Wetz, Anna Julienne

[["dc.bibliographiccitation.firstpage","58"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Anaesthesia and Intensive Care"],["dc.bibliographiccitation.lastpage","66"],["dc.bibliographiccitation.volume","45"],["dc.contributor.author","Wetz, A. J."],["dc.contributor.author","Richardt, E. M."],["dc.contributor.author","Schotola, H."],["dc.contributor.author","Bauer, M."],["dc.contributor.author","Bräuer, A."],["dc.date.accessioned","2020-12-10T18:38:26Z"],["dc.date.available","2020-12-10T18:38:26Z"],["dc.date.issued","2017"],["dc.description.abstract","Acute kidney injury (AKI) is frequently observed after cardiac surgery (CS) with cardiopulmonary bypass (CPB). Multiple mechanisms underlie this phenomenon, including CPB-dependent haemolysis. Haemoglobin is released during haemolysis, and free haemoglobin (frHb) causes tubular cell injury after exceeding the binding capacity of haptoglobin (Hp). The objective of this study was to investigate the influence of perioperative changes in frHb and Hp levels on the incidence of CS-associated (CSA) AKI. After receiving local ethics committee approval and obtaining informed consent from our patients, we analysed the data pertaining to 154 patients undergoing CPB surgery. We recorded frHb and Hp concentrations pre-, intra- and postoperatively and defined AKI using the Kidney Disease Improving Global Outcomes (KDIGO) classification. We observed that frHb levels increased significantly during surgery and then decreased at ten hours thereafter and that Hp levels decreased during surgery and remained at low levels until the first postoperative day. We noted a moderate negative correlation between frHb and Hp levels. AKI was identified in 45.5% of patients; however, there was no significant difference in frHb or Hp levels between patients with and without AKI. We did not observe a relationship between frHb or Hp levels and CSA AKI and thus could not confirm the hypothesis that patients with higher baseline Hp concentrations experience a lower incidence of AKI than patients with lower baseline Hp concentrations."],["dc.identifier.doi","10.1177/0310057X1704500109"],["dc.identifier.eissn","1448-0271"],["dc.identifier.isi","000392257300009"],["dc.identifier.issn","0310-057X"],["dc.identifier.pmid","28072936"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/77317"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","PUB_WoS_Import"],["dc.publisher","Australian Soc Anaesthetists"],["dc.relation.issn","1448-0271"],["dc.relation.issn","0310-057X"],["dc.title","Haptoglobin and free haemoglobin during cardiac surgery-is there a link to acute kidney injury?"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","27"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Der Anaesthesist"],["dc.bibliographiccitation.lastpage","33"],["dc.bibliographiccitation.volume","67"],["dc.contributor.author","Grote, R."],["dc.contributor.author","Wetz, A. J."],["dc.contributor.author","Bräuer, A."],["dc.contributor.author","Menzel, M."],["dc.date.accessioned","2020-12-10T14:08:05Z"],["dc.date.available","2020-12-10T14:08:05Z"],["dc.date.issued","2017"],["dc.identifier.doi","10.1007/s00101-017-0384-3"],["dc.identifier.eissn","1432-055X"],["dc.identifier.issn","0003-2417"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/70373"],["dc.language.iso","de"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","Vorwärmung entsprechend der „S3 Leitlinie Vermeidung von unbeabsichtigter perioperativer Hypothermie 2014“"],["dc.title.alternative","Prewarming according to the AWMF S3 guidelines on preventing inadvertant perioperative hypothermia 2014. Retrospective analysis of 7786 patients"],["dc.title.subtitle","Retrospektive Analyse von 7786 Patienten"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","Acta Anaesthesiologica Scandinavica"],["dc.bibliographiccitation.lastpage","5"],["dc.bibliographiccitation.volume","64"],["dc.contributor.author","Grote, Rolf"],["dc.contributor.author","Wetz, Anna"],["dc.contributor.author","Bräuer, Anselm"],["dc.contributor.author","Menzel, Matthias"],["dc.date.accessioned","2020-01-14T10:38:14Z"],["dc.date.accessioned","2021-10-27T13:22:05Z"],["dc.date.available","2020-01-14T10:38:14Z"],["dc.date.available","2021-10-27T13:22:05Z"],["dc.date.issued","2019"],["dc.description.abstract","BACKGROUND: Prevention of inadvertent hypothermia is recommended for procedures >30 minutes because hypothermia increases the risk of myocardial ischemia, intraoperative blood loss, transfusion and wound complications. Therefore, short warming interruptions between pre-warming and intraoperative warming might result in lower hypothermia rates. The aim of this retrospective investigation was to determine whether the incidence of inadvertent intraoperative hypothermia was affected by the warming interruption. METHODS: The lowest intraoperative body core temperature value and the warming interruption time were taken from anaesthesia records. Body core temperature was recorded continuously, and a patient was classified to be hypothermic if the lowest recorded temperature value was <36°C. Hypothermia rates and the correlation between warming interruption times and intraoperative hypothermia rates were calculated. RESULTS: Five thousand eighty-four patients were analysed. The intraoperative hypothermia rate was 15.3%. Nineteen patients (0.4%) had a recorded temperature of <35.0°C. An increase in forced-air warming interruption time was significantly associated with an increase in intraoperative hypothermia rates (P < .0001). Patients with interruptions in forced-air warming >20 minutes showed significantly higher hypothermia rates than those with interruptions of ≤20 minutes (P < .0001). CONCLUSION: Intraoperative hypothermia rates increased significantly with longer forced-air warming interruptions between pre-warming and intraoperative warming. Short warming interruptions can preserve the effect of pre-warming and are associated with low intraoperative hypothermia rates."],["dc.identifier.doi","10.1111/aas.13521"],["dc.identifier.eissn","1399-6576"],["dc.identifier.issn","0001-5172"],["dc.identifier.pmid","31828757"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/17088"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/92067"],["dc.language.iso","en"],["dc.notes.intern","Migrated from goescholar"],["dc.relation.eissn","1399-6576"],["dc.relation.issn","1399-6576"],["dc.relation.issn","0001-5172"],["dc.relation.orgunit","Universitätsmedizin Göttingen"],["dc.rights","CC BY 4.0"],["dc.rights.uri","https://creativecommons.org/licenses/by/4.0"],["dc.subject.ddc","610"],["dc.title","Short interruptions between pre‐warming and intraoperative warming are associated with low intraoperative hypothermia rates"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.artnumber","183"],["dc.bibliographiccitation.journal","CRITICAL CARE"],["dc.bibliographiccitation.volume","19"],["dc.contributor.author","Wetz, Anna J."],["dc.contributor.author","Braeuer, Anselm"],["dc.contributor.author","Quintel, Michael"],["dc.contributor.author","Heise, Daniel"],["dc.date.accessioned","2018-11-07T09:58:22Z"],["dc.date.available","2018-11-07T09:58:22Z"],["dc.date.issued","2015"],["dc.description.abstract","Introduction: Postoperative acute kidney injury (AKI) is a frequently observed phenomenon after cardiac surgery with cardio-pulmonary bypass (CPB); this severe complication is associated with adverse patient outcomes. There are multiple mechanisms involved in AKI during cardiac surgery, including CPB-dependent hemolysis. An IV infusion of sodium bicarbonate, which leads to urine alkalization, may play a role in preventing AKI. Recently, several trials have investigated the effect of sodium bicarbonate and reported controversial results. The purpose of this investigation was to investigate the following question. Under what circumstances can sodium bicarbonate prevent postoperative AKI? Methods: We analyzed data from 342 patients undergoing CPB surgery at the University Hospital Goettingen, Germany. A total of 174 patients received a preemptive dose of sodium bicarbonate. Directly after the induction of anesthesia, the continuous infusion of 0.15 mmol/kg body weight/h was started and continued until 2 pm on the first postoperative day. Patients who were not treated with sodium bicarbonate formed the control group (n = 168). To verify the AKI risk configuration of each group, we surveyed risk factors and determined the commonly used clinical predictive score according to Thakar and colleagues. We recorded the concentration of free hemoglobin (fhb) to estimate the amount of CPB-dependent