Options
Göpfert, Martin C.
Loading...
Preferred name
Göpfert, Martin C.
Official Name
Göpfert, Martin C.
Alternative Name
Goepfert, Martin C.
Göpfert, Martin
Goepfert, Martin
Gopfert, Martin C.
Gopfert, Martin
Göpfert, M. C.
Goepfert, M. C.
Gopfert, M. C.
Main Affiliation
Now showing 1 - 10 of 69
2013Conference Abstract [["dc.bibliographiccitation.issue","8"],["dc.bibliographiccitation.journal","Pathogens and Global Health"],["dc.bibliographiccitation.volume","107"],["dc.contributor.author","Andres, Marta"],["dc.contributor.author","Karak, Somdatta"],["dc.contributor.author","Joo, Seol-hee"],["dc.contributor.author","Piepenbrock, David"],["dc.contributor.author","Göpfert, Martin C."],["dc.date.accessioned","2018-11-07T09:16:41Z"],["dc.date.available","2018-11-07T09:16:41Z"],["dc.date.issued","2013"],["dc.format.extent","406"],["dc.identifier.isi","000335056200021"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/27988"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Maney Publishing"],["dc.publisher.place","Leeds"],["dc.relation.issn","2047-7732"],["dc.relation.issn","2047-7724"],["dc.title","DROSOPHILA MELANOGASTER AS A MODEL TO UNDERSTAND HEARING IN DISEASE VECTORS"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]Details WOS2013Journal Article [["dc.bibliographiccitation.firstpage","20130528"],["dc.bibliographiccitation.issue","1759"],["dc.bibliographiccitation.journal","Proceedings of the Royal Society B: Biological Sciences"],["dc.bibliographiccitation.volume","280"],["dc.contributor.author","Greggers, Uwe"],["dc.contributor.author","Koch, Gesche"],["dc.contributor.author","Schmidt, Viola"],["dc.contributor.author","Dürr, Aron"],["dc.contributor.author","Floriou-Servou, Amalia"],["dc.contributor.author","Piepenbrock, David"],["dc.contributor.author","Göpfert, Martin C."],["dc.contributor.author","Menzel, Randolf"],["dc.date.accessioned","2022-03-01T11:46:53Z"],["dc.date.available","2022-03-01T11:46:53Z"],["dc.date.issued","2013"],["dc.description.abstract","Honeybees, like other insects, accumulate electric charge in flight, and when their body parts are moved or rubbed together. We report that bees emit constant and modulated electric fields when flying, landing, walking and during the waggle dance. The electric fields emitted by dancing bees consist of low- and high-frequency components. Both components induce passive antennal movements in stationary bees according to Coulomb's law. Bees learn both the constant and the modulated electric field components in the context of appetitive proboscis extension response conditioning. Using this paradigm, we identify mechanoreceptors in both joints of the antennae as sensors. Other mechanoreceptors on the bee body are potentially involved but are less sensitive. Using laser vibrometry, we show that the electrically charged flagellum is moved by constant and modulated electric fields and more strongly so if sound and electric fields interact. Recordings from axons of the Johnston organ document its sensitivity to electric field stimuli. Our analyses identify electric fields emanating from the surface charge of bees as stimuli for mechanoreceptors, and as biologically relevant stimuli, which may play a role in social communication."],["dc.description.abstract","Honeybees, like other insects, accumulate electric charge in flight, and when their body parts are moved or rubbed together. We report that bees emit constant and modulated electric fields when flying, landing, walking and during the waggle dance. The electric fields emitted by dancing bees consist of low- and high-frequency components. Both components induce passive antennal movements in stationary bees according to Coulomb's law. Bees learn both the constant and the modulated electric field components in the context of appetitive proboscis extension response conditioning. Using this paradigm, we identify mechanoreceptors in both joints of the antennae as sensors. Other mechanoreceptors on the bee body are potentially involved but are less sensitive. Using laser vibrometry, we show that the electrically charged flagellum is moved by constant and modulated