Havemann-Reinecke, Ursula

[["dc.bibliographiccitation.journal","European Psychiatry"],["dc.bibliographiccitation.volume","22"],["dc.contributor.author","Havemann-Reinecke, Ursula"],["dc.contributor.author","Hein, C."],["dc.contributor.author","Gorski, S."],["dc.contributor.author","Wurche-Gier, M."],["dc.contributor.author","Jacobs, S."],["dc.contributor.author","Schneider, Udo"],["dc.date.accessioned","2018-11-07T11:04:21Z"],["dc.date.available","2018-11-07T11:04:21Z"],["dc.date.issued","2007"],["dc.format.extent","S42"],["dc.identifier.doi","10.1016/j.eurpsy.2007.01.168"],["dc.identifier.isi","000245473000153"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/51824"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Elsevier France-editions Scientifiques Medicales Elsevier"],["dc.publisher.place","Paris"],["dc.relation.issn","0924-9338"],["dc.title","Psychiatric comorbidity of dependent tobacco smokers"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1"],["dc.bibliographiccitation.issue","12"],["dc.bibliographiccitation.journal","German Journal of Psychiatry"],["dc.bibliographiccitation.lastpage","7"],["dc.contributor.author","Wedekind, Dirk"],["dc.contributor.author","Jacobs, Stefan"],["dc.contributor.author","Poser, Wolfgang"],["dc.contributor.author","Rüther, Eckart"],["dc.contributor.author","Schneider, Udo"],["dc.contributor.author","Cimander, Konrad"],["dc.contributor.author","Engel, Kirsten"],["dc.contributor.author","Havemann-Reinecke, Ursula"],["dc.date.accessioned","2019-07-10T08:13:27Z"],["dc.date.available","2019-07-10T08:13:27Z"],["dc.date.issued","2009"],["dc.description.abstract","Objective: Previous reports on heroin and cocaine addicts showed drug-related and gender differences in psychiatric comorbidity, which has relevant consequences for treatment. However, studies vary substantially with respect to methods and timeframes. Studies on German patient groups are scarce. Methods: Data on psychiatric and somatic comorbidity, substance addiction history, present intake patterns and sociodemography were obtained from 43 female (n=11) and male (n=32) heroin and cocaine addicts in acute inpatient detoxification treatment or specified long-term treatment. A European Addiction-Severity-Index (EuropASI) based centre questionnaire and the Mini-DIPS were applied. Results: Treatment groups did not differ in psychiatric comorbidity. Female subjects, however, had a significantly higher prevalence of psychiatric comorbid diagnoses (p<.05), mostly anxiety and affective disorders which significantly correlated with low occupational status (p<.05).Patients in long-term treatment abused more other substances and had an earlier onset of regular substance abuse (in particular alcohol and cannabis) (p<.05). Conclusion: Heroin and cocaine addicted females are more likely than males to have affective and anxiety disorders. Long-term treatment attenders appear to be more severely addicted (earlier onset and additional abuse) than acute treatment patients but do not differ in comorbidity. However, no axis-II diagnoses were recorded and the sample-size was small. Results should be regarded as preliminary (German J Psychiatry 2009; 12: 1-7)."],["dc.identifier.fs","541930"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/5950"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/61249"],["dc.language.iso","en"],["dc.notes.intern","Merged from goescholar"],["dc.relation.issn","1455-1033"],["dc.relation.orgunit","Universitätsmedizin Göttingen"],["dc.rights","Goescholar"],["dc.rights.uri","https://goescholar.uni-goettingen.de/licenses"],["dc.subject.ddc","610"],["dc.title","Psychiatric Comorbidity and Gender Effects in Heroin and Cocaine-Addicted Patients in Specified Long-Term Treatment and Acute Inpatient Detoxification Treatment"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.artnumber","91"],["dc.bibliographiccitation.journal","BMC Psychiatry"],["dc.bibliographiccitation.volume","10"],["dc.contributor.author","Ribbe, Katja"],["dc.contributor.author","Friedrichs, Heidi"],["dc.contributor.author","Begemann, Martin"],["dc.contributor.author","Grube, Sabrina"],["dc.contributor.author","Papiol, Sergi"],["dc.contributor.author","Kästner, Anne"],["dc.contributor.author","Gerchen, Martin Fungisai"],["dc.contributor.author","Ackermann, Verena"],["dc.contributor.author","Tarami, Asieh"],["dc.contributor.author","Treitz, Annika"],["dc.contributor.author","Flögel, Marlene"],["dc.contributor.author","Adler, Lothar"],["dc.contributor.author","Aldenhoff, Josef