Bodemer, Walter Wolfgang

[["dc.bibliographiccitation.firstpage","2433"],["dc.bibliographiccitation.issue","10"],["dc.bibliographiccitation.journal","Journal of General Virology"],["dc.bibliographiccitation.lastpage","2436"],["dc.bibliographiccitation.volume","77"],["dc.contributor.author","Dittmer, U."],["dc.contributor.author","Petry, H."],["dc.contributor.author","Stahl-Hennig, C."],["dc.contributor.author","Nisslein, T."],["dc.contributor.author","Spring, M."],["dc.contributor.author","Luke, W."],["dc.contributor.author","Bodemer, W."],["dc.contributor.author","Kaup, F.-J."],["dc.contributor.author","Hunsmann, G."],["dc.date.accessioned","2022-10-06T13:24:54Z"],["dc.date.available","2022-10-06T13:24:54Z"],["dc.date.issued","1996"],["dc.identifier.doi","10.1099/0022-1317-77-10-2433"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/114699"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-602"],["dc.relation.eissn","1465-2099"],["dc.relation.issn","0022-1317"],["dc.relation.orgunit","Deutsches Primatenzentrum"],["dc.title","T cell apoptosis in human immunodeficiency virus type 2- and simian immunodeficiency virus-infected macaques"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","105"],["dc.bibliographiccitation.issue","1-3"],["dc.bibliographiccitation.journal","Journal of Biotechnology"],["dc.bibliographiccitation.lastpage","110"],["dc.bibliographiccitation.volume","44"],["dc.contributor.author","Dittmer, U."],["dc.contributor.author","Spring, M."],["dc.contributor.author","Petry, H."],["dc.contributor.author","Nisslein, T."],["dc.contributor.author","Rieckmann, P."],["dc.contributor.author","Lüke, W."],["dc.contributor.author","Stahl-Hennig, C."],["dc.contributor.author","Hunsmann, G."],["dc.contributor.author","Bodemer, W."],["dc.date.accessioned","2022-10-06T13:32:48Z"],["dc.date.available","2022-10-06T13:32:48Z"],["dc.date.issued","1996"],["dc.identifier.doi","10.1016/0168-1656(95)00160-3"],["dc.identifier.pii","0168165695001603"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/115456"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-602"],["dc.relation.issn","0168-1656"],["dc.relation.orgunit","Deutsches Primatenzentrum"],["dc.title","Cell-mediated immune response of macaques immunized with low doses of simian immunodeficiency virus (SIV)"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","S32"],["dc.bibliographiccitation.issue","Suppl. 2"],["dc.bibliographiccitation.journal","Blood Coagulation & Fibrinolysis"],["dc.bibliographiccitation.lastpage","S35"],["dc.bibliographiccitation.volume","6"],["dc.contributor.author","Bodemer, W."],["dc.contributor.author","Zedler, U."],["dc.contributor.author","Makoschey, B."],["dc.contributor.author","Hunsmann, G."],["dc.date.accessioned","2022-10-06T13:35:25Z"],["dc.date.available","2022-10-06T13:35:25Z"],["dc.date.issued","1995"],["dc.description.abstract","At present, the hepatitis A virus cannot be detected readily by cell culture techniques. However, the New World primateSaguinus fuscicolisis particularly susceptible to this virus. This paper gives details of thein vivodetection of hepatitis A virus inS. fuscicolis,and the method is used to show that a blood factor VIII preparation, suspected of being contaminated with hepatitis A, did not contain the virus."],["dc.identifier.doi","10.1097/00001721-199506002-00008"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/116092"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-602"],["dc.relation.issn","0957-5235"],["dc.relation.orgunit","Deutsches Primatenzentrum"],["dc.title","Detection of infectious hepatitis A virus in blood factor concentrates by experimental infection of the New World primate Saguinus fuscicollis"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.subtype","original_ja"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","132"],["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","Neoplasia"],["dc.bibliographiccitation.lastpage","142"],["dc.bibliographiccitation.volume","3"],["dc.contributor.author","Tarantul, V."],["dc.contributor.author","Nikolaev, A."],["dc.contributor.author","Hannig, H."],["dc.contributor.author","Kalmyrzaev, B."],["dc.contributor.author","Muchoyan, I."],["dc.contributor.author","Maximov, V."],["dc.contributor.author","Nenasheva, V."],["dc.contributor.author","Dubovaya, V."],["dc.contributor.author","Hunsmann, G."],["dc.contributor.author","Bodemer, W."],["dc.date.accessioned","2019-07-09T11:40:45Z"],["dc.date.available","2019-07-09T11:40:45Z"],["dc.date.issued","2001"],["dc.description.abstract","Several novel, differentially transcribed genes were identified in one centroblastic and one immunoblastic HIV-associated B-cell non-Hodgkin's lymphoma (BNHL) by subtractive cloning. In both lymphomas, we detected an upregulated transcription of several mitochondrial genes. In the centroblastic B-NHL, we found a high level transcription of nuclear genes including the interferon-inducible gene (INF-ind), the immunoglobulin light chain gene (IgL), the set oncogene, and several unknown genes. The data obtained on upregulated expression of the genes in human B-NHL of HIV-infected patients considerably overlap with those obtained