Ellrott, Thomas

[["dc.bibliographiccitation.firstpage","12"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Obesity Facts"],["dc.bibliographiccitation.lastpage","24"],["dc.bibliographiccitation.volume","10"],["dc.contributor.author","Hauck, Carolin"],["dc.contributor.author","Weiss, Annegret"],["dc.contributor.author","Schulte, Erica Marla"],["dc.contributor.author","Meule, Adrian"],["dc.contributor.author","Ellrott, Thomas"],["dc.date.accessioned","2018-11-07T10:28:49Z"],["dc.date.available","2018-11-07T10:28:49Z"],["dc.date.issued","2017"],["dc.description.abstract","Background/Aims: To assess the prevalence and correlates of addictive-like eating behavior in Germany. Methods: The German version of the Yale Food Addiction Scale (YFAS) 2.0 was used to investigate, for the first time, the prevalence of 'food addiction' in a representative sample aged 18-65 years (N = 1,034). Results: The prevalence of 'food addiction' measured by the YFAS 2.0 was 7.9%. Individuals meeting criteria for 'food addiction' had higher BMI and were younger than individuals not meeting the threshold. Underweight (15.0%) and obese (17.2%) individuals exhibited the highest prevalence rate of 'food addiction'. Addictive-like eating was not associated with sex, education level, or place of residence. Conclusion: YFAS 2.0 'food addiction' was met by nearly 8% of the population. There is a non-linear relationship between addictive-like eating and BMI, with the highest prevalence among underweight and obese persons. These findings suggest that 'food addiction' may be a contributor to overeating but may also reflect a distinct phenotype of problematic eating behavior not synonymous with obesity. Further, the elevated prevalence of YFAS 2.0 'food addiction' among underweight individuals may reflect an overlap with eating disorders and warrants attention in future research. (C) 2017 The Author(s) Published by S. Karger GmbH, Freiburg"],["dc.description.sponsorship","'Institut Danone - Ernahrung fur Gesundheit e.V.'; Institute Danone"],["dc.identifier.doi","10.1159/000456013"],["dc.identifier.isi","000396486900002"],["dc.identifier.pmid","28190017"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/14943"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/43510"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","PUB_WoS_Import"],["dc.publisher","Karger"],["dc.relation.issn","1662-4033"],["dc.relation.issn","1662-4025"],["dc.rights","Goescholar"],["dc.rights.uri","https://goescholar.uni-goettingen.de/licenses"],["dc.title","Prevalence of 'Food Addiction' as Measured with the Yale Food Addiction Scale 2.0 in a Representative German Sample and Its Association with Sex, Age and Weight Categories"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1573"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","Eating and Weight Disorders"],["dc.bibliographiccitation.lastpage","1582"],["dc.bibliographiccitation.volume","25"],["dc.contributor.author","Hauck, Carolin"],["dc.contributor.author","Schipfer, Melanie"],["dc.contributor.author","Ellrott, Thomas"],["dc.contributor.author","Cook, Brian"],["dc.date.accessioned","2020-12-10T14:14:46Z"],["dc.date.available","2020-12-10T14:14:46Z"],["dc.date.issued","2019"],["dc.identifier.doi","10.1007/s40519-019-00794-6"],["dc.identifier.eissn","1590-1262"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/16686"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/71479"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.notes.intern","Merged from goescholar"],["dc.rights","CC BY 4.0"],["dc.rights.uri","https://creativecommons.org/licenses/by/4.0"],["dc.title","The relationship between food addiction and patterns of disordered eating with exercise dependence: in amateur endurance athletes"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1123"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","Thrombosis and Haemostasis"],["dc.bibliographiccitation.lastpage","1129"],["dc.bibliographiccitation.volume","100"],["dc.contributor.author","Dellas, Claudia"],["dc.contributor.author","Schaefer, Katrin"],["dc.contributor.author","Rohm, Ilonka"],["dc.contributor.author","Lankeit, Mareike"],["dc.contributor.author","Ellrott, Thomas"],["dc.contributor.author","Faustin, Vivien"],["dc.contributor.author","Riggert, Joachim"],["dc.contributor.author","Hasenfuß, Gerd"],["dc.contributor.author","Konstantinides, Stavros"],["dc.date.accessioned","2017-09-07T11:48:08Z"],["dc.date.available","2017-09-07T11:48:08Z"],["dc.date.issued","2008"],["dc.description.abstract","Clinical studies have shown that elevated leptin levels are an independent cardiovascular risk factor. However, little is known about the existence of platelet resistance to leptin in the setting of obesity. We examined the effects of leptin on platelet aggregation in morbidly obese subjects (n=40; BMI, 41.6 +/- 1.1 kg/m(2); leptin, 49.7 +/- 3.4 ng/ml) in comparison to normal-weight controls (n=36 BMI, 23.3 +/- 0.4 kg/m(2); leptin, 6.5 +/- 0.7 ng/ml).The aggregatory response to increasing concentrations of adenosine