Banaschewski, Tobias

[["dc.bibliographiccitation.firstpage","137"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","Kindheit und Entwicklung"],["dc.bibliographiccitation.lastpage","147"],["dc.bibliographiccitation.volume","13"],["dc.contributor.author","Banaschewski, Tobias"],["dc.contributor.author","Roessner, Veit"],["dc.contributor.author","Uebel, Henrik"],["dc.contributor.author","Rothenberger, A."],["dc.date.accessioned","2018-11-07T10:52:53Z"],["dc.date.available","2018-11-07T10:52:53Z"],["dc.date.issued","2004"],["dc.description.abstract","Attention-deficit/hyperactivity disorder (ADHD) is an early-onset, clinically and presumably etiologically heterogeneous disorder characterized by inattention, hyperactivity, and impulsivity. Although the specific neurobiological basis of ADHD remains unknown, research in the fields of genetics, adverse environmental factors, neuropsychology, psychophysiology, neuroimaging, and neurochemistry has made substantial progress and several models of the underlying pathophysiology have been suggested. This comprehensive review summarizes the relevant findings in these various areas and provides an update of current knowledge concerning the pathophysiology of ADHD."],["dc.identifier.doi","10.1026/0942-5403.13.3.137"],["dc.identifier.isi","000222453700002"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/49218"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Hogrefe & Huber Publishers"],["dc.relation.issn","0942-5403"],["dc.title","Neurobiology of attention-deficit/hyperactivity disorder"],["dc.type","review"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1"],["dc.bibliographiccitation.journal","European Child & Adolescent Psychiatry"],["dc.bibliographiccitation.lastpage","4"],["dc.bibliographiccitation.volume","16"],["dc.contributor.author","Rothenberger, Aribert"],["dc.contributor.author","Roessner, Veit"],["dc.contributor.author","Banaschewski, Tobias"],["dc.contributor.author","Leckman, James F."],["dc.date.accessioned","2018-11-07T11:01:45Z"],["dc.date.available","2018-11-07T11:01:45Z"],["dc.date.issued","2007"],["dc.description.abstract","In daily clinical practice of child psychiatry tic disorders (TD) and attention-deficit/ hyperactivity disorder (ADHD) as well as their co-existence are common and need careful evidence-based approaches in differential diagnostics and treatment choice. Hence, their relationship at different levels (e.g., psychopathology, neuropsychology, neurobiology, treatment) needs to be elucidated in more detail. The articles of this supplement deal with this subject. It is unclear if TD + ADHD represents an own clinical entity and which component (TD vs. ADHD) is most important in case of co-existence with respect to accompanying problems like emotional or cognitive disturbances. This editorial gives a short overview of the essential research topics concerning TD + ADHD and shows where (in this ECAP supplement and other literature) empirically based answers can be found. Probably, the identification of separate or common \"endophenotypes\" for TD-only, ADHD-only and for TD + ADHD would help to sort out the complexities and this supplement may nurture such a perspective of future research for a better understanding and treatment of youths with TD + ADHD and their families."],["dc.identifier.doi","10.1007/s00787-007-1001-9"],["dc.identifier.isi","000248689900001"],["dc.identifier.pmid","17665277"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/51219"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Dr Dietrich Steinkopff Verlag"],["dc.relation.issn","1018-8827"],["dc.title","Co-existence of tic disorders and attention-deficit/hyperactivity disorder-recent advances in understanding and treatment"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.subtype","letter_note"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","167"],["dc.bibliographiccitation.issue","2-3"],["dc.bibliographiccitation.journal","Behavioral and Brain Sciences"],["dc.bibliographiccitation.lastpage","+"],["dc.bibliographiccitation.volume","33"],["dc.contributor.author","Rothenberger, Aribert"],["dc.contributor.author","Banaschewski, Tobias"],["dc.contributor.author","Becker, Andreas"],["dc.contributor.author","Roessner, Veit"],["dc.date.accessioned","2018-11-07T08:44:20Z"],["dc.date.available","2018-11-07T08:44:20Z"],["dc.date.issued","2010"],["dc.description.abstract","In developmental