hemolysis. The definition of AKI was acquired by applying the AKI-network (AKIN) classification over the course of five postoperative days. Results: Patients who received the sodium bicarbonate infusion showed a significantly lower incidence (35.6 vs. 50%) of AKI than that of patients who did not receive the infusion (p = 0.01). AKIN levels 2 and 3 were also more frequent when sodium bicarbonate was not administered. Particularly, in the low-risk cohort (<3 Thakar points), the incidence of AKI was significantly reduced (26 vs. 46%) when patients received sodium bicarbonate (p = 0.01), whereas in the high-risk patients, no significant reduction was observed. Conclusion: In this study, we observed that low-risk patients particularly benefited from the preventive treatment with sodium bicarbonate. The incidence of AKI was significantly reduced in low-risk patients while there was no statistically significant difference in the high-risk patient cohort."],["dc.description.sponsorship","Open-Access-Publikationsfonds 2015"],["dc.identifier.doi","10.1186/s13054-015-0906-9"],["dc.identifier.isi","000356480100001"],["dc.identifier.pmid","25899029"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/13465"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/37351"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Biomed Central Ltd"],["dc.relation.issn","1364-8535"],["dc.relation.issn","1466-609X"],["dc.rights","CC BY 4.0"],["dc.rights.uri","https://creativecommons.org/licenses/by/4.0"],["dc.title","Does sodium bicarbonate infusion really have no effect on the incidence of acute kidney injury after cardiac surgery? A prospective observational trial"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","282"],["dc.bibliographiccitation.journal","Journal of Clinical Anesthesia"],["dc.bibliographiccitation.lastpage","289"],["dc.bibliographiccitation.volume","34"],["dc.contributor.author","Wetz, Anna J."],["dc.contributor.author","Perl, Thorsten"],["dc.contributor.author","Brandes, Ivo Florian"],["dc.contributor.author","Harden, Markus"],["dc.contributor.author","Bauer, Martin"],["dc.contributor.author","Braeuer, Anselm"],["dc.date.accessioned","2018-11-07T10:06:45Z"],["dc.date.available","2018-11-07T10:06:45Z"],["dc.date.issued","2016"],["dc.description.abstract","Study objective: Perioperative hypothermia is a frequently observed phenomenon of general anesthesia and is associated with adverse patient outcome. Recently, a significant influence of core temperature before induction of anesthesia has been reported. However, there are still little existing data on core temperature before induction of anesthesia and no data regarding potential risk factors for developing preoperative hypothermia. The purpose of this investigation was to estimate the incidence of hypothermia before anesthesia and to determine if certain factors predict its incidence. Design/setting/patients: Data from 7 prospective studies investigating core temperature previously initiated at our department were analyzed. Patients undergoing a variety of elective surgical procedures were included. Interventions/measurements: Core temperature was measured before induction of anesthesia with an oral (314 patients), infrared tympanic (143 patients), or tympanic contact thermometer (36 patients). Available potential predictors included American Society of Anesthesiologists status, sex, age, weight, height, body mass index, adipose ratio, and lean body weight. Association with preoperative hypothermia was assessed separately for each predictor using logistic regression. Independent predictors were identified using multivariable logistic regression. Main results: A total of 493 patients were included in the study. Hypothermia was found in 105 patients (21.3%; 95% confidence interval, 17.8%-25.2%). The median core temperature was 36.3 degrees C (25th-75th percentiles, 36.0 degrees C-36.7 degrees C). Two independent factors for preoperative hypothermia were identified: male sex and age (>52 years). Conclusions: As a consequence of the high incidence of hypothermia before anesthesia, measuring core temperature should be mandatory 60 to 120 minutes before induction to identify and provide adequate treatment to hypothermic patients. (C) 2016 Elsevier Inc. All rights