B."],["dc.contributor.author","Becker-Emner, Marianne"],["dc.contributor.author","Becker, Thomas"],["dc.contributor.author","Czernik, Adelheid"],["dc.contributor.author","Dose, Matthias"],["dc.contributor.author","Folkerts, Here"],["dc.contributor.author","Freese, Roland"],["dc.contributor.author","Guenther, Rolf"],["dc.contributor.author","Herpertz, Sabine"],["dc.contributor.author","Hesse, Dirk"],["dc.contributor.author","Kruse, Gunther"],["dc.contributor.author","Kunze, Heinrich"],["dc.contributor.author","Franz, Michael"],["dc.contributor.author","Lohrer, Frank"],["dc.contributor.author","Maier, Wolfgang"],["dc.contributor.author","Mielke, Andreas"],["dc.contributor.author","Müller-Isberner, Rüdiger"],["dc.contributor.author","Oestereich, Cornelia"],["dc.contributor.author","Pajonk, Frank-Gerald"],["dc.contributor.author","Pollmächer, Thomas"],["dc.contributor.author","Schneider, Udo"],["dc.contributor.author","Schwarz, Hans-Joachim"],["dc.contributor.author","Kröner-Herwig, Birgit"],["dc.contributor.author","Havemann-Reinecke, Ursula"],["dc.contributor.author","Frahm, Jens"],["dc.contributor.author","Stühmer, Walter"],["dc.contributor.author","Falkai, Peter"],["dc.contributor.author","Brose, Nils"],["dc.contributor.author","Nave, Klaus-Armin"],["dc.contributor.author","Ehrenreich, Hannelore"],["dc.date.accessioned","2017-09-07T11:46:37Z"],["dc.date.available","2017-09-07T11:46:37Z"],["dc.date.issued","2010"],["dc.description.abstract","Background: Schizophrenia is the collective term for an exclusively clinically diagnosed, heterogeneous group of mental disorders with still obscure biological roots. Based on the assumption that valuable information about relevant genetic and environmental disease mechanisms can be obtained by association studies on patient cohorts of ≥ 1000 patients, if performed on detailed clinical datasets and quantifiable biological readouts, we generated a new schizophrenia data base, the GRAS (Göttingen Research Association for Schizophrenia) data collection. GRAS is the necessary ground to study genetic causes of the schizophrenic phenotype in a 'phenotype-based genetic association study' (PGAS). This approach is different from and complementary to the genome-wide association studies (GWAS) on schizophrenia. Methods: For this purpose, 1085 patients were recruited between 2005 and 2010 by an invariable team of traveling investigators in a cross-sectional field study that comprised 23 German psychiatric hospitals. Additionally, chart records and discharge letters of all patients were collected. Results: The corresponding dataset extracted and presented in form of an overview here, comprises biographic information, disease history, medication including side effects, and results of comprehensive cross-sectional psychopathological, neuropsychological, and neurological examinations. With >3000 data points per schizophrenic subject, this data base of living patients, who are also accessible for follow-up studies, provides a wide-ranging and standardized phenotype characterization of as yet unprecedented detail. Conclusions: The GRAS data base will serve as prerequisite for PGAS, a novel approach to better understanding 'the schizophrenias' through exploring the contribution of genetic variation to the schizophrenic phenotypes."],["dc.format.extent","20"],["dc.identifier.doi","10.1186/1471-244X-10-91"],["dc.identifier.gro","3150558"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/5803"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/7333"],["dc.language.iso","en"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","final"],["dc.rights","Goescholar"],["dc.rights.uri","https://goescholar.uni-goettingen.de/licenses"],["dc.title","The cross-sectional GRAS sample: a comprehensive phenotypical data collection of schizophrenic patients"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dc.type.peerReviewed","no"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","219"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","Alcohol and Alcoholism"],["dc.bibliographiccitation.lastpage","223"],["dc.bibliographiccitation.volume","36"],["dc.contributor.author","Schneider, Udo"],["dc.contributor.author","Altmann, A."],["dc.contributor.author","Baumann, M."],["dc.contributor.author","Bernzen, J."],["dc.contributor.author","Bertz, B."],["dc.contributor.author","Bimber, U."],["dc.contributor.author","Broese, T."],["dc.contributor.author","Broocks, Andreas"],["dc.contributor.author","Burtscheidt, W."],["dc.contributor.author","Cimander, K. F."],["dc.contributor.author","Degkwitz, P."],["dc.contributor.author","Driessen, M. D."],["dc.contributor.author","Ehrenreich, Hannelore"],["dc.contributor.author","Fischbach, E."],["dc.contributor.author","Folkerts, H. W."],["dc.contributor.author","Frank, H."],["dc.contributor.author","Gurth, D."],["dc.contributor.author","Havemann-Reinecke, Ursula"],["dc.contributor.author","Heber, W."],["dc.contributor.author","Heuer, J. F."],["dc.contributor.author","Hingsammer, A."],["dc.contributor.author","Jacobs, S."],["dc.contributor.author","Krampe, Henning"],["dc.contributor.author","Lange, W."],["dc.contributor.author","Lay, T."],["dc.contributor.author","Leimbach, M."],["dc.contributor.author","Lemke, Matthias