Ahsen, Nicolas von

[["dc.bibliographiccitation.firstpage","871"],["dc.bibliographiccitation.issue","10"],["dc.bibliographiccitation.journal","Journal of Psychiatric Research"],["dc.bibliographiccitation.lastpage","875"],["dc.bibliographiccitation.volume","41"],["dc.contributor.author","Wilhelm, Julia"],["dc.contributor.author","von Ahsen, Nicolas"],["dc.contributor.author","Hillemacher, Thomas"],["dc.contributor.author","Bayerlein, Kristina"],["dc.contributor.author","Frieling, Helge"],["dc.contributor.author","Kornhuber, Johannes"],["dc.contributor.author","Bleich, Stefan"],["dc.date.accessioned","2018-11-07T10:57:13Z"],["dc.date.available","2018-11-07T10:57:13Z"],["dc.date.issued","2007"],["dc.description.abstract","Aim of this study was to investigate the possible association of apolipoprotein E (ApoE) gene polymorphism with a history of alcohol withdrawal seizures. We included 194 patients with alcohol dependence who were divided into patients with (SZ+) and without (SZ-) previous alcohol withdrawal seizures. ApoE genotypes were deter-mined using PCR. For statistical analysis we examined the number of ApoE alleles (ApoE2: n = 36; ApoE3: n = 311; ApoE4: n = 41). A significant positive association with a positive history of withdrawal seizures (SZ+) was found in the ApoE3 allele group (Fisher's exact test: p = 0.006) while a significant negative association was observed in the ApoE2 allele group (Fisher's exact test: p = 0.029). For the ApoE4 allele group no significant differences were found regarding a history of withdrawal seizures. Our findings suggest an association between the apolipoprotein E3 gene variant and an elevated risk of alcohol withdrawal seizures. These preliminary results must be validated in further studies. (c) 2006 Elsevier Ltd. All rights reserved."],["dc.identifier.doi","10.1016/j.jpsychires.2006.07.011"],["dc.identifier.isi","000247131300008"],["dc.identifier.pmid","16959267"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/50189"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Pergamon-elsevier Science Ltd"],["dc.relation.issn","0022-3956"],["dc.title","Apolipoprotein E gene polymorphism and previous alcohol withdrawal seizures"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","249"],["dc.bibliographiccitation.issue","5"],["dc.bibliographiccitation.journal","Ophthalmic Research"],["dc.bibliographiccitation.lastpage","256"],["dc.bibliographiccitation.volume","40"],["dc.contributor.author","Roedl, J. B."],["dc.contributor.author","Bleich, Stefan"],["dc.contributor.author","Schloetzer-Schrehardt, Ursula"],["dc.contributor.author","von Ahsen, Nicolas"],["dc.contributor.author","Kornhuber, Johannes"],["dc.contributor.author","Naumann, G. O. H."],["dc.contributor.author","Kruse, F. E."],["dc.contributor.author","Juenemann, A. G. M."],["dc.date.accessioned","2018-11-07T11:20:31Z"],["dc.date.available","2018-11-07T11:20:31Z"],["dc.date.issued","2008"],["dc.description.abstract","Aims: We assessed homocysteine (Hcy) levels in tear fluid and plasma of patients with primary open-angle glaucoma (POAG). We determined the association between Hcy levels, dry eye syndrome and B vitamin status. Methods: This prospective case-control study included 36 patients with POAG and 36 controls. Hcy concentrations were measured by high-performance liquid chromatography. Results: Patients with POAG had significantly higher mean Hcy levels both in tear fluid (205 +/- 84 nmol/l; p < 0.001, t test) and in plasma (13.43 +/- 3.53 mu mol/l; p = 0.001, t test) than control subjects (130 +/- 53 nmol/l and 10.50 +/- 3.33 mu mol/l, respectively). Hcy in tear fluid was significantly correlated with plasma Hcy in POAG patients (r = 0.459; p = 0.005, Pearson's correlation), but not in controls (r = 0.068; p = 0.695). POAG patients with dry eye disease had significantly higher Hcy levels both in tear fluid and plasma than