Mollenhauer, Brit

[["dc.bibliographiccitation.artnumber","121"],["dc.bibliographiccitation.journal","Frontiers in Aging Neuroscience"],["dc.bibliographiccitation.volume","8"],["dc.contributor.affiliation","Lerche, Stefanie;"],["dc.contributor.affiliation","Heinzel, Sebastian;"],["dc.contributor.affiliation","Alves, Guido W.;"],["dc.contributor.affiliation","Barone, Paolo;"],["dc.contributor.affiliation","Behnke, Stefanie;"],["dc.contributor.affiliation","Ben-Shlomo, Yoav;"],["dc.contributor.affiliation","Berendse, Henk;"],["dc.contributor.affiliation","Bloem, Bastiaan R.;"],["dc.contributor.affiliation","Burn, David;"],["dc.contributor.affiliation","Dodel, Richard;"],["dc.contributor.affiliation","Grosset, Donald G.;"],["dc.contributor.affiliation","Hipp, Geraldine;"],["dc.contributor.affiliation","Hu, Michele T.;"],["dc.contributor.affiliation","Kasten, Meike;"],["dc.contributor.affiliation","Krüger, Rejko;"],["dc.contributor.affiliation","Liepelt-Scarfone, Inga;"],["dc.contributor.affiliation","Maetzler, Walter;"],["dc.contributor.affiliation","Moccia, Marcello;"],["dc.contributor.affiliation","Mollenhauer, Brit;"],["dc.contributor.affiliation","Oertel, Wolfgang;"],["dc.contributor.affiliation","Roeben, Benjamin;"],["dc.contributor.affiliation","Walter, Uwe;"],["dc.contributor.affiliation","Wirdefeldt, Karin;"],["dc.contributor.affiliation","Berg, Daniela;"],["dc.contributor.author","Lerche, Stefanie"],["dc.contributor.author","Heinzel, Sebastian"],["dc.contributor.author","Alves, Guido W."],["dc.contributor.author","Barone, Paolo"],["dc.contributor.author","Behnke, Stefanie"],["dc.contributor.author","Ben-Shlomo, Yoav"],["dc.contributor.author","Berendse, Henk W."],["dc.contributor.author","Bloem, Bastiaan R."],["dc.contributor.author","Burns, David J."],["dc.contributor.author","Dodel, Richard"],["dc.contributor.author","Grosset, Donald G."],["dc.contributor.author","Hipp, Geraldine"],["dc.contributor.author","Hu, Michele T. M."],["dc.contributor.author","Kasten, Meike"],["dc.contributor.author","Krüger, Rejko"],["dc.contributor.author","Liepelt-Scarfone, Inga"],["dc.contributor.author","Moccia, Marcello"],["dc.contributor.author","Mollenhauer, Brit"],["dc.contributor.author","Oertel, Wolfgang"],["dc.contributor.author","Roeben, Benjamin"],["dc.contributor.author","Walter, Uwe"],["dc.contributor.author","Wirdefeldt, Karin"],["dc.contributor.author","Maetzler, Walter"],["dc.contributor.author","Berg, Daniela"],["dc.date.accessioned","2018-11-07T10:14:05Z"],["dc.date.available","2018-11-07T10:14:05Z"],["dc.date.issued","2016"],["dc.date.updated","2022-02-09T13:23:10Z"],["dc.identifier.doi","10.3389/fnagi.2016.00121"],["dc.identifier.eissn","1663-4365"],["dc.identifier.isi","000376574700001"],["dc.identifier.pmid","27303289"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/13374"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/40560"],["dc.language.iso","en"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Frontiers Media S.A."],["dc.relation.eissn","1663-4365"],["dc.relation.issn","1663-4365"],["dc.rights","Goescholar"],["dc.rights.uri","https://goescholar.uni-goettingen.de/licenses"],["dc.title","Aiming for Study Comparability in Parkinson's Disease: Proposal for a Modular Set of Biomarker Assessments to be Used in Longitudinal Studies"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.subtype","letter_note"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","24"],["dc.bibliographiccitation.journal","Parkinsonism & Related Disorders"],["dc.bibliographiccitation.lastpage","26"],["dc.bibliographiccitation.volume","75"],["dc.contributor.author","Hopfner, Franziska"],["dc.contributor.author","Mueller, Stefanie H."],["dc.contributor.author","Szymczak, Silke"],["dc.contributor.author","Junge, Olaf"],["dc.contributor.author","Tittmann, Lukas"],["dc.contributor.author","May, Sandra"],["dc.contributor.author","Lohmann, Katja"],["dc.contributor.author","Grallert, Harald"],["dc.contributor.author","Lieb, Wolfgang"],["dc.contributor.author","Strauch, Konstantin"],["dc.contributor.author","Müller-Nurasyid, Martina"],["dc.contributor.author","Berger, Klaus"],["dc.contributor.author","Schormair, Barbara"],["dc.contributor.author","Winkelmann, Juliane"],["dc.contributor.author","Mollenhauer, Brit"],["dc.contributor.author","Trenkwalder, Claudia"],["dc.contributor.author","Maetzler, Walter"],["dc.contributor.author","Berg, Daniela"],["dc.contributor.author","Kasten, Meike"],["dc.contributor.author","Klein, Christine"],["dc.contributor.author","Höglinger, Günter