earlier for the B-NHL of simian immunodeficiency virus-infected monkeys. In the centroblastic lymphoma, one transcript revealed a fusion of the 3'-untranslated region of the set gene and the C-terminal region of the IgL gene. This chimeric sequence was confirmed by a site-directed polymerase chain reaction performed with total cDNA and genomic DNA. The expected amplification product was obtained in both cases pointing to a genomic rearrangement. The IgL-set fusion sequence was not found in cDNA preparations and genomic DNA of the immunoblastic HIV-associated B-NHL. Further studies are necessary to determine whether these genes contribute to lymphoma development or can be used as therapeutic targets."],["dc.identifier.doi","10.1038/sj.neo.7900137"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/11236"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/58238"],["dc.language.iso","en"],["dc.notes.intern","Merged from goescholar"],["dc.rights","Goescholar"],["dc.rights.uri","https://goescholar.uni-goettingen.de/licenses"],["dc.title","Detection of Abundantly Transcribed Genes and Gene Translocation in Human Immunodeficiency Virus-Associated Non—Hodgkin's Lymphoma"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","403"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","Journal of Acquired Immune Deficiency Syndromes"],["dc.bibliographiccitation.lastpage","407"],["dc.bibliographiccitation.volume","20"],["dc.contributor.author","Miller, M."],["dc.contributor.author","Shohat, B."],["dc.contributor.author","Shaklai, M."],["dc.contributor.author","Ron, D."],["dc.contributor.author","Rapaport, L."],["dc.contributor.author","Gordon, C."],["dc.contributor.author","Kott, E."],["dc.contributor.author","Bodemer, W."],["dc.contributor.author","Hannig, H."],["dc.contributor.author","Hunsmann, G."],["dc.date.accessioned","2022-10-06T13:35:31Z"],["dc.date.available","2022-10-06T13:35:31Z"],["dc.date.issued","1999"],["dc.description.abstract","We tested the possibility that lymphocytes and serum obtained directly from a patient with HTLV-I-associated myelopathy/tropical spastic paraparesis (HAM/TSP) induce infection in rats. Inbred Fischer F344 immunosupressed rats were inoculated intravenously with 10 × 106 peripheral blood mononuclear cells (PBMC; 3 rats) and serum (3 rats) obtained from a HAM/TSP patient, who was seropositive and polymerase chain reaction (PCR)-positive for the HTLV-I proviral genome. Antibodies to HTLV-I appeared in the rat sera 2 months later; rat peripheral blood lymphocytes, spleen, salivary gland, and spinal cord were found to contain the proviral genome. Control rats inoculated with normal donor PBMC and serum tested negative for the HTLV-I antibodies and for the HTLV-I proviral genome by PCR. The positive control F344 rats inoculated with 5 × 106 cells of a SLB-1 HTLV-I cell line were found to be infected after 2 months. This study demonstrates for the first time that HTLV-I can be transmitted not only by human cellular components but also by human cell-free sera in a rat model."],["dc.identifier.doi","10.1097/00042560-199904010-00012"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/116116"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-602"],["dc.relation.issn","1077-9450"],["dc.relation.orgunit","Deutsches Primatenzentrum"],["dc.title","Transmission of HTLV-I to Rats via Peripheral Blood Mononuclear Cells and Serum From a Patient With HTLV-I-Associated Myelopathy/Tropical Spastic Paraparesis (HAM/TSP) With Amyotrophic Lateral Sclerosis (ALS)-Like Features"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dc.type.subtype","original_ja"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","173"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Molecular Brain Research"],["dc.bibliographiccitation.lastpage","176"],["dc.bibliographiccitation.volume","34"],["dc.contributor.author","Krasemann, S."],["dc.contributor.author","Zerr, I."],["dc.contributor.author","Weber, T."],["dc.contributor.author","Poser, S."],["dc.contributor.author","Kretzschmar, H."],["dc.contributor.author","Hunsmann, G."],["dc.contributor.author","Bodemer, W."],["dc.date.accessioned","2022-10-06T13:32:49Z"],["dc.date.available","2022-10-06T13:32:49Z"],["dc.date.issued","1995"],["dc.identifier.doi","10.1016/0169-328X(95)00175-R"],["dc.identifier.pii","0169328X9500175R"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/115459"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-602"],["dc.relation.issn","0169-328X"],["dc.relation.orgunit","Deutsches Primatenzentrum"],["dc.title","Prion disease associated with a novel nine octapeptide repeat insertion in the PRNP gene"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","114"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Journal of Medical Virology"],["dc.bibliographiccitation.lastpage","120"],["dc.bibliographiccitation.volume","65"],["dc.contributor.author","Blaschke, S."],["dc.contributor.author","Hannig, H."],["dc.contributor.author","Buske, Christian"],["dc.contributor.author","Kaup, F. J."],["dc.contributor.author","Hunsmann, G."],["dc.contributor.author","Bodemer, W."],["dc.date.accessioned","2018-11-07T08:43:06Z"],["dc.date.available","2018-11-07T08:43:06Z"],["dc.date.issued","2001"],["dc.description.abstract","During