reserved."],["dc.identifier.doi","10.1016/j.jclinane.2016.03.065"],["dc.identifier.isi","000384952700058"],["dc.identifier.pmid","27687393"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/39155"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Elsevier Science Inc"],["dc.relation.issn","1873-4529"],["dc.relation.issn","0952-8180"],["dc.title","Unexpectedly high incidence of hypothermia before induction of anesthesia in elective surgical patients"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.artnumber","3"],["dc.bibliographiccitation.journal","CRITICAL CARE"],["dc.bibliographiccitation.volume","19"],["dc.contributor.author","Wetz, Anna J."],["dc.contributor.author","Richardt, Eva M."],["dc.contributor.author","Wand, Saskia"],["dc.contributor.author","Kunze, Nils"],["dc.contributor.author","Schotola, Hanna"],["dc.contributor.author","Quintel, Michael"],["dc.contributor.author","Braeuer, Anselm"],["dc.contributor.author","Moerer, Onnen"],["dc.date.accessioned","2018-11-07T10:02:21Z"],["dc.date.available","2018-11-07T10:02:21Z"],["dc.date.issued","2015"],["dc.description.abstract","Introduction: Postoperative acute kidney injury (AKI) is a frequently observed complication after on-pump cardiac surgery (CS) and is associated with adverse patient outcomes. Early identification of patients at risk is essential for the prevention of AKI after CS. In this study, we analysed whether urinary tissue inhibitor of metalloproteinase 2 (TIMP-2) combined with urine insulin-like growth factor binding protein 7 (IGFBP-7) ([TIMP-2] x [IGFBP-7]) is an adequate diagnostic test to identify early AKI after on-pump CS. Methods: In 42 patients undergoing coronary artery bypass graft surgery, we surveyed individual risk factors for AKI and defined AKI by applying the Kidney Disease: Improving Global Outcomes (KDIGO) classification during the day of surgery and the following 2 days after surgery. Concentrations of urinary TIMP-2 multiplied by IGFBP-7 were recorded at four time points: at baseline pre-surgery, at the end of surgery, 4 hours after cardiopulmonary bypass (CPB) and at 8: 00 AM on the first postoperative day. Results: In total, 38% of the patients experienced AKI. The results showed a median baseline [TIMP-2] x [IGFBP-7] concentration of 0.3 (ng/ml)(2)/1,000, decreasing at the end of surgery and then increasing at the next measurement point 4 hours after CPB and further on the first postoperative day. On the first postoperative day, patients with AKI had significantly higher concentrations of [TIMP-2] x [IGFBP-7]. On the day of surgery, the concentration did not significantly differ between patients classified as KDIGO 0 or KDIGO 1 or 2. Previously published cutoff points of 0.3 and 2 were not confirmed in our study cohort. Conclusion: [TIMP-2] x [IGFBP-7] concentration can be used as a diagnostic test to identify patients at increased risk of AKI after CS on the first postoperative day. At earlier time points, no significant difference in [TIMP-2] x [IGFBP-7] concentration was found between patients classified as KDIGO 0 or KDIGO 1 or 2."],["dc.description.sponsorship","Astute Medical"],["dc.identifier.doi","10.1186/s13054-014-0717-4"],["dc.identifier.isi","000351851200001"],["dc.identifier.pmid","25560277"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/11548"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/38205"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Biomed Central Ltd"],["dc.relation.issn","1364-8535"],["dc.relation.issn","1466-609X"],["dc.rights","CC BY 4.0"],["dc.rights.uri","https://creativecommons.org/licenses/by/4.0"],["dc.title","Quantification of urinary TIMP-2 and IGFBP-7: an adequate diagnostic test to predict acute kidney injury after cardiac surgery?"