U."],["dc.contributor.author","Gasser, Thomas"],["dc.contributor.author","Deuschl, Günther"],["dc.contributor.author","Franke, André"],["dc.contributor.author","Krawczak, Michael"],["dc.contributor.author","Dempfle, Astrid"],["dc.contributor.author","Kuhlenbäumer, Gregor"],["dc.date.accessioned","2021-04-14T08:25:55Z"],["dc.date.available","2021-04-14T08:25:55Z"],["dc.date.issued","2020"],["dc.identifier.doi","10.1016/j.parkreldis.2020.05.003"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/81769"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-399"],["dc.relation.issn","1353-8020"],["dc.title","Private variants in PRKN are associated with late-onset Parkinson's disease"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","593"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","Movement Disorders"],["dc.bibliographiccitation.lastpage","596"],["dc.bibliographiccitation.volume","31"],["dc.contributor.author","Lawton, Michael"],["dc.contributor.author","Kasten, Meike"],["dc.contributor.author","May, Margaret T."],["dc.contributor.author","Mollenhauer, Brit"],["dc.contributor.author","Schaumburg, Martina"],["dc.contributor.author","Liepelt-Scarfone, Inga"],["dc.contributor.author","Maetzler, Walter"],["dc.contributor.author","Vollstedt, Eva-Juliane"],["dc.contributor.author","Hu, Michele T. M."],["dc.contributor.author","Berg, Daniela"],["dc.contributor.author","Ben-Shlomo, Yoav"],["dc.date.accessioned","2018-11-07T10:16:04Z"],["dc.date.available","2018-11-07T10:16:04Z"],["dc.date.issued","2016"],["dc.description.abstract","IntroductionHarmonizing data across cohorts is important for validating findings or combining data in meta-analyses. We replicate and validate a previous conversion of MoCA to MMSE in PD. MethodsWe used five studies with 1,161 PD individuals and 2,091 observations measured with both the MoCA and MMSE. We compared a previously published conversion table using equipercentile equating with log-linear smoothing to our internally derived scores. ResultsBoth conversions found good agreement within and across the studies when comparing true and converted MMSE (mean difference: 0.05; standard deviation: 1.84; median difference: 0; interquartile range: -1 to 1, using internal conversion). ConclusionsThese results show that one can get a reliable and valid conversion between two commonly used measures of cognition in PD studies. These approaches need to be applied to other scales and domains to enable large-scale collaborative analyses across multiple PD cohorts. (c) 2016 International Parkinson and Movement Disorder Society"],["dc.identifier.doi","10.1002/mds.26498"],["dc.identifier.isi","000374546300026"],["dc.identifier.pmid","26861697"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/14046"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/40959"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Wiley-blackwell"],["dc.relation.issn","1531-8257"],["dc.relation.issn","0885-3185"],["dc.rights","Goescholar"],["dc.rights.uri","https://goescholar.uni-goettingen.de/licenses"],["dc.title","Validation of conversion between mini-mental state examination and montreal cognitive assessment"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.artnumber","1512.e1"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","Neurobiology of Aging"],["dc.bibliographiccitation.volume","35"],["dc.contributor.author","Koschmidder, Eva"],["dc.contributor.author","Mollenhauer, Brit"],["dc.contributor.author","Kasten, Meike"],["dc.contributor.author","Klein, Christine"],["dc.contributor.author","Lohmann, Katja"],["dc.date.accessioned","2018-11-07T09:39:54Z"],["dc.date.available","2018-11-07T09:39:54Z"],["dc.date.issued","2014"],["dc.description.abstract","VPS35 mutations have been identified as a cause of autosomal dominantly inherited Parkinson's disease (PD). VPS35 interacts with VPS26A in the retromer complex that links mitochondrial and lysosomal pathways, which have both been shown to be dysfunctional in PD. Thus, mutations in VPS26A may be associated with PD. To test this hypothesis, we screened 245 idiopathic PD patients and 185 control subjects for mutations in the retromer subunit VPS26A. We found 2 novel missense variants in patients and 2 known missense variants in control subjects. The missense variants were unlikely to be disease causing, suggesting that VPS26A