the course of simian immunodeficiency virus (SIV) infection, nearly 15% of rhesus macaques (Macaca mulatta) and up to 40% of cynomolgus macaques (Macaca fascicularis) developed SIV-associated non-Hodgkin's lymphomas. Most of these malignant lymphomas harbored lymphocryptoviruses, which are closely related to the human Epstein-Barr virus (EBV; Herpesvirus M. mulatta and Herpesvirus M. fascicularis). To characterize the oncogenic role of simian EBV infection for lymphomagenesis during SIV infection, expression of the EBV-encoded latent membrane protein-1 (LMP-1) was analyzed in malignant lymphomas of SIV-infected rhesus macaques. Nine seropositive rhesus macaques suffering from B-cell lymphomas during the late phase of SIV infection were euthanized. Latency stages of EBV infection within malignant lymphomas and simian EBV-infected lymphoblastoid cell lines (LCL8664, H50) were characterized by analyzing expression of the EBV-encoded nuclear antigens EBNA-1, EBNA-2, and small RNAs EBER1/2. In parallel, the presence of viral LMP-1 transcripts was assessed by reverse transcription-polymerase chain reaction (RT-PCR) and in situ hybridization. Results were compared with findings in AIDS-associated malignant lymphomas in two patients infected with human immunodeficiency virus (HIV)-1. Rhesus macaques developed high-grade B-cell lymphomas of the centroblastic (five of nine), immunoblastic (two of nine), centroblastic-centrocytic (one of nine), and Burkitt-like (one of nine) subtypes within 18-29 months postinfection with SIV(mac)251/32H. The presence of Herpesvirus M. mulatta was detected in eight of nine cases. Transcription of the viral oncogene LMP-1 could be demonstrated within the simian EBV-infected cell lines as well as in four of nine SIV-associated malignant lymphomas. These four cases and both of the HIV-1-related non-Hodgkin's lymphomas expressed the full spectrum of latent EBV gene products (LMP-1, EBER1/2, EBNA-1, EBNA-2) and were thus classified as latency type III stages of EBV infection. Simian EBV infection was demonstrated in 90% of lymphomas in SIV-infected rhesus macaques. Analysis of LMP-1 expression suggests an important role for this viral oncogene in the pathogenesis of both SIV and HIV-1-associated malignant lymphomas. (C) 2001 Wiley-Liss, Inc."],["dc.identifier.doi","10.1002/jmv.2009"],["dc.identifier.isi","000170257200017"],["dc.identifier.pmid","11505452"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/19879"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.relation.issn","0146-6615"],["dc.title","Expression of the simian Epstein-Barr virus-encoded latent membrane protein-1 in malignant lymphomas of SIV-infected rhesus macaques"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","269"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","Journal of Medical Virology"],["dc.bibliographiccitation.lastpage","274"],["dc.bibliographiccitation.volume","56"],["dc.contributor.author","Miller, M."],["dc.contributor.author","Achiron, A."],["dc.contributor.author","Shaklai, M."],["dc.contributor.author","Stark, P."],["dc.contributor.author","Maayan, S."],["dc.contributor.author","Hannig, H."],["dc.contributor.author","Hunsmann, G."],["dc.contributor.author","Bodemer, W."],["dc.contributor.author","Shohat, B."],["dc.date.accessioned","2022-10-06T13:24:50Z"],["dc.date.available","2022-10-06T13:24:50Z"],["dc.date.issued","1998"],["dc.identifier.doi","10.1002/(SICI)1096-9071(199811)56:3<269::AID-JMV16>3.0.CO;2-9"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/114688"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-602"],["dc.relation.eissn","1096-9071"],["dc.relation.issn","0146-6615"],["dc.relation.orgunit","Deutsches Primatenzentrum"],["dc.title","Ethnic cluster of HTLV-I infection in Israel among the Mashhadi Jewish population"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1307"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","Journal of General Virology"],["dc.bibliographiccitation.lastpage","1315"],["dc.bibliographiccitation.volume","76"],["dc.contributor.author","Dittmer, U."],["dc.contributor.author","Stahl-Hennig, C."],["dc.contributor.author","Coulibaly, C."],["dc.contributor.author","Nisslein, T."],["dc.contributor.author","Luke, W."],["dc.contributor.author","Fuchs, D."],["dc.contributor.author","Bodemer, W."],["dc.contributor.author","Petry, H."],["dc.contributor.author","Hunsmann, G."],["dc.date.accessioned","2022-10-06T13:24:53Z"],["dc.date.available","2022-10-06T13:24:53Z"],["dc.date.issued","1995"],["dc.identifier.doi","10.1099/0022-1317-76-6-1307"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/114698"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-602"],["dc.relation.eissn","1465-2099"],["dc.relation.issn","0022-1317"],["dc.relation.orgunit","Deutsches Primatenzentrum"],["dc.title","Repeated exposure of rhesus macaques to low doses of simian immunodeficiency virus (SIV) did not protect them against the consequences of a high-dose SIV challenge"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dspace.entity.type","Publication"]]