Falkai, Peter Gaston

[["dc.bibliographiccitation.firstpage","126"],["dc.bibliographiccitation.journal","Brain Research"],["dc.bibliographiccitation.lastpage","134"],["dc.bibliographiccitation.volume","1301"],["dc.contributor.author","Parlapani, Eleni"],["dc.contributor.author","Schmitt, Andrea"],["dc.contributor.author","Erdmann, Andrea"],["dc.contributor.author","Bernstein, Hans-Gert"],["dc.contributor.author","Breunig, Barbara"],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Petroianu, Georg A."],["dc.contributor.author","von Wilmsdorff, Martina"],["dc.contributor.author","Schneider-Axmann, Thomas"],["dc.contributor.author","Honer, William G."],["dc.contributor.author","Falkai, Peter"],["dc.date.accessioned","2018-11-07T11:22:01Z"],["dc.date.available","2018-11-07T11:22:01Z"],["dc.date.issued","2009"],["dc.description.abstract","There is evidence for migrational disturbances in the entorhinal cortex (ERC) in schizophrenia that supports a neurodevelopmental origin of the disorder. Since impaired myelin basic protein (MBP) gene expression during the migration phase could lead to abnormalities in final laminar position, we performed layer specific measurements of MBP expression in the ERC and hypothesised that migrational disturbances of pre-alpha-cell clusters relate to decreased MBP expression. Paraffin embedded sections of the left entorhinal cortex of 16 schizophrenia patients and 10 control subjects were stained for MBP using routine immunohistochemistry. on each section representative regions of interest were scanned to attain optimal quality images of the gray matter. Results were correlated to previous published disturbed dispersion of pre-alpha-cell clusters in adjacent brain sections. Mean MBP stain-intensity was significantly reduced in schizophrenia patients. Absolute MBP stain-intensity was significantly reduced in layers III and IV in patients. A significant correlation of MBP stain-intensity with the distance of the deep pole of the pre-alpha-cell cluster from the gray white matter junction was observed in the ERC of schizophrenia patients. The present data provide evidence for reduced MBP expression in the ERC in schizophrenia, which implies deficits in axonal myelination and disturbed connectivity. MBP gene is expressed in oligodendrocytes and neuronal populations during embryonic development, which are important in establishing the structure of the cerebral cortex. Correlation between reduced MBP as a sign of down-regulated MBP gene expression and disorganization of pre-alpha-cell clusters supports a neurodevelopmental origin of pathological processes in schizophrenia. (C) 2009 Elsevier B.V. All rights reserved."],["dc.identifier.doi","10.1016/j.brainres.2009.09.007"],["dc.identifier.isi","000272100200014"],["dc.identifier.pmid","19747901"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/55908"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Elsevier Science Bv"],["dc.relation.issn","0006-8993"],["dc.title","Association between myelin basic protein expression and left entorhinal cortex pre-alpha cell layer disorganization in schizophrenia"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.journal","Schizophrenia Bulletin"],["dc.bibliographiccitation.volume","35"],["dc.contributor.author","Parlapani, Eleni"],["dc.contributor.author","Schmitt, A."],["dc.contributor.author","Schulenberg, Wiebke"],["dc.contributor.author","Bernstein, Hans-Gert"],["dc.contributor.author","Bogerts, Bernhard"],["dc.contributor.author","Falkai, Peter Gaston"],["dc.date.accessioned","2018-11-07T08:32:32Z"],["dc.date.available","2018-11-07T08:32:32Z"],["dc.date.issued","2009"],["dc.format.extent","229"],["dc.identifier.isi","000263964700659"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/17359"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Oxford Univ Press"],["dc.publisher.place","Oxford"],["dc.relation.conference","12th International Congress on Schizophrenia Research"],["dc.relation.eventlocation","San Diego, CA"],["dc.relation.issn","0586-7614"],["dc.title","DECREASED GYRIFICATION-INDEX (GI) IN THE CEREBELLAR VERMIS OF SCHIZOPHRENIA PATIENTS AND AN ANIMAL MODEL OF SCHIZOPHRENIA"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","89"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","Revista de psiquiatria clínica"],["dc.bibliographiccitation.lastpage","96"],["dc.bibliographiccitation.volume","36"],["dc.contributor.author","Schmitt, Andrea"],["dc.contributor.author","Otto, Sylvia"],["dc.contributor.author","Jatzko, Alexander"],["dc.contributor.author","Ruf, Matthias"],["dc.contributor.author","Demirakca, Traute"],["dc.contributor.author","Tost, Heike"],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Parlapani, Eleni"],["dc.contributor.author","Falkai, Peter"],["dc.contributor.author","Braus, Dieter