mutations are not a frequent cause of PD. (C) 2014 Elsevier Inc. All rights reserved."],["dc.identifier.doi","10.1016/j.neurobiolaging.2013.12.016"],["dc.identifier.isi","000333970800037"],["dc.identifier.pmid","24417787"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/33396"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Elsevier Science Inc"],["dc.relation.issn","1558-1497"],["dc.relation.issn","0197-4580"],["dc.title","Mutations in VPS26A are not a frequent cause of Parkinson's disease"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.artnumber","147"],["dc.bibliographiccitation.journal","Frontiers in Aging Neuroscience"],["dc.bibliographiccitation.volume","8"],["dc.contributor.author","Heinzel, Sebastian"],["dc.contributor.author","Roeben, Benjamin"],["dc.contributor.author","Ben-Shlomo, Yoav"],["dc.contributor.author","Lerche, Stefanie"],["dc.contributor.author","Alves, Guido"],["dc.contributor.author","Barone, Paolo"],["dc.contributor.author","Behnke, Stefanie"],["dc.contributor.author","Berendse, Henk W."],["dc.contributor.author","Bloem, Bastiaan R."],["dc.contributor.author","Burns, David J."],["dc.contributor.author","Dodel, Richard"],["dc.contributor.author","Grosset, Donald G."],["dc.contributor.author","Hu, Michele T. M."],["dc.contributor.author","Kasten, Meike"],["dc.contributor.author","Krueger, Rejko"],["dc.contributor.author","Moccia, Marcello"],["dc.contributor.author","Mollenhauer, Brit"],["dc.contributor.author","Oertel, Wolfgang"],["dc.contributor.author","Suenkel, Ulrike"],["dc.contributor.author","Walter, Uwe"],["dc.contributor.author","Wirdefeldt, Karin"],["dc.contributor.author","Liepelt-Scarfone, Inga"],["dc.contributor.author","Maetzler, Walter"],["dc.contributor.author","Berg, Daniela"],["dc.date.accessioned","2018-11-07T10:12:40Z"],["dc.date.available","2018-11-07T10:12:40Z"],["dc.date.issued","2016"],["dc.description.abstract","A growing body of evidence supports a prodromal neurodegenerative process preceding the clinical onset of Parkinson's disease (PD). Studies have identified several different prodromal markers that may have the potential to predict the conversion from healthy to clinical PD but use considerably different approaches. We systematically reviewed 35 longitudinal studies reporting prodromal PD features and evaluated the methodological quality across 10 different predefined domains. We found limitations in the following domains: PD diagnosis (57% of studies), prodromal marker assessments (51%), temporal information on prodromal markers or PD diagnosis (34%), generalizability of results (17%), statistical methods (accounting for at least age as confounder; 17%), study design (14%), and sample size (9%). However, no limitations regarding drop-out (or bias investigation), or report of inclusion/exclusion criteria or prodromal marker associations were revealed. Lessons learned from these limitations and additional aspects of current prodromal marker studies in PD are discussed to provide a basis for the evaluation of findings and the improvement of future research in prodromal PD. The observed heterogeneity of studies, limitations and analyses might be addressed in future longitudinal studies using a, yet to be established, modular minimal set of assessments improving comparability of findings and enabling data sharing and combined analyses across studies."],["dc.identifier.doi","10.3389/fnagi.2016.00147"],["dc.identifier.isi","000378534100002"],["dc.identifier.pmid","27445791"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/13476"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/40281"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Frontiers Media Sa"],["dc.relation.issn","1663-4365"],["dc.rights","Goescholar"],["dc.rights.uri","https://goescholar.uni-goettingen.de/licenses"],["dc.title","Prodromal Markers in Parkinson's Disease: Limitations in Longitudinal Studies and Lessons Learned"],["dc.type","review"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1245"],["dc.bibliographiccitation.issue","7"],["dc.bibliographiccitation.journal","Movement Disorders"],["dc.bibliographiccitation.lastpage","1248"],["dc.bibliographiccitation.volume","35"],["dc.contributor.author","Hopfner, Franziska"],["dc.contributor.author","Mueller, Stefanie