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.artnumber","55"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","BMC Anesthesiology"],["dc.bibliographiccitation.volume","19"],["dc.contributor.author","Bräuer, Anselm"],["dc.contributor.author","Müller, Michaela M."],["dc.contributor.author","Wetz, Anna J."],["dc.contributor.author","Quintel, Michael"],["dc.contributor.author","Brandes, Ivo F."],["dc.date.accessioned","2019-07-09T11:51:12Z"],["dc.date.available","2019-07-09T11:51:12Z"],["dc.date.issued","2019"],["dc.description.abstract","Background Perioperative hypothermia is still very common and associated with numerous adverse effects. The effects of benzodiazepines, administered as premedication, on thermoregulation have been studied with conflicting results. We investigated the hypotheses that premedication with flunitrazepam would lower the preoperative core temperature and that prewarming could attenuate this effect. Methods After approval by the local research ethics committee 50 adult cardiac surgical patients were included in this prospective, randomized, controlled, single-centre study with two parallel groups in a university hospital setting. Core temperature was measured using a continuous, non-invasive zero-heat flux thermometer from 30 min before administration of the oral premedication until beginning of surgery. An equal number of patients was randomly allocated via a computer-generated list assigning them to either prewarming or control group using the sealed envelope method for blinding. The intervention itself could not be blinded. In the prewarming group patients received active prewarming using an underbody forced-air warming blanket. The data were analysed using Student’s t-test, Mann-Whitney U-test and Fisher’s exact test. Results Of the randomized 25 patients per group 24 patients per group could be analysed. Initial core temperature was 36.7 ± 0.2 °C and dropped significantly after oral premedication to 36.5 ± 0.3 °C when the patients were leaving the ward and to 36.4 ± 0.3 °C before induction of anaesthesia. The patients of the prewarming group had a significantly higher core temperature at the beginning of surgery (35.8 ± 0.4 °C vs. 35.5 ± 0.5 °C, p = 0.027), although core temperature at induction of anaesthesia was comparable. Despite prewarming, core temperature did not reach baseline level prior to premedication (36.7 ± 0.2 °C). Conclusions Oral premedication with benzodiazepines on the ward lowered core temperature significantly at arrival in the operating room. This drop in core temperature cannot be offset by a short period of active prewarming. Trial registration This trial was prospectively registered with the German registry of clinical trials under the trial number DRKS00005790 on 20th February 2014."],["dc.format.extent","7"],["dc.identifier.doi","10.1186/s12871-019-0725-7"],["dc.identifier.pmid","30987594"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/16070"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/59895"],["dc.language.iso","en"],["dc.notes.intern","Merged from goescholar"],["dc.publisher","BioMed Central"],["dc.rights","CC BY 4.0"],["dc.rights.uri","https://creativecommons.org/licenses/by/4.0"],["dc.title","Influence of oral premedication and prewarming on core temperature of cardiac surgical patients: a prospective, randomized, controlled trial"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","587"],["dc.bibliographiccitation.issue","5"],["dc.bibliographiccitation.journal","Anaesthesia and Intensive Care"],["dc.bibliographiccitation.lastpage","592"],["dc.bibliographiccitation.volume","44"],["dc.contributor.author","Popov, A. F."],["dc.contributor.author","Schotola, Hanna"],["dc.contributor.author","Wetz, Anna J."],["dc.contributor.author","Bergmann, I."],["dc.contributor.author","Danner, Bernhard Christoph"],["dc.contributor.author","Schoendube, Friedrich Albert"],["dc.contributor.author","Bauer, M."],["dc.contributor.author","Braeuer, Anselm"],["dc.date.accessioned","2020-12-10T18:38:26Z"],["dc.date.available","2020-12-10T18:38:26Z"],["dc.date.issued","2016"],["dc.description.abstract","At the end of cardiopulmonary bypass, there are invariably several hundred millilitres of residual pump blood in the reservoir, which can either be re-transfused or discarded. The objective of this prospective observational study was to investigate the quality of the residual pump blood, focusing on plasma free haemoglobin (pfHb) and blood cell counts. Fifty-one consecutive patients were included in the study. Forty-nine units of residual pump blood and 58 units of transfused red blood cell (RBC) concentrates were analysed. The mean preoperative pfHb of the patients was 0.057 +/- 0.062 g/l, which increased gradually to 0.55 +/- 0.36 g/l on arrival in the intensive care unit postoperatively. On the first postoperative day, the mean pfHb had returned to within the normal range. Our data showed that