diphosphate (ADP) (2, 3,4,and 5 mu M) was significantly increased in platelets from obese compared to lean donors, reflecting a left shift in the dose-response curve. Plasma leptin levels, but not BMI, were significantly higher in subjects with stronger (above the median) compared to weaker (below the median) platelet aggregation at all ADP concentrations tested. In further experiments, stimulation (preincubation) with leptin (500 ng/ml) promotedADP-induced platelet aggregation by approximately 25%, and there was no difference between platelets from obese and those from lean donors regarding the responsiveness to leptin (p=0.99). Western blotting revealed that leptin induced phosphorylation of JAK2 and STAT3 to a similar extent in platelets from both groups. Expression of potential mediators of leptin resistance (SOCS3 and PTP IB) also did not differ in platelets from obese and control subjects. In conclusion,our data indicate that platelets from obese donors show increased aggregatory response to ADP, and that this might partly be the consequence of increased circulating leptin levels. Platelets from obese donors are not resistant to the enhancing effects of leptin on ADP-induced platelet aggregation."],["dc.identifier.doi","10.1160/TH08-05-0314"],["dc.identifier.gro","3143206"],["dc.identifier.isi","000261880900030"],["dc.identifier.pmid","19132239"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/6290"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/695"],["dc.notes.intern","WoS Import 2017-03-10 / Funder: German Research Foundation; University of Goettingen"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","final"],["dc.notes.submitter","PUB_WoS_Import"],["dc.publisher","Schattauer Gmbh-verlag Medizin Naturwissenschaften"],["dc.relation.issn","0340-6245"],["dc.rights","Goescholar"],["dc.rights.uri","https://goescholar.uni-goettingen.de/licenses"],["dc.title","Absence of leptin resistance in platelets from morbidly obese individuals may contribute to the increased thrombosis risk in obesity"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.subtype","original"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","328"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","Obesity science & practice"],["dc.bibliographiccitation.lastpage","337"],["dc.bibliographiccitation.volume","8"],["dc.contributor.affiliation","Raddatz, Dirk; 2\r\nDepartment of Gastroenterology, Gastrointestinal Oncology and Endocrinology\r\nUniversity Medical Center Göttingen\r\nGöttingen Germany"],["dc.contributor.affiliation","Ellrott, Thomas; 3\r\nInstitute for Nutrition and Psychology\r\nUniversity Medical Center Göttingen\r\nGöttingen Germany"],["dc.contributor.author","Rademacher, Jan‐Gerd"],["dc.contributor.author","Raddatz, Dirk"],["dc.contributor.author","Ellrott, Thomas"],["dc.date.accessioned","2022-04-04T12:44:03Z"],["dc.date.available","2022-04-04T12:44:03Z"],["dc.date.issued","2021"],["dc.date.updated","2022-06-15T00:10:07Z"],["dc.description.abstract","Abstract Objective Serum concentrations of the orexigenic hormone ghrelin fluctuate in anticipation of food intake. Moreover, presentation of food images causes an increase in serum ghrelin levels. Thus, the visual system may have a quantifiable role in the development of hunger via the endocrine system. The influence of macronutrient visualization on ghrelin has not yet been investigated. Methods In four separate sessions, ghrelin concentrations, insulin, and glucose levels were compared before and after the presentation of different pictures to 14 male participants. Pictures included neutral, non‐food‐related items or isocaloric dishes whose macronutrient composition corresponded predominately to protein/fat, simple carbohydrates, or complex carbohydrates. Results While pre/post ghrelin concentrations numerically increased in all sessions, significant increases were only observed following neutral and protein/fat pictures. The differences were not significant between food groups and compared to neutral images. Insulin levels decreased in all groups, but no significant differences were observed between sessions. The glucose concentrations were within the euglycemic range. Conclusion The results did not reproduce the induction of ghrelin secretion in different food images. Therefore, it is unclear whether the visual perception of food influences ghrelin secretion or whether separation into macronutrients changes the hormone response. Further research is required to differentiate the interactions of sensory‐specific satiety."],["dc.description.sponsorship","Open-Access-Publikationsfonds 2022"],["dc.identifier.doi","10.1002/osp4.577"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/106199"],["dc.language.iso","en"],["dc.relation.issn","2055-2238"],["dc.rights","CC BY 4.0"],["dc.title","Influence of food images with different macronutrient compositions on serum ghrelin levels: Analysis in healthy males"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.subtype","original_ja"],["dspace.entity.type","Publication"]]