H."],["dc.contributor.author","Szymczak, Silke"],["dc.contributor.author","Junge, Olaf"],["dc.contributor.author","Tittmann, Lukas"],["dc.contributor.author","May, Sandra"],["dc.contributor.author","Lohmann, Katja"],["dc.contributor.author","Grallert, Harald"],["dc.contributor.author","Lieb, Wolfgang"],["dc.contributor.author","Strauch, Konstantin"],["dc.contributor.author","Müller‐Nurasyid, Martina"],["dc.contributor.author","Berger, Klaus"],["dc.contributor.author","Schormair, Barbara"],["dc.contributor.author","Winkelmann, Juliane"],["dc.contributor.author","Mollenhauer, Brit"],["dc.contributor.author","Trenkwalder, Claudia"],["dc.contributor.author","Maetzler, Walter"],["dc.contributor.author","Berg, Daniela"],["dc.contributor.author","Kasten, Meike"],["dc.contributor.author","Klein, Christine"],["dc.contributor.author","Höglinger, Günter U."],["dc.contributor.author","Gasser, Thomas"],["dc.contributor.author","Deuschl, Günther"],["dc.contributor.author","Franke, André"],["dc.contributor.author","Krawczak, Michael"],["dc.contributor.author","Dempfle, Astrid"],["dc.contributor.author","Kuhlenbäumer, Gregor"],["dc.date.accessioned","2021-04-14T08:26:16Z"],["dc.date.available","2021-04-14T08:26:16Z"],["dc.date.issued","2020"],["dc.identifier.doi","10.1002/mds.28037"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/81882"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-399"],["dc.relation.eissn","1531-8257"],["dc.relation.issn","0885-3185"],["dc.title","Rare Variants in Specific Lysosomal Genes Are Associated With Parkinson's Disease"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","282"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","Neuroepidemiology"],["dc.bibliographiccitation.lastpage","297"],["dc.bibliographiccitation.volume","45"],["dc.contributor.author","Lerche, Stefanie"],["dc.contributor.author","Liepelt-Scarfone, Inga"],["dc.contributor.author","Alves, Guido"],["dc.contributor.author","Barone, Paolo"],["dc.contributor.author","Behnke, Stefanie"],["dc.contributor.author","Ben-Shlomo, Yoav"],["dc.contributor.author","Berendse, Henk W."],["dc.contributor.author","Burns, David J."],["dc.contributor.author","Dodel, Richard"],["dc.contributor.author","Grosset, Donald G."],["dc.contributor.author","Heinzel, Sebastian"],["dc.contributor.author","Hu, Michele T. M."],["dc.contributor.author","Kasten, Meike"],["dc.contributor.author","Krueger, Rejko"],["dc.contributor.author","Maetzler, Walter"],["dc.contributor.author","Moccia, Marcello"],["dc.contributor.author","Mollenhauer, Brit"],["dc.contributor.author","Oertel, Wolfgang"],["dc.contributor.author","Roeben, Benjamin"],["dc.contributor.author","Suenkel, Ulrike"],["dc.contributor.author","Walter, Uwe"],["dc.contributor.author","Wirdefeldt, Karin"],["dc.contributor.author","Berg, Daniela"],["dc.date.accessioned","2018-11-07T10:02:39Z"],["dc.date.available","2018-11-07T10:02:39Z"],["dc.date.issued","2015"],["dc.description.abstract","Background: Enormous effort is being put into the identification and characterization of symptoms that may be used as predictive and progression markers in Parkinson's disease (PD). An impressive number of PD patients and individuals at risk for or in the prodromal stage of PD are currently followed in longitudinal studies; however, there does not exist an overview on the kind of markers evaluated and the assessments used. Methods: Information on the design, sample size, evaluated markers and assessments of 21 studies of the Joint Programme - Neurodegenerative Disease Research BioLoC-PD working group were collected by questionnaire. The studies were classified into at risk/prodromal or clinical PD cohorts. The assessments were grouped into quantitative assessments, investigator-rated assessments, investigator inter-views, patient-rated questionnaires and caregiver-rated questionnaires. Results: Compilation of these data revealed an interesting consensus on evaluated markers, but there was an enormous variability of assessments. Furthermore, there is a remarkable similarity in the markers assessed and evaluation methods applied in the risk/prodromal and clinical PD cohorts. Conclusions: The inventory of the longitudinal cohorts that are part of the BioLoC-PD consortium reveals that there is a growing consensus on the markers that should be assessed in longitudinal cohort studies in PD. However, controversy still exists on the specific type of assessment. To allow comparison of data and common analyses it will be essential to harmonize scales and assessment outcomes. (C) 2015 S. Karger