Lang, Christine

[["dc.bibliographiccitation.firstpage","135"],["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","Psychiatry Research"],["dc.bibliographiccitation.lastpage","146"],["dc.bibliographiccitation.volume","128"],["dc.contributor.author","Weniger, Godehard"],["dc.contributor.author","Lange, C."],["dc.contributor.author","Ruther, Eckart"],["dc.contributor.author","Irle, Eva"],["dc.date.accessioned","2018-11-07T10:45:28Z"],["dc.date.available","2018-11-07T10:45:28Z"],["dc.date.issued","2004"],["dc.description.abstract","The goal of this study was to assess facial affect recognition abilities in subjects with various schizophrenia subtypes and subjects with major depression. A total of six disorganized, 21 paranoid and 18 residual subjects with schizophrenia (DSM-IV criteria) were compared with 21 subjects with major depression (DSM-1V criteria) and 30 matched healthy control subjects. Two experimental tasks requiring the sorting and rating of emotional facial expressions were applied. Disorganized and paranoid subjects showed strong impairments in the sorting of emotional facial expressions. Depressive subjects displayed only minor deficits, and residual subjects were unimpaired. Subjects with disorganized schizophrenia rated emotional facial expressions as more aroused, and depressive subjects rated them as less aroused, than the other study groups. Our study demonstrates strong deficits in facial affect recognition in subjects with schizophrenia and pronounced disorganized or psychotic symptoms. Deficits in facial affect recognition are specific to schizophrenia. They may be considered as a state marker of schizophrenia. (C) 2004 Elsevier Ireland Ltd. All rights reserved."],["dc.identifier.doi","10.1016/j.psychres.2003.12.027"],["dc.identifier.isi","000224994600004"],["dc.identifier.pmid","15488956"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/47509"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Elsevier Sci Ireland Ltd"],["dc.relation.issn","0165-1781"],["dc.title","Differential impairments of facial affect recognition in schizophrenia subtypes and major depression"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","330"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","Acta Psychiatrica Scandinavica"],["dc.bibliographiccitation.lastpage","331"],["dc.bibliographiccitation.volume","119"],["dc.contributor.author","Irle, Eva"],["dc.contributor.author","Lange, C."],["dc.contributor.author","Sachsse, Ulrich"],["dc.contributor.author","Weniger, Godehard"],["dc.date.accessioned","2018-11-07T08:30:49Z"],["dc.date.available","2018-11-07T08:30:49Z"],["dc.date.issued","2009"],["dc.identifier.doi","10.1111/j.1600-0447.2009.01351.x"],["dc.identifier.isi","000263855500012"],["dc.identifier.pmid","19207126"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/16984"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Wiley-blackwell Publishing, Inc"],["dc.relation.issn","0001-690X"],["dc.title","Further evidence that post-traumatic stress disorder but not dissociative disorders are related to amygdala and hippocampal size reduction in trauma-exposed individuals"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.subtype","letter_note"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","20"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Seizure"],["dc.bibliographiccitation.lastpage","32"],["dc.bibliographiccitation.volume","11"],["dc.contributor.author","Exner, C."],["dc.contributor.author","Boucsein, K."],["dc.contributor.author","Lange, C."],["dc.contributor.author","Winter, H."],["dc.contributor.author","Weniger, Godehard"],["dc.contributor.author","Steinhoff, B. J."],["dc.contributor.author","Irle, Eva"],["dc.date.accessioned","2018-11-07T10:33:47Z"],["dc.date.available","2018-11-07T10:33:47Z"],["dc.date.issued","2002"],["dc.description.abstract","The search for a special neuropsychological profile of frontal lobe epilepsy subjects (FLE) has so far led to inconclusive results. In this paper we compared the preoperative neuropsychological performance of FLE and temporal lobe epilepsy (TLE) subjects. We further investigated whether frontal lobe lesions of epileptogenic cause produce the same type of cognitive dysfunction as do tumours of the frontal lobe. Sixteen FLE subjects were compared to 16 TLE subjects as well as to a group of 10 subjects after the removal of frontal lobe tumors (TUM) and a healthy control group, A set of neuropsychological test measures routinely used for presurgical evaluation, an emotional conceptualization task and two associative learning tasks were administered. We found that subjects with frontal lobe damage were significantly impaired relative to controls on a wide range of cognitive functions independent of neurological cause. FLE subjects could hardly be discriminated from TLE subjects as both groups showed a similarly reduced level of neuropsychological performance. Our results demonstrate the devastating effect that frontal lobe epilepsy can have on cognitive functioning. Routinely used neuropsychological test measures lack the specificity to distinguish between frontal and temporal lobe epilepsy. Highly specialized measures are necessary to reveal differences. (C) 2002 BEA Trading Ltd."],["dc.identifier.doi","10.1053/seiz.2001.0572"],["dc.identifier.isi","000174109800004"],["dc.identifier.pmid","11888256"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/44700"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","W B Saunders Co Ltd"],["dc.relation.issn","1059-1311"],["dc.title","Neuropsychological performance in frontal lobe epilepsy"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","281"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","Acta Psychiatrica Scandinavica"],["dc.bibliographiccitation.lastpage","290"],["dc.bibliographiccitation.volume","118"],["dc.contributor.author","Weniger, Godehard"],["dc.contributor.author","Lange, C."],["dc.contributor.author","Sachsse, Ulrich"],["dc.contributor.author","Irle, Eva"],["dc.date.accessioned","2018-11-07T11:10:09Z"],["dc.date.available","2018-11-07T11:10:09Z"],["dc.date.issued","2008"],["dc.description.abstract","Objective: Trauma-exposed individuals with post-traumatic stress disorder (PTSD) display reduced amygdala and hippocampal size and impaired cognition. However, studies on trauma-exposed individuals with dissociative amnesia (DA) or dissociative identity disorder (DID) are lacking. Method: Twenty-three young women who had experienced severe childhood sexual/physical abuse, diagnosed with DA/DID or PTSD, and 25 healthy control subjects were subjected to 3D structural magnetic resonance imaging of amygdala and hippocampus and a clinical and neuropsychological investigation. Results: Compared with controls, trauma-exposed subjects with PTSD (n = 10) displayed significantly reduced amygdala and hippocampal size and significantly impaired cognition. By contrast, trauma-exposed subjects with DA or DID (n = 13) displayed normal amygdala and hippocampal size and normal cognition. Conclusion: We report for the first time volumetric results in subjects with DA/DID without PTSD as comorbid diagnosis. Our results indicate preserved amygdala and hippocampal size and preserved cognition in subjects with these disorders."],["dc.description.sponsorship","Deutsche Forschungsgemeinschaft [IR 15/8]"],["dc.identifier.doi","10.1111/j.1600-0447.2008.01246.x"],["dc.identifier.isi","000259207900004"],["dc.identifier.pmid","18759808"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/53155"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Wiley-blackwell"],["dc.relation.issn","0001-690X"],["dc.title","Amygdala and hippocampal volumes and cognition in adult survivors of childhood abuse with dissociative disorders"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]