electric fields and more strongly so if sound and electric fields interact. Recordings from axons of the Johnston organ document its sensitivity to electric field stimuli. Our analyses identify electric fields emanating from the surface charge of bees as stimuli for mechanoreceptors, and as biologically relevant stimuli, which may play a role in social communication."],["dc.identifier.doi","10.1098/rspb.2013.0528"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/103834"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-531"],["dc.relation.eissn","1471-2954"],["dc.relation.issn","0962-8452"],["dc.rights.uri","https://royalsociety.org/journals/ethics-policies/data-sharing-mining/"],["dc.title","Reception and learning of electric fields in bees"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dspace.entity.type","Publication"]]Details DOI2006Journal Article [["dc.bibliographiccitation.firstpage","511"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","European Biophysics Journal"],["dc.bibliographiccitation.lastpage","516"],["dc.bibliographiccitation.volume","35"],["dc.contributor.author","Stoop, R."],["dc.contributor.author","Kern, A."],["dc.contributor.author","Göpfert, Martin C."],["dc.contributor.author","Smirnov, D. A."],["dc.contributor.author","Dikanev, T. V."],["dc.contributor.author","Bezrucko, B. P."],["dc.date.accessioned","2022-03-01T11:44:34Z"],["dc.date.available","2022-03-01T11:44:34Z"],["dc.date.issued","2006"],["dc.identifier.doi","10.1007/s00249-006-0059-5"],["dc.identifier.pii","59"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/103052"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-531"],["dc.relation.eissn","1432-1017"],["dc.relation.issn","0175-7571"],["dc.title","A generalization of the van-der-Pol oscillator underlies active signal amplification in Drosophila hearing"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dspace.entity.type","Publication"]]Details DOI2012Journal Article [["dc.bibliographiccitation.firstpage","1042"],["dc.bibliographiccitation.issue","5"],["dc.bibliographiccitation.journal","Cell"],["dc.bibliographiccitation.lastpage","1054"],["dc.bibliographiccitation.volume","150"],["dc.contributor.author","Senthilan, Pingkalai R."],["dc.contributor.author","Piepenbrock, David"],["dc.contributor.author","Ovezmyradov, Guvanch"],["dc.contributor.author","Nadrowski, Bjoern"],["dc.contributor.author","Bechstedt, Susanne"],["dc.contributor.author","Pauls, Stephanie"],["dc.contributor.author","Winkler, Margret"],["dc.contributor.author","Möbius, Wiebke"],["dc.contributor.author","Howard, Jonathon"],["dc.contributor.author","Göpfert, Martin C."],["dc.date.accessioned","2018-11-07T09:07:03Z"],["dc.date.available","2018-11-07T09:07:03Z"],["dc.date.issued","2012"],["dc.description.abstract","The Drosophila auditory organ shares equivalent transduction mechanisms with vertebrate hair cells, and both are specified by atonal family genes. Using a whole-organ knockout strategy based on atonal, we have identified 274 Drosophila auditory organ genes. Only four of these genes had previously been associated with fly hearing, yet one in five of the genes that we identified has a human cognate that is implicated in hearing disorders. Mutant analysis of 42 genes shows that more than half of them contribute to auditory organ function, with phenotypes including hearing loss, auditory hypersusceptibility, and ringing ears. We not only discover ion channels and motors important for hearing, but also show that auditory stimulus processing involves chemoreceptor proteins as well as phototransducer components. Our findings demonstrate mechanosensory roles for ionotropic receptors and visual rhodopsins and indicate that different sensory modalities utilize common signaling cascades."],["dc.identifier.doi","10.1016/j.cell.2012.06.043"],["dc.identifier.isi","000308500200017"],["dc.identifier.pmid","22939627"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/25700"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Cell Press"],["dc.relation.issn","0092-8674"],["dc.title","Drosophila Auditory Organ Genes and Genetic Hearing