F."],["dc.date.accessioned","2018-11-07T08:35:29Z"],["dc.date.available","2018-11-07T08:35:29Z"],["dc.date.issued","2009"],["dc.description.abstract","Objectives: In first-episode schizophrenia patients, functional magnetic resonance imaging (fMRI) has shown prefronto-parietal dysfunction during acoustic and visual stimulation. The aim of this study was to investigate the prefronto-parietal network in elderly schizophrenia patients using the same paradigm. Additionally, we hypothesized favourable effects on brain activation by the atypical antipsychotic clozapine compared to typical neuroleptics. Methods: We investigated 18 elderly, chronic schizophrenia patients and 21 elderly healthy controls. Nine schizophrenia patients had been medicated with clozapine and 9 had been receiving typical neuroleptics over decades. In addition to assessments with psychopathological and neuropsychological rating scales we used an acoustic and visual stimulation paradigm in a 1.5 Tesla MRI scanner to investigate BOLD-response in different brain areas. Results: Compared to healthy controls schizophrenia patients showed decreased brain activation in the prefrontal and parietal cortex as well as medial anterior cingulate gyrus compared to healthy controls. In these regions, patients medicated with clozapine showed increased BOLD-response compared to patients treated with typical neuroleptics. Discussion: Our study confirmed prefronto-parietal network disturbances in elderly schizophrenia patients thus pointing to the preservation of brain activation deficits and the influence of neurodevelopmental disturbances in chronic schizophrenia until old-age. Conclusion: The atypical antipsychotic clozapine seems to facilitate brain activation even in elderly, chronic schizophrenia patients."],["dc.identifier.doi","10.1590/S0101-60832009000300002"],["dc.identifier.fs","548703"],["dc.identifier.isi","000269276800002"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/5793"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/18078"],["dc.language.iso","es"],["dc.notes.intern","Migrated from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Univ Sao Paulo, Inst Psiquiatria"],["dc.relation.issn","0101-6083"],["dc.relation.orgunit","Universitätsmedizin Göttingen"],["dc.rights","Goescholar"],["dc.rights.access","openAccess"],["dc.rights.uri","https://goescholar.uni-goettingen.de/licenses"],["dc.subject.ddc","610"],["dc.title","Parieto-prefrontal dysfunction during visuo-auditory information processing in elderly, chronic schizophrenic patients and medication effects"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","201"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","The World Journal of Biological Psychiatry"],["dc.bibliographiccitation.lastpage","215"],["dc.bibliographiccitation.volume","12"],["dc.contributor.author","Schmitt, Andrea"],["dc.contributor.author","Leonardi-Essmann, Fernando"],["dc.contributor.author","Durrenberger, Pascal F."],["dc.contributor.author","Parlapani, Eleni"],["dc.contributor.author","Schneider-Axmann, Thomas"],["dc.contributor.author","Spanagel, Rainer"],["dc.contributor.author","Arzberger, Thomas"],["dc.contributor.author","Kretzschmar, Hans A."],["dc.contributor.author","Herrera-Marschitz, Mario"],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Reynolds, Richard"],["dc.contributor.author","Falkai, Peter"],["dc.contributor.author","Gebicke-Haerter, Peter J."],["dc.date.accessioned","2018-11-07T08:57:28Z"],["dc.date.available","2018-11-07T08:57:28Z"],["dc.date.issued","2011"],["dc.description.abstract","Objectives. The role of neuroinflammation in schizophrenia has been an issue for long time. There are reports supporting the hypothesis of ongoing inflammation and others denying it. This may be partly ascribed to the origin of the materials (CSF, blood, brain tissue) or to the genes selected for the respective studies. Moreover, in some locations, inflammatory genes may be up-regulated, others may be down-regulated. Methods. Genome-wide microarrays have been used for expression profiling in post-mortem brains of schizophrenia patients. Array data have been analyzed by gene set enrichment analysis (GSEA) and further confirmed with selected genes by real-time PCR. Results. In Brodman Area 22 of left superior temporal cortex, at least 70 genes (19%) out of 369 down-regulated genes (P < 0.05) belonged to the immune system. 