AG, Basel"],["dc.identifier.doi","10.1159/000439221"],["dc.identifier.isi","000366276700006"],["dc.identifier.pmid","26523894"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/38272"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Karger"],["dc.relation.issn","1423-0208"],["dc.relation.issn","0251-5350"],["dc.title","Methods in Neuroepidemiology Characterization of European Longitudinal Cohort Studies in Parkinson's Disease - Report of the JPND Working Group BioLoC-PD"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1859"],["dc.bibliographiccitation.issue","12"],["dc.bibliographiccitation.journal","Genes"],["dc.bibliographiccitation.volume","12"],["dc.contributor.author","Koch, Sebastian"],["dc.contributor.author","Laabs, Björn-Hergen"],["dc.contributor.author","Kasten, Meike"],["dc.contributor.author","Vollstedt, Eva-Juliane"],["dc.contributor.author","Becktepe, Jos"],["dc.contributor.author","Brüggemann, Norbert"],["dc.contributor.author","Franke, Andre"],["dc.contributor.author","Krämer, Ulrike M."],["dc.contributor.author","Kuhlenbäumer, Gregor"],["dc.contributor.author","Lieb, Wolfgang"],["dc.contributor.author","Caliebe, Amke"],["dc.contributor.author","Mollenhauer, Brit"],["dc.contributor.author","Neis, Miriam"],["dc.contributor.author","Trenkwalder, Claudia"],["dc.contributor.author","Schäffer, Eva"],["dc.contributor.author","Usnich, Tatiana"],["dc.contributor.author","Wittig, Michael"],["dc.contributor.author","Klein, Christine"],["dc.contributor.author","König, Inke R."],["dc.contributor.author","Lohmann, Katja"],["dc.contributor.author","Krawczak, Michael"],["dc.contributor.editor","Ebert, Allison D."],["dc.date.accessioned","2022-01-11T14:08:06Z"],["dc.date.available","2022-01-11T14:08:06Z"],["dc.date.issued","2021"],["dc.description.abstract","Idiopathic Parkinson’s disease (PD) is a complex multifactorial disorder caused by the interplay of both genetic and non-genetic risk factors. Polygenic risk scores (PRSs) are one way to aggregate the effects of a large number of genetic variants upon the risk for a disease like PD in a single quantity. However, reassessment of the performance of a given PRS in independent data sets is a precondition for establishing the PRS as a valid tool to this end. We studied a previously proposed PRS for PD in a separate genetic data set, comprising 1914 PD cases and 4464 controls, and were able to replicate its ability to differentiate between cases and controls. We also assessed theoretically the prognostic value of the PD-PRS, i.e., its ability to predict the development of PD in later life for healthy individuals. As it turned out, the PD-PRS alone can be expected to perform poorly in this regard. Therefore, we conclude that the PD-PRS could serve as an important research tool, but that meaningful PRS-based prognosis of PD at an individual level is not feasible."],["dc.description.abstract","Idiopathic Parkinson’s disease (PD) is a complex multifactorial disorder caused by the interplay of both genetic and non-genetic risk factors. Polygenic risk scores (PRSs) are one way to aggregate the effects of a large number of genetic variants upon the risk for a disease like PD in a single quantity. However, reassessment of the performance of a given PRS in independent data sets is a precondition for establishing the PRS as a valid tool to this end. We studied a previously proposed PRS for PD in a separate genetic data set, comprising 1914 PD cases and 4464 controls, and were able to replicate its ability to differentiate between cases and controls. We also assessed theoretically the prognostic value of the PD-PRS, i.e., its ability to predict the development of PD in later life for healthy individuals. As it turned out, the PD-PRS alone can be expected to perform poorly in this regard. Therefore, we conclude that the PD-PRS could serve as an important research tool, but that meaningful PRS-based prognosis of PD at an individual level is not feasible."],["dc.identifier.doi","10.3390/genes12121859"],["dc.identifier.pii","genes12121859"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/97934"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-507"],["dc.publisher","MDPI"],["dc.relation.eissn","2073-4425"],["dc.rights","Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/)."],["dc.rights.uri","https://creativecommons.org/licenses/by/4.0/"],["dc.title","Validity and Prognostic Value of a Polygenic Risk Score for Parkinson’s Disease"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]