"],["dc.contributor.author","Straub, S."],["dc.contributor.author","Schroeter, M. L."],["dc.contributor.author","Diehl-Schmid, Janine"],["dc.contributor.author","Danek, A."],["dc.contributor.author","von Arnim, Christine A. F."],["dc.contributor.author","Bertram, L."],["dc.contributor.author","Einsiedler, B."],["dc.contributor.author","Fassbender, Klaus"],["dc.contributor.author","Feneberg, Emily"],["dc.contributor.author","Fliessbach, Klaus"],["dc.contributor.author","Foerstl, Hans"],["dc.contributor.author","Huppertz, H-J"],["dc.contributor.author","Jahn, Holger"],["dc.contributor.author","Jessen, Frank"],["dc.contributor.author","Kasper, E."],["dc.contributor.author","Kassubek, Jan"],["dc.contributor.author","Kestler, Hans A."],["dc.contributor.author","Kornhuber, Johannes"],["dc.contributor.author","Kubisch, C."],["dc.contributor.author","Landwehrmeyer, Bernhard"],["dc.contributor.author","Lauer, Martin"],["dc.contributor.author","Lausser, L."],["dc.contributor.author","Lill, C. M."],["dc.contributor.author","Ludolph, A."],["dc.contributor.author","Maler, M."],["dc.contributor.author","Muche, Rainer"],["dc.contributor.author","Pinkhardt, Elmar H."],["dc.contributor.author","Prudlo, Johannes"],["dc.contributor.author","Riedl, Lina"],["dc.contributor.author","Schneider, Anja"],["dc.contributor.author","Schomburg, R."],["dc.contributor.author","Teipel, Stefan J."],["dc.contributor.author","Uttner, Ingo"],["dc.contributor.author","Volk, Alexander E."],["dc.contributor.author","Otto, Markus"],["dc.date.accessioned","2018-11-07T10:10:49Z"],["dc.date.available","2018-11-07T10:10:49Z"],["dc.date.issued","2016"],["dc.format.extent","407"],["dc.identifier.isi","000382568400474"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/39933"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Wiley-blackwell"],["dc.publisher.place","Hoboken"],["dc.relation.conference","10th International Conference on Frontotemporal Dementias"],["dc.relation.eventlocation","Munich, GERMANY"],["dc.relation.issn","1471-4159"],["dc.relation.issn","0022-3042"],["dc.title","Multivariate diagnostic approaches in Frontotemporal lobar degeneration Data from the german FTLDc"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]