POAG patients without dry eye disease. There was no association between Hcy levels and B vitamin status in subjects with POAG. Conclusions: The study suggests increased Hcy levels in tear fluid and plasma of patients with POAG. Elevated Hcy levels might be a risk factor for POAG and dry eye syndrome in subjects with glaucoma. Copyright (C) 2008 S. Karger AG, Basel."],["dc.identifier.doi","10.1159/000127832"],["dc.identifier.isi","000258318600006"],["dc.identifier.pmid","18437035"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/9359"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/55551"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Karger"],["dc.relation.issn","0030-3747"],["dc.rights","Goescholar"],["dc.rights.uri","https://goescholar.uni-goettingen.de/licenses"],["dc.title","Increased homocysteine levels in tear fluid of patients with primary open-angle glaucoma"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","2749"],["dc.bibliographiccitation.issue","12"],["dc.bibliographiccitation.journal","Neuroreport"],["dc.bibliographiccitation.lastpage","2752"],["dc.bibliographiccitation.volume","11"],["dc.contributor.author","Bleich, Stefan"],["dc.contributor.author","Degner, Detlef"],["dc.contributor.author","Bandelow, Borwin"],["dc.contributor.author","von Ahsen, Nicolas"],["dc.contributor.author","Ruther, Eckart"],["dc.contributor.author","Kornhuber, Johannes"],["dc.date.accessioned","2018-11-07T10:33:14Z"],["dc.date.available","2018-11-07T10:33:14Z"],["dc.date.issued","2000"],["dc.description.abstract","An adaptive consequence of prolonged ethanol consumption is a compensatory up-regulation of NMDA receptors in certain brain areas. Taking into account that homocysteine and its breakdown products (i.e. homocysteic acid) are putative neurotransmitters and agonists at the NMDA receptor, the aim of this study was to assess the influence of levels of homocysteine on alcohol withdrawal seizures. Six patients with chronic alcoholism who suffered from withdrawal seizures had significantly higher levels of homocysteine on admission (84.7 +/- 29.8 mu mol/l) than patients (n = 26) who did not develop seizures (30.2 +/- 23.2 mu mol/l; U = 8.0, p = 0.0007). Furthermore, seizure patients had significantly lower levels of folate and significantly higher blood alcohol concentrations. Using a logistic regression analysis, withdrawal seizures were best predicted by a high homocysteine level on admission (P < 0.01; odds ratio = 1.05). Homocysteine levels on admission may be a useful screening method to identify patients at risk for withdrawal seizures. NeuroReport 11:2749-2752 (C) 2000 Lippincott Williams & Wilkins."],["dc.identifier.doi","10.1097/00001756-200008210-00028"],["dc.identifier.isi","000089003300036"],["dc.identifier.pmid","10976956"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/44558"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Lippincott Williams & Wilkins"],["dc.relation.issn","0959-4965"],["dc.title","Plasma homocysteine is a predictor of alcohol withdrawal seizures"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","683"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","Journal of Neural Transmission"],["dc.bibliographiccitation.lastpage","689"],["dc.bibliographiccitation.volume","111"],["dc.contributor.author","Jordan, W."],["dc.contributor.author","Berger, C."],["dc.contributor.author","Cohrs, Stefan"],["dc.contributor.author","Rodenbeck, Andrea"],["dc.contributor.author","Mayer, G."],["dc.contributor.author","Niedmann, P. D."],["dc.contributor.author","von Ahsen, Nicolas"],["dc.contributor.author","Ruther, Eckart"],["dc.contributor.author","Kornhuber, Johannes"],["dc.contributor.author","Bleich, Stefan"],["dc.date.accessioned","2018-11-07T10:48:34Z"],["dc.date.available","2018-11-07T10:48:34Z"],["dc.date.issued","2004"],["dc.description.abstract","Assessment of serum total homocysteine (tHcy) in patients with obstructive sleep apnea (OSA) syndrome is highly relevant since both are strongly associated with stroke and cognitive dysfunction. Seven of 16 untreated OSA patients showed tHcy levels exceeding 11.7 mumol/l. The circadian pattern of serum tHcy in untreated and treated patients (p < 0.001) implied a diagnostic impact of blood sampling time. Treatment with continuous positive airway pressure (CPAP) effectively lowered tHcy levels in patients by about 30% (p < 0.005) and thus probably the (hyper)homocysteinemia-related cognitive dysfunction and the risk for cardio-/cerebrovascular diseases."],["dc.identifier.doi","10.1007/s00702-004-0130-2"],["dc.identifier.isi","000221692600003"],["dc.identifier.pmid","15168215"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/48229"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.publisher.place","Wien"],["dc.relation.issn","0300-9564"],["dc.title","CPAP-therapy effectively lowers serum homocysteine in obstructive sleep apnea syndrome"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","721"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","American Journal of Ophthalmology"],["dc.bibliographiccitation.lastpage","723"],["dc.bibliographiccitation.volume","139"],["dc.contributor.author","Junemann, AGM"],["dc.contributor.author","von Ahsen, Nicolas"],["dc.contributor.author","Reulbach, Udo"],["dc.contributor.author","Roedl, J."],["dc.contributor.author","Bonsch, D."],["dc.contributor.author","Kornhuber, Johannes"],["dc.contributor.author","Kruse, F. E."],["dc.contributor.author","Bleich, Stefan"],["dc.date.accessioned","2018-11-07T11:09:07Z"],["dc.date.available","2018-11-07T11:09:07Z"],["dc.date.issued","2005"],["dc.description.abstract","PURPOSE: To estimate the prevalence of C677T single nucleotide polymorphism in the 5,10-methylentetrahy drofolate reductase (MTHFR) gene in primary open-angle glaucoma (POAG) and pseudoexfoliation open, angle glaucoma (PEXG). DESIGN: Case-control study METHODS: MTHFR was assessed in 147 patients (76 POAG, 71 PEXG) and 71 control subjects with cataract. Associations of genotypes were assessed by Armitage's trend test and the corresponding odds ratio (OR) for allele positivity with 95% confidence interval (CI). RESULTS: We observed significant evidence of a higher prevalence of C677T in POAG (9% homozygote, 49% heterozygote, 42% wildtype, P =.01, OR = 2.38, 95% CI 1.23-4.62), but not in PEXG (9% homozygote, 41% heterozygote, 50% wildtype, P = .09, OR = 1.78, 95% CI 0.91-3.50) compared with the controls (3% homozygote, 34% heterozygote, 63% wildtype). CONCLUSIONS: The MTHFR C677T variant leading to moderate hyperhomocysteinemia may play a role in the pathogenesis of POAG acting as a genetic risk factor. (c) 2005 by Elsevier Inc. All rights reserved."],["dc.identifier.doi","10.1016/j.ajo.2004.09.081"],["dc.identifier.isi","000228222300026"],["dc.identifier.pmid","15808177"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/52937"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Elsevier Science Inc"],["dc.relation.issn","0002-9394"],["dc.title","C677T variant in the methylentetrahydrofolate reductase gene is a genetic risk factor for primary open-angle glaucoma"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1499"],["dc.bibliographiccitation.issue","12"],["dc.bibliographiccitation.journal","Journal of Neural Transmission"],["dc.bibliographiccitation.lastpage","1504"],["dc.bibliographiccitation.volume","109"],["dc.contributor.author","Bleich, Stefan"],["dc.contributor.author","Junemann, A."],["dc.contributor.author","von Ahsen, Nicolas"],["dc.contributor.author","Lausen, B."],["dc.contributor.author","Ritter, K."],["dc.contributor.author","Beck, G."],["dc.contributor.author","Naumann, GOH"],["dc.contributor.author","Kornhuber, Johannes"],["dc.date.accessioned","2018-11-07T09:46:09Z"],["dc.date.available","2018-11-07T09:46:09Z"],["dc.date.issued","2002"],["dc.description.abstract","Homocysteine