Favaro, Plinio D.

[["dc.bibliographiccitation.artnumber","95"],["dc.bibliographiccitation.issue","5"],["dc.bibliographiccitation.journal","Journal of High Energy Physics"],["dc.bibliographiccitation.volume","2022"],["dc.contributor.author","Patra, S."],["dc.contributor.author","Bhardwaj, V."],["dc.contributor.author","Trabelsi, K."],["dc.contributor.author","Adachi, I."],["dc.contributor.author","Aihara, H."],["dc.contributor.author","Al Said, S."],["dc.contributor.author","Asner, D. M."],["dc.contributor.author","Atmacan, H."],["dc.contributor.author","Aushev, T."],["dc.contributor.author","Ayad, R."],["dc.contributor.author","Das, S."],["dc.contributor.author","Frey, A."],["dc.contributor.author","Hernandez, M."],["dc.contributor.author","Jin, Y."],["dc.contributor.author","Li, J."],["dc.contributor.author","Li, Y."],["dc.contributor.author","Paul, S."],["dc.contributor.authorgroup","The BELLE collaboration"],["dc.date.accessioned","2022-06-01T09:39:15Z"],["dc.date.accessioned","2022-08-16T12:38:16Z"],["dc.date.available","2022-06-01T09:39:15Z"],["dc.date.available","2022-08-16T12:38:16Z"],["dc.date.issued","2022-05-16"],["dc.date.updated","2022-07-29T11:35:17Z"],["dc.description.abstract","Abstract\r\n \r\n We present a search for the charged lepton-flavor-violating decays ϒ(1S) → ℓ±ℓ′∓ and radiative charged lepton-flavour-violating decays ϒ(1S) → γℓ±ℓ′∓ [ℓ,ℓ′ = e, μ, τ] using the 158 million ϒ(2S) sample collected by the Belle detector at the KEKB collider. This search uses ϒ(1S) mesons produced in ϒ(2S) → π+π− ϒ(1S) transitions. We do not find any significant signal, so we provide upper limits on the branching fractions at the 90% confidence level."],["dc.identifier.citation","Journal of High Energy Physics. 2022 May 16;2022(5):95"],["dc.identifier.doi","10.1007/JHEP05(2022)095"],["dc.identifier.pii","18383"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/108422"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/112737"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-572"],["dc.relation.eissn","1029-8479"],["dc.rights","CC BY 4.0"],["dc.rights.holder","The Author(s)"],["dc.rights.uri","https://creativecommons.org/licenses/by/4.0"],["dc.subject","e\r\n +-e\r\n − Experiments"],["dc.title","Search for charged lepton flavor violating decays of ϒ (1S)"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.subtype","original_ja"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.artnumber","e2006838"],["dc.bibliographiccitation.issue","12"],["dc.bibliographiccitation.journal","PLOS Biology"],["dc.bibliographiccitation.volume","16"],["dc.contributor.author","Favaro, Plinio D."],["dc.contributor.author","Huang, Xiaojie"],["dc.contributor.author","Hosang, Leon"],["dc.contributor.author","Stodieck, Sophia"],["dc.contributor.author","Cui, Lei"],["dc.contributor.author","Liu, Yu-Zhang"],["dc.contributor.author","Engelhardt, Karl-Alexander"],["dc.contributor.author","Schmitz, Frank"],["dc.contributor.author","Dong, Yan"],["dc.contributor.author","Löwel, Siegrid"],["dc.contributor.author","Schlüter, Oliver M."],["dc.date.accessioned","2019-07-09T11:49:59Z"],["dc.date.available","2019-07-09T11:49:59Z"],["dc.date.issued","2018"],["dc.description.abstract","The disc-large (DLG)-membrane-associated guanylate kinase (MAGUK) family of proteins forms a central signaling hub of the glutamate receptor complex. Among this family, some proteins regulate developmental maturation of glutamatergic synapses, a process vulnerable to aberrations, which may lead to neurodevelopmental disorders. As is typical for paralogs, the DLG-MAGUK proteins postsynaptic density (PSD)-95 and PSD-93 share similar functional domains and were previously thought to regulate glutamatergic synapses similarly. Here, we show that they play opposing roles in glutamatergic synapse maturation. Specifically, PSD-95 promoted, whereas PSD-93 inhibited maturation of immature α-amino-3-hydroxy-5-methyl-4-isoxazole propionic acid-type glutamate receptor (AMPAR)-silent synapses in mouse cortex during development. Furthermore, through experience-dependent regulation of its protein levels, PSD-93 directly inhibited PSD-95's promoting effect on silent synapse maturation in the visual cortex. The concerted function of these two paralogs governed the critical period of juvenile ocular dominance plasticity (jODP), and fine-tuned visual perception during development. In contrast to the silent synapse-based mechanism of adjusting visual perception, visual acuity improved by different mechanisms. Thus, by controlling the pace of silent synapse maturation, the opposing but properly balanced actions of PSD-93 and PSD-95 are essential for fine-tuning cortical networks for receptive field integration during developmental critical periods, and imply aberrations in either direction of this process as potential causes for neurodevelopmental disorders."],["dc.description.sponsorship","Open-Access-Publikationsfonds 2018"],["dc.identifier.doi","10.1371/journal.pbio.2006838"],["dc.identifier.pmid","30586380"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/15797"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/15829"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/59674"],["dc.language.iso","en"],["dc.notes.intern","Merged from goescholar"],["dc.relation.issn","1545-7885"],["dc.rights","CC BY 4.0"],["dc.rights.uri","https://creativecommons.org/licenses/by/4.0"],["dc.subject.ddc","610"],["dc.subject.ddc","612"],["dc.title","An opposing function of paralogs in balancing developmental synapse maturation"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]