23 from those 70 genes were randomly selected for real-time PCR. Six reached significance level at P < 0.05. Conclusions. The present data support a brain-specific view of the role immune-modulatory genes may play in the left superior temporal cortex in schizophrenia, because immune functions in the patients are not disturbed. In keeping with comparable, previous studies supporting the notion that schizophrenia is a disease of the synapse, we hypothesize that dysregulation of immune-related genes modifies synaptic functions and stability in this region."],["dc.identifier.doi","10.3109/15622975.2010.530690"],["dc.identifier.isi","000289439200004"],["dc.identifier.pmid","21091092"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/23409"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Informa Healthcare"],["dc.relation.issn","1562-2975"],["dc.title","Regulation of immune-modulatory genes in left superior temporal cortex of schizophrenia patients: a genome-wide microarray study"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","35"],["dc.bibliographiccitation.journal","European Archives of Psychiatry and Clinical Neuroscience"],["dc.bibliographiccitation.lastpage","39"],["dc.bibliographiccitation.volume","258"],["dc.contributor.author","Schmitt, Andrea"],["dc.contributor.author","Parlapani, Eleni"],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Wobrock, Thomas"],["dc.contributor.author","Falkai, Peter"],["dc.date.accessioned","2018-11-07T11:09:24Z"],["dc.date.available","2018-11-07T11:09:24Z"],["dc.date.issued","2008"],["dc.description.abstract","To a large extend schizophrenia has been shown to be heritable, with neuregulin-1 (NRG1) one of the candidate genes considered to play a role in the pathophysiology of the disorder. While several polymorphisms within this gene have been reported to be associated with schizophrenia, the impact of NRG1 risk genotypes on disturbed brain function and symptoms of the disease is unknown and might be elucidated using post-mortem studies. Neuregulins are signalling proteins and the NRG1 family encodes at least 15 different splice variants, classified into four isoforms. They play an important role in cell differentiation, migration, myelination and proliferation of oligodendrocytes and neurons. Dysfunction in these processes may be related to neurodevelopmental disturbances in schizophrenia. NRG1 isoforms are differentially expressed in relevant brain regions of schizophrenia patients such as the prefrontal cortex and hippocampus and may contribute to pathophysiological processes. Different NRG1 genotypes have been shown to influence gene expression of isoforms and the risk-associated variants are in primarily noncoding and promoter regions, probably operating by altering gene expression or splicing. In addition, NRG1 regulates the expression of the nicotinic acetylcholine receptor, and expression of the 7-aminobutyric acid (GABA(A)) and N-methyl-D-aspartate receptor in the brain. However, the contribution of NRG1 risk genotypes to expression of isoforms and cognitive or psychotic symptoms in patients remain to be investigated in prospective post-mortem studies."],["dc.identifier.doi","10.1007/s00406-008-5019-x"],["dc.identifier.isi","000262752900007"],["dc.identifier.pmid","18985292"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/53001"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Dr Dietrich Steinkopff Verlag"],["dc.relation.issn","0940-1334"],["dc.title","Impact of neuregulin-1 on the pathophysiology of schizophrenia in human post-mortem studies"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","101"],["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","European Archives of Psychiatry and Clinical Neuroscience"],["dc.bibliographiccitation.lastpage","111"],["dc.bibliographiccitation.volume","260"],["dc.contributor.author","Schmitt, Andrea"],["dc.contributor.author","Koschel, Jiri"],["dc.contributor.author","Zink, Mathias"],["dc.contributor.author","Bauer, Manfred"],["dc.contributor.author","Sommer, Clemens"],["dc.contributor.author","Frank, Josef"],["dc.contributor.author","Treutlein, Jens"],["dc.contributor.author","Schulze, Thomas"],["dc.contributor.author","Schneider-Axmann, Thomas"],["dc.contributor.author","Parlapani, Eleni"],["dc.contributor.author","Rietschel, Marcella"],["dc.contributor.author","Falkai, Peter"],["dc.contributor.author","Henn, Fritz A."],["dc.date.accessioned","2018-11-07T08:45:24Z"],["dc.date.available","2018-11-07T08:45:24Z"],["dc.date.issued","2010"],["dc.description.abstract","To