Zilles, David

[["dc.bibliographiccitation.issue","5"],["dc.bibliographiccitation.journal","Pharmacopsychiatry"],["dc.bibliographiccitation.volume","40"],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Meyer, J."],["dc.contributor.author","Zilles, David"],["dc.contributor.author","Gruber, Eva"],["dc.contributor.author","Falkai, Peter Gaston"],["dc.date.accessioned","2018-11-07T10:59:21Z"],["dc.date.available","2018-11-07T10:59:21Z"],["dc.date.issued","2007"],["dc.format.extent","221"],["dc.identifier.isi","000249873600085"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/50679"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Georg Thieme Verlag Kg"],["dc.publisher.place","Stuttgart"],["dc.relation.conference","25th Symposium of the Arbeitsgemeinschaft-Neuropsychopharmakologie-und-Pharmakopsychiatrie"],["dc.relation.eventlocation","Munich, GERMANY"],["dc.relation.issn","0176-3679"],["dc.title","Genetic polymorphisms of serotonergic and dopaminergic neurotransmission differentially affect neurofunctional subsystems of working memory"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","547"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","Molecular Psychiatry"],["dc.bibliographiccitation.lastpage","553"],["dc.bibliographiccitation.volume","21"],["dc.contributor.author","van Erp, Theo G. M."],["dc.contributor.author","Hibar, D. P."],["dc.contributor.author","Rasmussen, J. M."],["dc.contributor.author","Glahn, D. C."],["dc.contributor.author","Pearlson, G. D."],["dc.contributor.author","Andreassen, Ole A."],["dc.contributor.author","Agartz, Ingrid"],["dc.contributor.author","Westlye, L. T."],["dc.contributor.author","Haukvik, Unn K."],["dc.contributor.author","Dale, Anders M."],["dc.contributor.author","Melle, Ingrid"],["dc.contributor.author","Hartberg, C. B."],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Kraemer, Bernd"],["dc.contributor.author","Zilles, David"],["dc.contributor.author","Donohoe, Gary"],["dc.contributor.author","Kelly, S."],["dc.contributor.author","McDonald, Colm"],["dc.contributor.author","Morris, D. W."],["dc.contributor.author","Cannon, Dara M."],["dc.contributor.author","Corvin, Aiden"],["dc.contributor.author","Machielsen, M. W. J."],["dc.contributor.author","Koenders, L."],["dc.contributor.author","de Haan, Laurens"],["dc.contributor.author","Veltman, D. J."],["dc.contributor.author","Satterthwaite, T. D."],["dc.contributor.author","Wolf, D. H."],["dc.contributor.author","Gur, Ruben C."],["dc.contributor.author","Gur, R. E."],["dc.contributor.author","Potkin, Steven G."],["dc.contributor.author","Mathalon, D. H."],["dc.contributor.author","Mueller, B. A."],["dc.contributor.author","Preda, A."],["dc.contributor.author","Macciardi, F."],["dc.contributor.author","Ehrlich, S."],["dc.contributor.author","Walton, E."],["dc.contributor.author","Hass, J."],["dc.contributor.author","Calhoun, V. D."],["dc.contributor.author","Bockholt, H. J."],["dc.contributor.author","Sponheim, S. R."],["dc.contributor.author","Shoemaker, J. M."],["dc.contributor.author","van Haren, Neeltje E. M."],["dc.contributor.author","Pol, H. E. H."],["dc.contributor.author","Ophoff, Roel A."],["dc.contributor.author","Kahn, Rene S."],["dc.contributor.author","Roiz-Santianez, R."],["dc.contributor.author","Crespo-Facorro, B."],["dc.contributor.author","Wang, L."],["dc.contributor.author","Alpert, K. I."],["dc.contributor.author","Jonsson, E. G."],["dc.contributor.author","Dimitrova, R."],["dc.contributor.author","Bois, C."],["dc.contributor.author","Whalley, H. C."],["dc.contributor.author","McIntosh, Andrew M."],["dc.contributor.author","Lawrie, Stephen M."],["dc.contributor.author","Hashimoto, Ryota"],["dc.contributor.author","Thompson, Paul M."],["dc.contributor.author","Turner, Jessica A."],["dc.date.accessioned","2018-11-07T10:16:34Z"],["dc.date.available","2018-11-07T10:16:34Z"],["dc.date.issued","2016"],["dc.description.abstract","The profile of brain structural abnormalities in schizophrenia is still not fully understood, despite decades of research using brain scans. To validate a prospective meta-analysis approach to analyzing multicenter neuroimaging data, we analyzed brain MRI scans from 2028 schizophrenia patients and 2540 healthy controls, assessed with standardized methods at 15 centers worldwide. We identified subcortical brain volumes that differentiated patients