R."],["dc.contributor.author","Leweke, M."],["dc.contributor.author","Mangholz, A."],["dc.contributor.author","Massing, W."],["dc.contributor.author","Meyenberg, R."],["dc.contributor.author","Porzig, J."],["dc.contributor.author","Quattert, T."],["dc.contributor.author","Redner, C."],["dc.contributor.author","Ritzel, G."],["dc.contributor.author","Rollnik, J. D."],["dc.contributor.author","Sauvageoll, R."],["dc.contributor.author","Schlafke, D."],["dc.contributor.author","Schmid, G."],["dc.contributor.author","Schroder, H."],["dc.contributor.author","Schwichtenberg, U."],["dc.contributor.author","Schwoon, D."],["dc.contributor.author","Seifert, J."],["dc.contributor.author","Sickelmann, I."],["dc.contributor.author","Sieveking, C. F."],["dc.contributor.author","Spiess, C."],["dc.contributor.author","Stiegemann, H. H."],["dc.contributor.author","Stracke, R."],["dc.contributor.author","Straetgen, H. D."],["dc.contributor.author","Subkowski, P."],["dc.contributor.author","Thomasius, R."],["dc.contributor.author","Tretzel, H."],["dc.contributor.author","Verner, L. J."],["dc.contributor.author","Vitens, J."],["dc.contributor.author","Wagner, T."],["dc.contributor.author","Weirich, S."],["dc.contributor.author","Weiss, I."],["dc.contributor.author","Wendorff, T."],["dc.contributor.author","Wetterling, T."],["dc.contributor.author","Wiese, B."],["dc.contributor.author","Wittfoot, J."],["dc.date.accessioned","2018-11-07T09:04:44Z"],["dc.date.available","2018-11-07T09:04:44Z"],["dc.date.issued","2001"],["dc.description.abstract","The goals of this study were to describe demographic variables, drinking history, and the B-month prevalence of Axis I comorbidity among alcohol-dependent subjects in Germany. The variables: amount of alcohol consumption, age at onset of the first alcohol consumed, age at onset of daily alcohol consumption, age at onset of withdrawal symptoms and number of detoxifications were related to the different comorbid disorders and gender. In this study, 556 patients from 25 alcohol treatment centres were enrolled between 1 January 1999 and 30 April 1999. After a minimum of 10 days of sobriety patients who fulfilled ICD-10 and DSM-IV criteria of alcohol dependence were interviewed for data collection using the Mini-DIPS (German version of the Anxiety Disorders Interview Schedule) and a standardized psychosocial interview. The 6-month prevalence of comorbid Axis I disorders was 53.1%. Among the patients with comorbidity, affective and anxiety disorders were most frequent. Comorbid stress disorder was associated with an early start of drinking, an early beginning of withdrawal symptoms, highest number of detoxifications, and the highest amount of alcohol consumed. Female patients with anxiety disorder consumed more alcohol and started earlier than females without this comorbid disorder. The data do not answer the question of the pathogenesis of comorbid disorders and alcoholism, but indicate that stress disorders in alcoholic patients and anxiety disorders in female alcoholics influence the course and severity of alcoholism."],["dc.identifier.doi","10.1093/alcalc/36.3.219"],["dc.identifier.isi","000169318700007"],["dc.identifier.pmid","11373258"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/25172"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Oxford Univ Press"],["dc.relation.issn","0735-0414"],["dc.title","Comorbid anxiety and affective disorder in alcohol-dependent patients seeking treatment: The first Multicentre Study in Germany"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","481"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","Alcoholism Clinical and Experimental Research"],["dc.bibliographiccitation.lastpage","488"],["dc.bibliographiccitation.volume","32"],["dc.contributor.author","Driessen, Martin"],["dc.contributor.author","Schulte, Silke"],["dc.contributor.author","Luedecke, Christel"],["dc.contributor.author","Schaefer, Ingo"],["dc.contributor.author","Sutmann, Frauke"],["dc.contributor.author","Ohlmeier, Martin D."],["dc.contributor.author","Kemper, Ulrich"],["dc.contributor.author","Koesters, Gertrud"],["dc.contributor.author","Chodzinski, Claudia"],["dc.contributor.author","Schneider, Udo"],["dc.contributor.author","Broese, Thomas"],["dc.contributor.author","Dette, Christian"],["dc.contributor.author","Havemann-Reinicke, Ulla"],["dc.date.accessioned","2018-11-07T11:17:16Z"],["dc.date.available","2018-11-07T11:17:16Z"],["dc.date.issued","2008"],["dc.description.abstract","Background: We investigated (1) the prevalence of posttraumatic stress disorder (PTSD) in treatment-seeking subjects with substance use dependence (SUD), (2) the association between comorbid PTSD and the severity and course of addiction and psychopathology, and (3) this association in patients with subsyndromal PTSD, and in trauma exposure without PTSD. Methods: In this cross-sectional study, 459 subjects in 14 German addiction treatment centers participated with alcohol-dependence (A) in 39.7%, drug-dependence (D) in 33.6%, or both (AD) 26.8%. The diagnostic measures included the International Diagnostic Checklists (IDCL), Posttraumatic Diagnostic Scale (PDS), Addiction Severity Index (ASI), and the Brief Psychiatric Rating Scale (BPRS). Associations between independent characteristics and outcomes were analysed by univariate and multivariate statistics. Results: 25.3% of the subjects had PTSD confirmed by both IDCL and PDS with higher rates in the AD (34.1%) and D (29.9%) groups compared with group A (15.4%, p < 0.001). In 22.8%, PTSD was subsyndromal (either IDCL or PDS positive) without significant differences between SUD groups, and 18.3% met PTSD trauma criteria A without PTSD (exposure). After controlling for SUD and gender, trauma subgroups significantly differed regarding the onset of alcohol-related symptoms (p < 0.02), numbers of previous admissions (p < 0.03), severity of SUD (p < 0.001), current craving (p < 0.02), and psychopathology (p < 0.001). We observed the worst outcome in PTSD, while trauma exposure had no effects. Conclusions: The prevalence of PTSD is higher in drug than in alcohol dependence. The more strictly PTSD is diagnosed (by interviewer and questionnaire) the more clearly are associations with characteristics of SUD. PTSD seems to be an independent risk factor for an unfavorable outcome of SUD."],["dc.identifier.doi","10.1111/j.1530-0277.2007.00591.x"],["dc.identifier.isi","000253502300014"],["dc.identifier.pmid","18215214"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/54767"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Blackwell Publishing"],["dc.relation.issn","0145-6008"],["dc.title","Trauma and PTSD in patients with alcohol, drug, or dual dependence: A multi-center study"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","390"],["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","The World Journal of Biological Psychiatry"],["dc.bibliographiccitation.lastpage","399"],["dc.bibliographiccitation.volume","11"],["dc.contributor.author","Wedekind, Dirk"],["dc.contributor.author","Jacobs, Stefan"],["dc.contributor.author","Karg, Iris"],["dc.contributor.author","Luedecke, Christel"],["dc.contributor.author","Schneider, Udo"],["dc.contributor.author","Cimander, Konrad"],["dc.contributor.author","Baumann, Pierre"],["dc.contributor.author","Ruether, Eckart"],["dc.contributor.author","Poser, Wolfgang"],["dc.contributor.author","Havemann-Reinecke, Ursula"],["dc.date.accessioned","2018-11-07T08:45:50Z"],["dc.date.available","2018-11-07T08:45:50Z"],["dc.date.issued","2010"],["dc.description.abstract","Sixty D,L-or L-methadone treated patients in maintenance therapy were interviewed for additional drug abuse and psychiatric comorbidity; 51.7% of the entire population had a comorbid Axis-I disorder, with a higher prevalence in females (P = 0.05). Comorbid patients tended to have higher abuse of benzodiazepines, alcohol, cannabis, and cocaine, but not of heroin. They had received a significantly lower D,L- (P<0.05) and L-methadone dose than non-comorbid subjects. The duration of maintenance treatment showed an inverse relationship to frequency of additional heroin intake (P<0.01). Patients with additional heroin intake over the past 30 days had been treated with a significantly lower L-methadone dosage (P<0.05) than patients without. Axis-I comorbidity appears to be decreased when relatively higher dosages of D,L-(and L-methadone) are administered; comorbid individuals, however, were on significantly lower dosages. Finally, L-, but not D,L-methadone seems to be more effective in reducing additional heroin abuse."],["dc.identifier.doi","10.3109/15622970802176487"],["dc.identifier.isi","000284143000029"],["dc.identifier.pmid","20218800"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/20540"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Taylor & Francis Ltd"],["dc.relation.issn","1814-1412"],["dc.relation.issn","1562-2975"],["dc.title","Psychiatric comorbidity and additional abuse of drugs in maintenance treatment with L- and D,L-methadone"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]