determine if NMDA receptor alterations are present in the cerebellum in schizophrenia, we measured NMDA receptor binding and gene expression of the NMDA receptor subunits in a post-mortem study of elderly patients with schizophrenia and non-affected subjects. Furthermore, we assessed influence of genetic variation in the candidate gene neuregulin-1 (NRG1) on the expression of the NMDA receptor in an exploratory study. Post-mortem samples from the cerebellar cortex of ten schizophrenic patients were compared with nine normal subjects. We investigated NMDA receptor binding by receptor autoradiography and gene expression of the NMDA receptor subunits NR1, NR2A, NR2B, NR2C and NR2D by in situ hybridization. For the genetic study, we genotyped the NRG1 polymorphism rs35753505 (SNP8NRG221533). Additionally, we treated rats with the antipsychotics haloperidol or clozapine and assessed cerebellar NMDA receptor binding and gene expression of subunits to examine the effects of antipsychotic treatment. Gene expression of the NR2D subunit was increased in the right cerebellum of schizophrenic patients compared to controls. Individuals carrying at least one C allele of rs35753505 (SNP8NRG221533) showed decreased expression of the NR2C subunit in the right cerebellum, compared to individuals homozygous for the T allele. Correlation with medication parameters and the animal model revealed no treatment effects. In conclusion, increased NR2D expression results in a hyperexcitable NMDA receptor suggesting an adaptive effect due to receptor hypofunction. The decreased NR2C expression in NRG1 risk variant may cause a deficit in NMDA receptor function. This supports the hypothesis of an abnormal glutamatergic neurotransmission in the right cerebellum in the pathophysiology of schizophrenia."],["dc.description.sponsorship","European Commission [LSHM-CT-2004-503039]"],["dc.identifier.doi","10.1007/s00406-009-0017-1"],["dc.identifier.isi","000275085300003"],["dc.identifier.pmid","19856012"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?goescholar/4148"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/20429"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Dr Dietrich Steinkopff Verlag"],["dc.relation.issn","0940-1334"],["dc.rights","Goescholar"],["dc.rights.uri","https://goescholar.uni-goettingen.de/licenses"],["dc.title","Gene expression of NMDA receptor subunits in the cerebellum of elderly patients with schizophrenia"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1726"],["dc.bibliographiccitation.issue","12"],["dc.bibliographiccitation.journal","Journal of Neural Transmission"],["dc.bibliographiccitation.lastpage","1727"],["dc.bibliographiccitation.volume","115"],["dc.contributor.author","Schmitt, A."],["dc.contributor.author","Schulenberg, Wiebke"],["dc.contributor.author","Parlapani, Eleni"],["dc.contributor.author","Bernstein, Hans-Gert"],["dc.contributor.author","Bogerts, Bernhard"],["dc.contributor.author","Falkai, Peter Gaston"],["dc.date.accessioned","2018-11-07T11:08:39Z"],["dc.date.available","2018-11-07T11:08:39Z"],["dc.date.issued","2008"],["dc.identifier.isi","000261180300056"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/52834"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.publisher.place","Wien"],["dc.relation.conference","BrainNet Europe 2nd International Conference on Human Brain Tissue Research"],["dc.relation.eventlocation","Munich, GERMANY"],["dc.relation.issn","0300-9564"],["dc.title","Decreased gyrification-index (GI) in the cerebellar vermis of schizophrenia patients and an animal model of schizophrenia"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.journal","Der Nervenarzt"],["dc.bibliographiccitation.volume","78"],["dc.contributor.author","Falkai, Peter"],["dc.contributor.author","Schmitt, A."],["dc.contributor.author","Parlapani, Eleni"],["dc.contributor.author","Gruber, Oliver"],["dc.date.accessioned","2018-11-07T10:57:24Z"],["dc.date.available","2018-11-07T10:57:24Z"],["dc.date.issued","2007"],["dc.format.extent","314"],["dc.identifier.isi","000253318800829"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/50237"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.publisher.place","New york"],["dc.relation.issn","0028-2804"],["dc.title","Functional significance of NRG-1 for the pathophysiology of the schizophrenia in human post-mortem trials"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","243"],["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","The World Journal of Biological