from controls, and ranked them according to their effect sizes. Compared with healthy controls, patients with schizophrenia had smaller hippocampus (Cohen's d = -0.46), amygdala (d = -0.31), thalamus (d = -0.31), accumbens (d = -0.25) and intracranial volumes (d = -0.12), as well as larger pallidum (d = 0.21) and lateral ventricle volumes (d = 0.37). Putamen and pallidum volume augmentations were positively associated with duration of illness and hippocampal deficits scaled with the proportion of unmedicated patients. Worldwide cooperative analyses of brain imaging data support a profile of subcortical abnormalities in schizophrenia, which is consistent with that based on traditional meta-analytic approaches. This first ENIGMA Schizophrenia Working Group study validates that collaborative data analyses can readily be used across brain phenotypes and disorders and encourages analysis and data sharing efforts to further our understanding of severe mental illness."],["dc.identifier.doi","10.1038/mp.2015.63"],["dc.identifier.isi","000372421500014"],["dc.identifier.pmid","26033243"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/13363"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/41060"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.relation.issn","1476-5578"],["dc.relation.issn","1359-4184"],["dc.rights","CC BY-NC-ND 4.0"],["dc.rights.uri","https://creativecommons.org/licenses/by-nc-nd/4.0"],["dc.title","Subcortical brain volume abnormalities in 2028 individuals with schizophrenia and 2540 healthy controls via the ENIGMA consortium"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","229"],["dc.bibliographiccitation.issue","5"],["dc.bibliographiccitation.journal","Pharmacopsychiatry"],["dc.bibliographiccitation.lastpage","230"],["dc.bibliographiccitation.volume","40"],["dc.contributor.author","Zilles, David"],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Burke, Sarah"],["dc.contributor.author","Falkai, Peter Gaston"],["dc.date.accessioned","2018-11-07T10:59:22Z"],["dc.date.available","2018-11-07T10:59:22Z"],["dc.date.issued","2007"],["dc.identifier.isi","000249873600125"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/50683"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Georg Thieme Verlag Kg"],["dc.publisher.place","Stuttgart"],["dc.relation.conference","25th Symposium of the Arbeitsgemeinschaft-Neuropsychopharmakologie-und-Pharmakopsychiatrie"],["dc.relation.eventlocation","Munich, GERMANY"],["dc.relation.issn","0176-3679"],["dc.title","The influence of genetic loading on working memory performance in schizophrenia"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.issue","5"],["dc.bibliographiccitation.journal","Australian and New Zealand Journal of Psychiatry"],["dc.bibliographiccitation.volume","44"],["dc.contributor.author","Zilles, David"],["dc.contributor.author","Hasan, Alkomiet"],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Degner, Detlef"],["dc.date.accessioned","2018-11-07T08:43:23Z"],["dc.date.available","2018-11-07T08:43:23Z"],["dc.date.issued","2010"],["dc.format.extent","489"],["dc.identifier.doi","10.3109/00048671003614163"],["dc.identifier.isi","000276761100013"],["dc.identifier.pmid","20397795"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/19951"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Informa Healthcare"],["dc.relation.issn","0004-8674"],["dc.title","Acute Polydipsia and Water Intoxication in First Episode Schizophrenia"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.subtype","letter_note"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","127"],["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","European Archives of Psychiatry and Clinical Neuroscience"],["dc.bibliographiccitation.lastpage","136"],["dc.bibliographiccitation.volume","265"],["dc.contributor.author","Kittel-Schneider, Sarah"],["dc.contributor.author","Wobrock, Thomas"],["dc.contributor.author","Scherk, Harald"],["dc.contributor.author","Schneider-Axmann, Thomas"],["dc.contributor.author","Trost, Sarah"],["dc.contributor.author","Zilles, David"],["dc.contributor.author","Wolf, C."],["dc.contributor.author","Schmitt, A."],["dc.contributor.author","Malchow, Berend"],["dc.contributor.author","Hasan, Alkomiet"],["dc.contributor.author","Backens, Martin"],["dc.contributor.author","Reith, W."],["dc.contributor.author","Falkai, Peter