psychopathology, differentiating between the coexistence and the clinical entity of two problem areas is of utmost importance. So far, logistic regression analysis has already provided helpful answers, as shown in studies on comorbidity of tic disorders. While the concept of bridging symptoms may be investigated adequately by both logistic regression and the network approach, the former (latent variable) seems to be of advantage with regard to the problems of multiple comorbidities and development."],["dc.identifier.doi","10.1017/S0140525X10000683"],["dc.identifier.isi","000279973200049"],["dc.identifier.pmid","20584387"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/20174"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.relation.issn","0140-525X"],["dc.title","Comorbidity: The case of developmental psychopathology"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","5"],["dc.bibliographiccitation.journal","European Child & Adolescent Psychiatry"],["dc.bibliographiccitation.lastpage","14"],["dc.bibliographiccitation.volume","16"],["dc.contributor.author","Banaschewski, Tobias"],["dc.contributor.author","Neale, Benjamin M."],["dc.contributor.author","Rothenberger, Aribert"],["dc.contributor.author","Roessner, Veit"],["dc.date.accessioned","2018-11-07T11:01:45Z"],["dc.date.available","2018-11-07T11:01:45Z"],["dc.date.issued","2007"],["dc.description.abstract","Background The causes and pathophysiological mechanisms of the common comorbidity of tic disorders and Attention-deficit/Hyperactivity disorder (TD + ADHD; about 50% of TD, about 20% of ADHD) still remain unclear. Studies with a 2 x 2 design comparing groups of children with TD, ADHD, TD + ADHD and healthy controls are in need and may disentangle the influences of TD versus ADHD in the case of comorbidity. Unfortunately, conceptual and methodological problems can restrain possible conclusions from these studies. Method A review of the literature on artifactual and non-artifactual models of comorbidity in general and specially for TD + ADHD was conducted. Results The first section describes various possible models of comorbidity and their corresponding hypotheses concerning expected patterns of findings comparing groups of children with TD, ADHD, TD + ADHD and healthy controls. In the second part re- search results concerning psychopathological, neuropsychological, neurophysiological, structural and functional imaging, as well as genetic characteristics are summarized. In the third section possible conclusions and their limitations due to conceptual and methodological problems possibly contributing to the ambiguous results are discussed. Finally, future research strategies and the need for full causal models are outlined. Conclusion Some components of the etiological pathways of TD + ADHD may well be shared with the 'pure' conditions while others may be unique."],["dc.identifier.doi","10.1007/S00787-007-1002-8"],["dc.identifier.isi","000248689900002"],["dc.identifier.pmid","17665278"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/51220"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Dr Dietrich Steinkopff Verlag"],["dc.relation.issn","1018-8827"],["dc.title","Comorbidity of tic disorders & ADHD - Conceptual and methodological considerations"],["dc.type","review"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","627"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","Behavioral and Brain Sciences"],["dc.bibliographiccitation.lastpage","+"],["dc.bibliographiccitation.volume","29"],["dc.contributor.author","Rothenberger, Aribert"],["dc.contributor.author","Roessner, Veit"],["dc.contributor.author","Banaschewski, Tobias"],["dc.date.accessioned","2018-11-07T08:51:55Z"],["dc.date.available","2018-11-07T08:51:55Z"],["dc.date.issued","2006"],["dc.description.abstract","Tourette Syndrome (TS) and Obsessive-Compulsive Disorder (OCD) are highly associated and often it is difficult to differentiate their symptomatology. In TS, habit forming neuronal systems may form habits of their own - sometimes similar to ritualized behavior. However, whereas in OCD merely the \"affect-loop\" is touched, in TS the \"sensorimotor-loop\" plays the major role, although some overlap can be seen in the clinical spectrum between TS and OCD. The latter is mainly related to the \"just-right\" phenomenon which shows a clear developmental course. An analogous behavioral model for TS and OCD with reference