haemoglobin, haematocrit, and erythrocyte counts of residual pump blood were approximately 40% of the values in standardised RBC concentrates. Plasma free haemoglobin was significantly higher in residual pump blood compared to RBC concentrates, and nearly twice as high as the pfHb in patient blood samples taken contemporaneously. Our findings indicate that residual pump blood pfHb levels are markedly higher compared to patients' blood and RBC concentrates, but that its administration does not significantly increase patients' pfHb levels."],["dc.identifier.doi","10.1177/0310057X1604400519"],["dc.identifier.eissn","1448-0271"],["dc.identifier.isi","000390616500010"],["dc.identifier.issn","0310-057X"],["dc.identifier.pmid","27608341"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/77316"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Australian Soc Anaesthetists"],["dc.relation.issn","1448-0271"],["dc.relation.issn","0310-057X"],["dc.title","The Effects of Residual Pump Blood on Patient Plasma Free Haemoglobin Levels Post Cardiac Surgery"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","406"],["dc.bibliographiccitation.issue","5"],["dc.bibliographiccitation.journal","Der Anaesthesist"],["dc.bibliographiccitation.lastpage","414"],["dc.bibliographiccitation.volume","63"],["dc.contributor.author","Braeuer, Anselm"],["dc.contributor.author","Brandes, Ivo Florian"],["dc.contributor.author","Perl, Tal Naggan"],["dc.contributor.author","Wetz, Anna J."],["dc.contributor.author","Bauer, M."],["dc.date.accessioned","2018-11-07T09:40:39Z"],["dc.date.available","2018-11-07T09:40:39Z"],["dc.date.issued","2014"],["dc.description.abstract","Prewarming is a useful and effective measure to reduce perioperative hypothermia. Due to A 23(3) of the German Infektionsschutzgesetz (Gesetz zur Verhutung und Bekampfung von Infektionskrankheiten beim Menschen, Infection Act, act on protection and prevention of infectious diseases in man) and the recommendations of the Hospital Hygiene and Infection Prevention Committee of the Robert Koch Institute, implementation of prewarming is clearly recommended. There are several technically satisfactory and practicable devices available allowing prewarming on the normal hospital ward, in the preoperative holding area or in the induction room of the operating theater (OR) The implementation of prewarming requires additional equipment and training of staff. Using a locally adapted concept for the implementation of prewarming does not lead to inefficiency in the perioperative process. In contrast, the implementation can help to achieve stable arrival times for patients in the OR."],["dc.identifier.doi","10.1007/s00101-014-2316-9"],["dc.identifier.isi","000336326100006"],["dc.identifier.pmid","24691948"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/33550"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.relation.issn","1432-055X"],["dc.relation.issn","0003-2417"],["dc.title","Prewarming. Yesterday's luxury, today's minimum requirements"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","179"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Clinical and Applied Thrombosis, Hemostasis"],["dc.bibliographiccitation.lastpage","185"],["dc.bibliographiccitation.volume","24"],["dc.contributor.author","Wand, Saskia"],["dc.contributor.author","Adam, Elisabeth Hannah"],["dc.contributor.author","Wetz, Anna Julienne"],["dc.contributor.author","Meybohm, Patrick"],["dc.contributor.author","Kunze-Szikszay, Nils"],["dc.contributor.author","Zacharowski, Kai"],["dc.contributor.author","Popov, Aron Frederick"],["dc.contributor.author","Moritz, Anton"],["dc.contributor.author","Moldenhauer, Lisa"],["dc.contributor.author","Kaiser, Julia"],["dc.contributor.author","Bauer, Martin"],["dc.contributor.author","Weber, Christian Friedrich"],["dc.date.accessioned","2020-12-10T18:38:30Z"],["dc.date.available","2020-12-10T18:38:30Z"],["dc.date.issued","2017"],["dc.identifier.doi","10.1177/1076029617693939"],["dc.identifier.eissn","1938-2723"],["dc.identifier.issn","1076-0296"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/77342"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","The Prevalence and Clinical Relevance of ASA Nonresponse After Cardiac Surgery: A Prospective Bicentric Study"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]