Gaston"],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Reif, A."],["dc.date.accessioned","2018-11-07T10:00:31Z"],["dc.date.available","2018-11-07T10:00:31Z"],["dc.date.issued","2015"],["dc.description.abstract","The diacylglycerol kinase eta (DGKH) gene, first identified in a genome-wide association study, is one of the few replicated risk genes of bipolar affective disorder (BD). Following initial positive studies, it not only was found to be associated with BD but also implicated in the etiology of other psychiatric disorders featuring affective symptoms, rendering DGKH a cross-disorder risk gene. However, the (patho-)physiological role of the encoded enzyme is still elusive. In the present study, we investigated primarily the influence of a risk haplotype on amygdala volume in patients suffering from schizophrenia or BD as well as healthy controls and four single nucleotide polymorphisms conveying risk. There was a significant association of the DGKH risk haplotype with increased amygdala volume in BD, but not in schizophrenia or healthy controls. These findings add to the notion of a role of DGKH in the pathogenesis of BD."],["dc.description.sponsorship","DFG [RTG 1253/1, RE1632/5-1]; BMBF (DZHI) [01EO1004]; IZKF [Z3-24]"],["dc.identifier.doi","10.1007/s00406-014-0513-9"],["dc.identifier.isi","000350305500005"],["dc.identifier.pmid","24958494"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/37825"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.publisher.place","Heidelberg"],["dc.relation.issn","1433-8491"],["dc.relation.issn","0940-1334"],["dc.title","Influence of DGKH variants on amygdala volume in patients with bipolar affective disorder and schizophrenia"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.journal","The International Journal of Neuropsychopharmacology"],["dc.bibliographiccitation.volume","15"],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Vieker, Henning"],["dc.contributor.author","Diekhof, Esther Kristina"],["dc.contributor.author","Trost, Sarah"],["dc.contributor.author","Fanelli, A."],["dc.contributor.author","Jakob, K."],["dc.contributor.author","Petrovic, A."],["dc.contributor.author","Weber, K."],["dc.contributor.author","Keil, M."],["dc.contributor.author","Zilles, David"],["dc.contributor.author","Falkai, Peter Gaston"],["dc.date.accessioned","2018-11-07T09:10:04Z"],["dc.date.available","2018-11-07T09:10:04Z"],["dc.date.issued","2012"],["dc.format.extent","214"],["dc.identifier.isi","000209062500785"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/26410"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Oxford Univ Press"],["dc.publisher.place","Oxford"],["dc.relation.issn","1469-5111"],["dc.relation.issn","1461-1457"],["dc.title","Imaging endophenotypic biomarkers for schizophrenic and affective psychoses in key neural circuits"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1914"],["dc.bibliographiccitation.issue","8"],["dc.bibliographiccitation.journal","Neuropsychopharmacology"],["dc.bibliographiccitation.lastpage","1923"],["dc.bibliographiccitation.volume","39"],["dc.contributor.author","Trost, Sarah"],["dc.contributor.author","Diekhof, Esther Kristina"],["dc.contributor.author","Zvonik, Kerstin"],["dc.contributor.author","Lewandowski, Mirjana"],["dc.contributor.author","Usher, Juliana"],["dc.contributor.author","Keil, Maria"],["dc.contributor.author","Zilles, David"],["dc.contributor.author","Falkai, Peter"],["dc.contributor.author","Dechent, Peter"],["dc.contributor.author","Gruber, Oliver"],["dc.date.accessioned","2018-11-07T09:38:30Z"],["dc.date.available","2018-11-07T09:38:30Z"],["dc.date.issued","2014"],["dc.description.abstract","Bipolar disorder (BD) is characterized by recurrent mood episodes ranging from severe depression to acute full-blown mania. Both states of this severe psychiatric disorder have been associated with alterations of reward processing in the brain. Here, we present results of a functional magnetic resonance imaging (fMRI) study on the neural correlates and functional interactions underlying reward gain processing and reward dismissal in favor of a long-term goal in bipolar patients. Sixteen medicated patients diagnosed with bipolar 1 disorder, euthymic to mildly depressed, and sixteen matched healthy controls performed the 'desire-reason dilemma' (DRD) paradigm demanding rejection of priorly conditioned