to \"just-right\" is suggested."],["dc.identifier.doi","10.1017/S0140525X06009484"],["dc.identifier.isi","000244676500037"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/22047"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Cambridge Univ Press"],["dc.relation.issn","0140-525X"],["dc.title","Habit formation in Tourette Syndrome with associated obsessive-compulsive behavior: At the crossroads of neurobiological modelling"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.subtype","letter_note"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","36"],["dc.bibliographiccitation.journal","European Child & Adolescent Psychiatry"],["dc.bibliographiccitation.lastpage","44"],["dc.bibliographiccitation.volume","16"],["dc.contributor.author","Roessner, Veit"],["dc.contributor.author","Becker, Andreas"],["dc.contributor.author","BanascheWski, Tobias"],["dc.contributor.author","Rothenberger, Aribert"],["dc.date.accessioned","2018-11-07T11:01:46Z"],["dc.date.available","2018-11-07T11:01:46Z"],["dc.date.issued","2007"],["dc.description.abstract","Objective In Chronic Tic Disorders (CTD) associated Attention Deficit Hyperactivity Disorder (ADHD) is very common. Hence, it is important to clarify how both conditions are related to cognitive dysfunctions in patients with CTD+ADHD comorbidity. Recent studies on neuropsychology revealed equivocal results, mostly due to methodological shortcomings like problems in sample composition. Thus better and more detailed information on this topic is needed to improve diagnostic and treatment approaches. Method Three tasks related to different domains of executive functions (the Matching Familiar Figures Test, the Stroop color-word interference task, and a computerized version of the Wisconsin Card Sorting Test) have been performed in two independent samples (altogether n = 138 children) both including four groups of children (CTD-only, CTD+ADHD, ADHD-only, healthy controls) matched for age and IQ. To specify the influence of either tics or ADHD-symptoms on executive functions and to answer the question of their interactive or additive relationship two-way analyses of variance (MANOVA) for the factors CTD (yes,no) x ADHD (yes,no) were conducted. Eta squared was calculated to reveal the effect sizes for each factor. For a deeper understanding of group differences and to better enable the comparison with data in literature, additional analyses of variance (ANOVA) with posthoc testing were applied. Results In summary, there was a main effect only for the factor ADHD reflected by decreased performance, while no main effect of the factor CTD could be found. Admittedly, the effects were not uniform in both samples. However, in all three tasks and both samples, uniformly no interaction between the main factors has been observed. Conclusions In cases of coexisting CTD+ADHD the factor ADHD shows the main negative impact on neuropsychological performance and this impact seems to be independent of any feature of the coexisting tics (additive model). This supports the notion to primarily treat the ADHD-symptoms in order to increase cognitive self regulatory abilities of these children. Contrarily, tics seem to have little impact on cognitive performance in most cases."],["dc.identifier.doi","10.1007/s00787-007-1005-5"],["dc.identifier.isi","000248689900005"],["dc.identifier.pmid","17665281"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/51223"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Dr Dietrich Steinkopff Verlag"],["dc.relation.issn","1018-8827"],["dc.title","Executive functions in children with chronic tic disorders with/without AND: new insights"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","6"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","European Archives of Psychiatry and Clinical Neuroscience"],["dc.bibliographiccitation.lastpage","9"],["dc.bibliographiccitation.volume","255"],["dc.contributor.author","Roessner, Veit"],["dc.contributor.author","Bleich, Stefan"],["dc.contributor.author","Banaschewski, Tobias"],["dc.contributor.author","Rothenberger, A."],["dc.date.accessioned","2018-11-07T08:27:09Z"],["dc.date.available","2018-11-07T08:27:09Z"],["dc.date.issued","2005"],["dc.description.abstract","Objective Young patients admitted to the hospital due to anorexia nervosa report reduced pleasure and impaired perception of smell while eating. So far, two studies on odour identification ability