Defects"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]Details DOI PMID PMC WOS2013Journal Article Discussion [["dc.bibliographiccitation.firstpage","28"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Neuroforum"],["dc.bibliographiccitation.lastpage","+"],["dc.bibliographiccitation.volume","19"],["dc.contributor.author","Moser, Tobias"],["dc.contributor.author","Löwel, Siegrid"],["dc.contributor.author","Goepfert, Martin C."],["dc.date.accessioned","2018-11-07T09:28:18Z"],["dc.date.available","2018-11-07T09:28:18Z"],["dc.date.issued","2013"],["dc.identifier.isi","000321725500008"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/30741"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Spektrum Akademischer Verlag-springer-verlag Gmbh"],["dc.relation.issn","0947-0875"],["dc.title","Cellular Mechanisms of Sensory Processing"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.subtype","letter_note"],["dspace.entity.type","Publication"]]Details WOS2009Journal Article [["dc.bibliographiccitation.firstpage","619"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","Psychological Medicine"],["dc.bibliographiccitation.lastpage","628"],["dc.bibliographiccitation.volume","21"],["dc.contributor.author","Wood, G. C."],["dc.contributor.author","Bentall, R. P."],["dc.contributor.author","Göpfert, M."],["dc.contributor.author","Edwards, R. H. T."],["dc.date.accessioned","2022-03-01T11:45:37Z"],["dc.date.available","2022-03-01T11:45:37Z"],["dc.date.issued","2009"],["dc.description.abstract","SYNOPSIS The psychiatric status of patients with chronic fatigue syndrome ( N = 34) and muscle disease ( N = 24) attending a general medical clinic was studied. Among fatigue patients 14 (41·2%) were cases and a further 9 (26·5%) were subcases of psychiatric disorder as defined by CATEGO. A variety of diagnoses was found. Significantly fewer of the muscle patients had a psychiatric disorder with 3 (12·5%) being cases and 1 (4%) a subcase. The relative risk of psychiatric disorder in patients with chronic fatigue syndrome compared to patients with muscle disease was 3·3:1."],["dc.description.abstract","SYNOPSIS The psychiatric status of patients with chronic fatigue syndrome ( N = 34) and muscle disease ( N = 24) attending a general medical clinic was studied. Among fatigue patients 14 (41·2%) were cases and a further 9 (26·5%) were subcases of psychiatric disorder as defined by CATEGO. A variety of diagnoses was found. Significantly fewer of the muscle patients had a psychiatric disorder with 3 (12·5%) being cases and 1 (4%) a subcase. The relative risk of psychiatric disorder in patients with chronic fatigue syndrome compared to patients with muscle disease was 3·3:1."],["dc.identifier.doi","10.1017/S003329170002225X"],["dc.identifier.pii","S003329170002225X"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/103396"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-531"],["dc.relation.eissn","1469-8978"],["dc.relation.issn","0033-2917"],["dc.title","A comparative psychiatric assessment of patients with chronic fatigue syndrome and muscle disease"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dspace.entity.type","Publication"]]Details DOI2014Conference Abstract [["dc.bibliographiccitation.journal","Abstracts of Papers of the American Chemical Society"],["dc.bibliographiccitation.volume","248"],["dc.contributor.author","Salgado, Vincent L."],["dc.contributor.author","Nesterov, Alexandre"],["dc.contributor.author","Kandasamy, Ramani A."],["dc.contributor.author","Spalthoff, Christian"],["dc.contributor.author","Göpfert, Martin C."],["dc.date.accessioned","2018-11-07T09:36:41Z"],["dc.date.available","2018-11-07T09:36:41Z"],["dc.date.issued","2014"],["dc.identifier.isi","000349165100596"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/32673"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Amer Chemical Soc"],["dc.publisher.place","Washington"],["dc.relation.eventlocation","San Francisco, CA"],["dc.relation.issn","0065-7727"],["dc.title","Action of insecticides on chordotonal organs"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]Details WOS2002Journal Article [["dc.bibliographiccitation.firstpage","715"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","Current Opinion in Neurobiology"],["dc.bibliographiccitation.lastpage","720"],["dc.bibliographiccitation.volume","12"],["dc.contributor.author","Robert, Daniel"],["dc.contributor.author","Göpfert, Martin C."],["dc.date.accessioned","2022-03-01T11:45:35Z"],["dc.date.available","2022-03-01T11:45:35Z"],["dc.date.issued","2002"],["dc.identifier.doi","10.1016/S0959-4388(02)00378-1"],["dc.identifier.pii","S0959438802003781"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/103382"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-531"],["dc.relation.issn","0959-4388"],["dc.title","Novel schemes for hearing and orientation in insects"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dspace.entity.type","Publication"]]Details DOI2003Journal Article [["dc.bibliographiccitation.firstpage","5514"],["dc.bibliographiccitation.issue","9"],["dc.bibliographiccitation.journal","Proceedings of the National Academy of Sciences"],["dc.bibliographiccitation.lastpage","5519"],["dc.bibliographiccitation.volume","100"],["dc.contributor.author","Göpfert, Martin C."],["dc.contributor.author","Robert, D."],["dc.date.accessioned","2022-03-01T11:46:22Z"],["dc.date.available","2022-03-01T11:46:22Z"],["dc.date.issued","2003"],["dc.identifier.doi","10.1073/pnas.0737564100"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/103643"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-531"],["dc.relation.eissn","1091-6490"],["dc.relation.issn","0027-8424"],["dc.title","Motion generation by Drosophila mechanosensory neurons"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dspace.entity.type","Publication"]]Details DOI2015Journal Article [["dc.bibliographiccitation.firstpage","626"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","Cell Death and Differentiation"],["dc.bibliographiccitation.lastpage","642"],["dc.bibliographiccitation.volume","22"],["dc.contributor.author","Matz, A."],["dc.contributor.author","Lee, S. J."],["dc.contributor.author","Schwedhelm-Domeyer, Nicola"],["dc.contributor.author","Zanini, Damiano"],["dc.contributor.author","Holubowska, Anna"],["dc.contributor.author","Kannan, M."],["dc.contributor.author","Farnworth, M. S."],["dc.contributor.author","Jahn, Olaf"],["dc.contributor.author","Göpfert, Martin C."],["dc.contributor.author","Stegmueller, Judith"],["dc.date.accessioned","2018-11-07T09:59:20Z"],["dc.date.available","2018-11-07T09:59:20Z"],["dc.date.issued","2015"],["dc.description.abstract","Neuronal health is essential for the long-term integrity of the brain. In this study, we characterized the novel E3 ubiquitin ligase ring finger protein 157 (RNF157), which displays a brain-dominant expression in mouse. RNF157 is a homolog of the E3 ligase mahogunin ring finger-1, which has been previously implicated in spongiform neurodegeneration. We identified RNF157 as a regulator of survival in cultured neurons and established that the ligase activity of RNF157 is crucial for this process. We also uncovered that independently of its ligase activity, RNF157 regulates dendrite growth and maintenance. We further identified the adaptor protein APBB1 (amyloid beta precursor protein-binding, family B, member 1 or Fe65) as an interactor and proteolytic substrate of RNF157 in the control of neuronal survival. Here, the nuclear localization of Fe65 together with its interaction partner RNA-binding protein SART3 (squamous cell carcinoma antigen recognized by T cells 3 or Tip110) is crucial to trigger apoptosis. In summary, we described that the E3 ligase RNF157 regulates important aspects of neuronal development."],["dc.identifier.doi","10.1038/cdd.2014.163"],["dc.identifier.isi","000350857200012"],["dc.identifier.pmid","25342469"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/37565"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Nature Publishing Group"],["dc.relation.issn","1476-5403"],["dc.relation.issn","1350-9047"],["dc.title","Regulation of neuronal survival and morphology by the E3 ubiquitin ligase RNF157"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]Details DOI PMID PMC WOS