reward stimuli to successfully pursue a superordinate goal. Both groups exhibited significant activations in reward-related brain regions, particularly in the mesolimbic reward system. However, bipolar patients showed reduced neural responses of the ventral striatum (vStr) when exploiting a reward stimulus, and exhibited a decreased suppression of the reward-related activation of the mesolimbic reward system while having to reject immediate reward in favor of the long-term goal. Further, functional interaction between the anteroventral prefrontal cortex and the vStr in the 'DRD' was significantly impaired in the bipolar group. These findings provide evidence for a reduced responsivity of the vStr to reward stimuli in BD, possibly related to clinical features like anhedonia. The disturbed top-down control of mesolimbic reward signals by prefrontal brain regions in BD can be interpreted in terms of a disease-related enhanced impulsivity, a trait marker of BD."],["dc.identifier.doi","10.1038/npp.2014.39"],["dc.identifier.isi","000337550600013"],["dc.identifier.pmid","24535101"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/33078"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Nature Publishing Group"],["dc.relation.issn","1740-634X"],["dc.relation.issn","0893-133X"],["dc.title","Disturbed Anterior Prefrontal Control of the Mesolimbic Reward System and Increased Impulsivity in Bipolar Disorder"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","258"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","The World Journal of Biological Psychiatry"],["dc.bibliographiccitation.lastpage","267"],["dc.bibliographiccitation.volume","14"],["dc.contributor.author","Zilles, David"],["dc.contributor.author","Jung, Raphael"],["dc.contributor.author","Gruber, Eva"],["dc.contributor.author","Falkai, Peter"],["dc.contributor.author","Gruber, Oliver"],["dc.date.accessioned","2018-11-07T09:25:13Z"],["dc.date.available","2018-11-07T09:25:13Z"],["dc.date.issued","2013"],["dc.description.abstract","Objectives. The traditional clinical dichotomy of schizophrenia and bipolar disorder has been challenged by recent findings of an at least in part common genetic basis. The investigation of neurocognitive functions like working memory may thereby contribute to elucidate common versus distinct pathophysiological processes of the major psychoses. To date direct comparisons of working memory functioning in schizophrenia and bipolar disorder have been rare and moreover have revealed inconsistent findings. In this study we aimed to further clarify the diagnostic specificity of working memory deficits in schizophrenia and bipolar disorder. Methods. Fifty patients with schizophrenia, 23 patients with bipolar disorder and 53 healthy controls were tested with regard to specific dysfunctions of verbal and visuospatial working memory components using a set of well-characterized, brain circuit-specific paradigms with established brain-behaviour relationships. Results. Patients with schizophrenia showed marked deficits across different working memory domains while bipolar patients performed intermediate with no significant differences compared to the control group. Working memory performance of patients with schizophrenia and bipolar disorder significantly differed in only one particular task requiring articulatory rehearsal of verbal information. Conclusions. While these results do not provide unequivocal support for the Kraepelinian dichotomy, they are consistent with recent findings suggesting the existence of a specific subgroup of schizophrenia patients phenotypically characterized by selective deficits of the articulatory rehearsal mechanism of verbal working memory."],["dc.identifier.doi","10.3109/15622975.2011.591825"],["dc.identifier.isi","000318650900003"],["dc.identifier.pmid","21745128"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/30012"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Informa Healthcare"],["dc.relation.issn","1562-2975"],["dc.title","Differential working memory performance as support for the Kraepelinian dichotomy between schizophrenia and bipolar disorder? An experimental neuropsychological study using circuit-specific working memory tasks"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","Pharmacopsychiatry"],["dc.bibliographiccitation.volume","44"],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Diekhof, Esther