Psychiatry"],["dc.bibliographiccitation.lastpage","250"],["dc.bibliographiccitation.volume","11"],["dc.contributor.author","Parlapani, Eleni"],["dc.contributor.author","Schmitt, Andrea"],["dc.contributor.author","Wirths, Oliver"],["dc.contributor.author","Bauer, Manfred"],["dc.contributor.author","Sommer, Clemens"],["dc.contributor.author","Rueb, Udo"],["dc.contributor.author","Skowronek, Markus H."],["dc.contributor.author","Treutlein, Jens"],["dc.contributor.author","Petroianu, Georg A."],["dc.contributor.author","Rietschel, Marcella"],["dc.contributor.author","Falkai, Peter"],["dc.date.accessioned","2018-11-07T08:45:49Z"],["dc.date.available","2018-11-07T08:45:49Z"],["dc.date.issued","2010"],["dc.description.abstract","One important risk gene in schizophrenia is neuregulin-1 (NRG1), which is expressed in different isoforms in the brain. To determine if alterations of NRG1 are present in schizophrenia, we measured gene expression of NRG1 and its main isoforms as well as the impact of genetic variation of NRG1 in an exploratory study examining three brain regions instead of only one as published so far. In all, we examined post-mortem samples from 11 schizophrenia patients and eight normal subjects. We investigated gene expression of total NRG1 and isoforms I, II and III by real-time PCR in the prefrontal cortex (Brodmann areas 9 and 10) and right hippocampal tissue. For the genetic study, we genotyped the NRG1 polymorphism SNP8NRG221533, which is within the core haplotype of the original publication. Compared to controls, gene expression of the NRG1 isoform I was decreased and isoform II increased in the prefrontal cortex (BA10) of schizophrenia patients. There were no statistically significant differences between individuals carrying at least one C allele of SNP8NRG221533 compared to individuals homozygous for the Tallele. The decreased expression of NRG1 isoform I and overexpression of isoform II may be related to deficits in receptor function as well as abnormal migration and myelination. However, our study sample was small and results of this exploratory study should be verified in a larger sample."],["dc.description.sponsorship","European Commission [LSHM-CT-2004-503039]"],["dc.identifier.doi","10.3109/15622970802022376"],["dc.identifier.isi","000284143000009"],["dc.identifier.pmid","20218788"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/20539"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Taylor & Francis Ltd"],["dc.relation.issn","1562-2975"],["dc.title","Gene expression of neuregulin-1 isoforms in different brain regions of elderly schizophrenia patients"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","255"],["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","Clinics (São Paulo)"],["dc.bibliographiccitation.lastpage","266"],["dc.bibliographiccitation.volume","63"],["dc.contributor.author","Schmitt, Andrea"],["dc.contributor.author","Parlapani, Eleni"],["dc.contributor.author","Bauer, Manfred"],["dc.contributor.author","Heinsen, Helmut"],["dc.contributor.author","Falkai, Peter"],["dc.date.accessioned","2021-06-01T10:48:28Z"],["dc.date.available","2021-06-01T10:48:28Z"],["dc.date.issued","2008"],["dc.description.abstract","It is widely accepted that neurobiological abnon-nalities underlie the symptoms of psychiatric disorders such as schizophrenia and unipolar or bipolar affective disorders. New molecular methods, computer-assisted quantification techniques and neurobiological investigation methods that can be applied to the human brain are all used inpost-mortem investigations of psychiatric disorders. The following article describes modem quantitative methods and recent post-mortem findings in schizophrenia and affective disorders. Using our brain bank as an example, necessary considerations of modem brain banking are addressed such as ethical considerations, clinical work-up, preparation techniques and the organization of a brain bank, the value of modem brain banking for investigations of psychiatric disorders is summarized."],["dc.identifier.doi","10.1590/S1807-59322008000200015"],["dc.identifier.isi","000255101600015"],["dc.identifier.pmid","18438581"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/85950"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-425"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Hospital Clinicas, Univ Sao Paulo"],["dc.relation.eissn","1807-5932"],["dc.relation.issn","1807-5932"],["dc.title","Is brain banking of psychiatric cases valuable for neurobiological research?"],["dc.type","review"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]