in eating disorders did not suggest any significant deficits. Therefore a new and more detailed method of olfactory testing may be needed, in order to determine the subjective impairment of olfaction. Method By using all three subtests of the recently developed smell test called \"Sniffin' Sticks\", the olfactory deficits were assessed in more detail and the results of female anorectic patients ( n = 17) were compared with those of healthy females ( n = 15). Results By examining the anorectic patients no deficits in the subtest odour identification were found. On the contrary, in the subtests odour discrimination and odour threshold deficits of the anorectic patients were detected. Conclusion Reduced olfactory perception might be considered as a common deficit in anorexia nervosa with possible influences and consequences for therapy."],["dc.identifier.doi","10.1007/s00406-004-0525-y"],["dc.identifier.isi","000227045700002"],["dc.identifier.pmid","15538597"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/16159"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Dr Dietrich Steinkopff Verlag"],["dc.relation.issn","0940-1334"],["dc.title","Olfactory deficits in anorexia nervosa"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","71"],["dc.bibliographiccitation.journal","European Child & Adolescent Psychiatry"],["dc.bibliographiccitation.lastpage","79"],["dc.bibliographiccitation.volume","13"],["dc.contributor.author","Roessner, Veit"],["dc.contributor.author","Banaschewski, Tobias"],["dc.contributor.author","Uebel, Henrik"],["dc.contributor.author","Becker, A."],["dc.contributor.author","Rothenberger, A."],["dc.date.accessioned","2018-11-07T10:47:35Z"],["dc.date.available","2018-11-07T10:47:35Z"],["dc.date.issued","2004"],["dc.description.abstract","Background. Recent research on structural and functional anatomy related to ADHD has concentrated on fronto-striatocerebellar circuits. These findings and resultant neurobiological models of ADHD may explain some of the disturbances. On the other hand, there is some evidence that the restricted look at anteriorposterior anomalies might neglect the important additional information of lateralization problems related to hemispheric connectivity. Objective. Hence, the role of lateralization in the pathophysiology of ADHD should be reconsidered. Method. After a short review of imaging studies in ADHD the special role of the corpus callosum (including the influence of its anomalies on general brain development, lateralization and functioning) is outlined and the first case of total agenesis of corpus callosum in a child with ADHD is presented and discussed within this context. Conclusions. In view of the remaining inconsistencies concerning structural and functional brain anomalies in ADHD, attention should be paid not only to anterior- posterior but also to leftright aspects of functional and structural brain anomalies. This should include disturbances probably related to anomalies of the corpus callosum, especially in regard to co-existing problems like dyslexia and developmental coordination disorder."],["dc.identifier.doi","10.1007/s00787-004-1007-5"],["dc.identifier.isi","000223466500007"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/47995"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.relation.issn","1018-8827"],["dc.title","Neuronal network models of ADHD - lateralization with respect to interhemispheric connectivity reconsidered"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","53"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Psychopathology"],["dc.bibliographiccitation.lastpage","59"],["dc.bibliographiccitation.volume","44"],["dc.contributor.author","Holtmann, Martin"],["dc.contributor.author","Becker, Andreas"],["dc.contributor.author","Banaschewski, Tobias"],["dc.contributor.author","Rothenberger, Aribert"],["dc.contributor.author","Roessner, Veit"],["dc.date.accessioned","2018-11-07T09:01:59Z"],["dc.date.available","2018-11-07T09:01:59Z"],["dc.date.issued","2011"],["dc.description.abstract","Background: In many severely mentally disordered children, the clinical presentation is complicated by comorbid affective and behavioral dysregulation. Recently, a highly heritable behavioral phenotype of simultaneous deviance on the nanxious/depressed, attention problems, and aggressive behavior syndrome scales has been identified on the Child Behavior Checklist Dysregulation