Kristina"],["dc.contributor.author","Zvonik, Kerstin"],["dc.contributor.author","Trost, Sarah"],["dc.contributor.author","Weber, K."],["dc.contributor.author","Petrovic, A."],["dc.contributor.author","Henseler, Ilona"],["dc.contributor.author","Zilles, David"],["dc.contributor.author","Melcher, T."],["dc.contributor.author","Keil, M."],["dc.contributor.author","Dechent, Peter"],["dc.contributor.author","Falkai, Peter Gaston"],["dc.date.accessioned","2018-11-07T08:52:00Z"],["dc.date.available","2018-11-07T08:52:00Z"],["dc.date.issued","2011"],["dc.identifier.doi","10.1055/s-0031-1292483"],["dc.identifier.isi","000296086300067"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/22064"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Georg Thieme Verlag Kg"],["dc.publisher.place","Stuttgart"],["dc.relation.conference","27th Symposium of the AGNP"],["dc.relation.eventlocation","Munich, GERMANY"],["dc.relation.issn","0176-3679"],["dc.title","Multi-functional MRI studies of disordered brain circuits in schizophrenic and affective psychoses"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","667"],["dc.bibliographiccitation.issue","8"],["dc.bibliographiccitation.journal","European Archives of Psychiatry and Clinical Neuroscience"],["dc.bibliographiccitation.lastpage","676"],["dc.bibliographiccitation.volume","262"],["dc.contributor.author","Zilles, David"],["dc.contributor.author","Meyer, Jobst"],["dc.contributor.author","Schneider-Axmann, Thomas"],["dc.contributor.author","Ekawardhani, Savira"],["dc.contributor.author","Gruber, Eva"],["dc.contributor.author","Falkai, Peter"],["dc.contributor.author","Gruber, Oliver"],["dc.date.accessioned","2018-11-07T09:03:05Z"],["dc.date.available","2018-11-07T09:03:05Z"],["dc.date.issued","2012"],["dc.description.abstract","Working memory deficits are found in different psychiatric populations and are most pronounced in schizophrenia. There is preliminary evidence from pharmacological studies that the verbal and visuospatial subcomponents of working memory are subject to differential neurotransmitter modulation. Here, we investigated the impact of well-known polymorphisms of the dopamine transporter gene (SLC6A3, DAT) and the catechol-O-methyl-transferase gene (COMT) as well as the serotonin transporter gene (SLC6A4, 5-HTT) on these specific working memory subcomponents in a mixed sample of patients and healthy individuals. Twenty healthy subjects and 80 patients diagnosed with schizophrenia, bipolar I disorder, or obsessive-compulsive disorder underwent genotyping for the DAT variable number of tandem repeats (VNTR), the COMT val/met-, and the 5-HTT promoter length polymorphism (5-HTTLPR) and neuropsychological testing using a battery of well-characterized, brain circuit-specific working memory tasks. DAT genotype revealed a significant and selective effect on visuospatial working memory, while there was no effect on verbal working memory functioning. 5-HTT genotype, by contrast, exerted a significant and selective effect on verbal working memory task performance. COMT genotype did not show any influence on either working memory domain. The results of the present study provide evidence for a differential impact of genetic polymorphisms of the dopaminergic and serotonergic systems on verbal and visuospatial working memory functioning. Together with prior evidence suggesting the existence of subgroups of schizophrenia patients exhibiting isolated deficits in only one working memory domain, this finding further supports the idea of endophenotypically and pathophysiologically distinct subgroups of schizophrenia with implications for personalized therapeutic approaches."],["dc.identifier.doi","10.1007/s00406-012-0312-0"],["dc.identifier.isi","000310812800005"],["dc.identifier.pmid","22454241"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/9471"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/24827"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.publisher.place","Heidelberg"],["dc.relation.issn","0940-1334"],["dc.rights","CC BY-NC-ND 3.0"],["dc.rights.uri","https://creativecommons.org/licenses/by-nc-nd/3.0"],["dc.title","Genetic polymorphisms of 5-HTT and DAT but not COMT differentially affect verbal and visuospatial working memory functioning"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]