Profile (CBCL-DP). The aim of the present pilot study was to determine an equivalent to the CBCL-DP using the Strengths and Difficulties Questionnaire (SDQ). Sampling and Methods: We applied stepwise linear discriminant analyses and receiver operating characteristic (ROC) analysis to data from 543 consecutively referred children and adolescents, aged 5-17 years. The CBCL and the SDQ were completed by parents as part of the diagnostic routine. ICD-10 discharge diagnoses were established in consensus conferences. Results: A combination of five SDQ items (SDQ-Dysregulation Profile, SDQ-DP) yielded the best discrimination of children with and without CBCL-DP and classified 81.0% of the subjects correctly leading to an area under the curve of 0.93. The content of the five SDQ-DP items mirrors well the mixed behavioral phenotype of anxious-depressive, aggressive and attention problems captured by the CBCL-DP. SDQ-DP status was highly correlated with CBCL-DP status and was best defined by a SDQ-DP score >= 5. Conclusions: The psychometric properties of the SDQ-DP have been robustly tested and validated. Based on these results, clinicians may use the SDQ-DP as a useful and economical screening measure to improve the assessment, prevention, and treatment of severe dysregulation in childhood and adolescence. Future investigations should study the longitudinal stability, heritability, and genetic associations of this behavioral phenotype. Copyright (C) 2010 S. Karger AG, Basel"],["dc.identifier.doi","10.1159/000318164"],["dc.identifier.isi","000284157300008"],["dc.identifier.pmid","21072000"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/8050"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/24567"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Karger"],["dc.relation.issn","0254-4962"],["dc.rights","Goescholar"],["dc.rights.uri","https://goescholar.uni-goettingen.de/licenses"],["dc.title","Psychometric Validity of the Strengths and Difficulties Questionnaire-Dysregulation Profile"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","352"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","European Archives of Psychiatry and Clinical Neuroscience"],["dc.bibliographiccitation.lastpage","359"],["dc.bibliographiccitation.volume","257"],["dc.contributor.author","Roessner, Veit"],["dc.contributor.author","Becker, Andreas"],["dc.contributor.author","Rothenberger, Aribert"],["dc.contributor.author","Rohde, Luis Augusto"],["dc.contributor.author","Banaschewski, Tobias"],["dc.date.accessioned","2018-11-07T10:58:52Z"],["dc.date.available","2018-11-07T10:58:52Z"],["dc.date.issued","2007"],["dc.description.abstract","This study aims to assess cross-cultural similarities and differences in broadband psychopathology in two naturalistic clinical samples of children diagnosed with Attention Deficit Hyperactivity Disorder (ADHD) combined type according to DSM-IV criteria or with Hyperkinetic Disorder (HD) according to ICD-10 criteria. We compared two clinical samples of children with ADHD combined type (Brazil, N = 248) and HD (Germany; N = 154) to controls (Brazil N = 71; Germany N = 135) using the Child Behavior Checklist (CBCL). ROC-curves (Receiver Operating Characteristic) were determined to evaluate the discriminating validity of the CBCL Attention Problem scale. A two-factorial ANOVA was computed across all 8 scales of the CBCL. Although Brazilian parents reported significantly higher scores on all CBCL scales than German parents (P < 0.05), a similar CBCL profile was detected in both cultures. Despite the use of different diagnostic systems (DSM-IV vs. ICD-10) and the presence of other clinical differences, the similar broadband psychopathological profile of the CBCL in the two samples provides evidence that dimensional symptoms associated with the categorical diagnosis of ADHD combined type might be comparable in two clinical settings with diverse cultural background."],["dc.identifier.doi","10.1007/s00406-007-0738-y"],["dc.identifier.isi","000249647300008"],["dc.identifier.pmid","17629732"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/50565"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Dr Dietrich Steinkopff Verlag"],["dc.relation.issn","0940-1334"],["dc.title","A cross-cultural comparison between samples of Brazilian and German children with ADHD/HD using the child behavior checklist"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]