levels and the frequency of heterozygous methylenetetrahydrofolate reductase (MTHFR) C677T mutation are increased in open-angle glaucoma. Since homocysteine can induce vascular injury, alterations in extracellular matrix remodelling, and neuronal cell death, these findings may have important implications for understanding glaucomatous optic neuropathy."],["dc.identifier.doi","10.1007/s007020200097"],["dc.identifier.isi","000180070000007"],["dc.identifier.pmid","12486490"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/34802"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.publisher.place","Wien"],["dc.relation.issn","0300-9564"],["dc.title","Homocysteine and risk of open-angle glaucoma"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","117"],["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","The Pharmacogenomics Journal"],["dc.bibliographiccitation.lastpage","121"],["dc.bibliographiccitation.volume","8"],["dc.contributor.author","Wilhelm, Julia"],["dc.contributor.author","Frieling, Helge"],["dc.contributor.author","von Ahsen, Nicolas"],["dc.contributor.author","Hillemacher, Thomas"],["dc.contributor.author","Kornhuber, Johannes"],["dc.contributor.author","Bleich, Stefan"],["dc.date.accessioned","2018-11-07T11:16:48Z"],["dc.date.available","2018-11-07T11:16:48Z"],["dc.date.issued","2008"],["dc.description.abstract","There is growing evidence that disadvantageous influences of the apolipoprotein E4 allele in the central nervous system are modified by environmental and dietary conditions. The present study investigated the gene-environment interaction of apolipoprotein E4 with homocysteine serum levels in patients with alcohol dependence with regard to alcohol-related hippocampal volume loss using volumetric high-resolution magnetic resonance imaging. We included 52 patients with alcohol-dependence. ApoE genotypes, homocysteine serum levels and hippocampal volumes were determined. We found a significant impact of homocysteine ( F = 13.2; df = 1; P < 0.001; 1-beta = 0.95), not for ApoE4 genotype (F = 0.482; df = 1; P = 0.49; 1 - beta = 0.05) on hippocampal volume. There was a significant interaction between both factors ( ApoE4 x Hcy; F = 8.8; df 1; P = 0.005; 1 - beta = 0.80). The ApoE4 allele constitutes a risk factor for hippocampal volume loss in patients with alcohol dependence under the conditions of hyperhomocysteinemia. We suggest that the disadvantageous effects of apolipoprotein E4 on alcohol-related brain volume loss are based on certain gene - environment interactions."],["dc.identifier.doi","10.1038/sj.tpj.6500453"],["dc.identifier.isi","000254123900005"],["dc.identifier.pmid","17420762"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/54677"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Nature Publishing Group"],["dc.relation.issn","1470-269X"],["dc.title","Apolipoprotein E polymorphism, homocysteine serum levels and hippocampal volume in patients with alcoholism: an investigation of a gene-environment interaction"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","162"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","American Journal of Ophthalmology"],["dc.bibliographiccitation.lastpage","164"],["dc.bibliographiccitation.volume","138"],["dc.contributor.author","Bleich, Stefan"],["dc.contributor.author","Roedl, J."],["dc.contributor.author","von Ahsen, N."],["dc.contributor.author","Schlotzer-Schrehardt, U."],["dc.contributor.author","Reulbach, Udo"],["dc.contributor.author","Beck, G."],["dc.contributor.author","Kruse, F. E."],["dc.contributor.author","Naumann, GOH"],["dc.contributor.author","Kornhuber, Johannes"],["dc.contributor.author","Junemann, AGM"],["dc.date.accessioned","2018-11-07T10:47:24Z"],["dc.date.available","2018-11-07T10:47:24Z"],["dc.date.issued","2004"],["dc.description.abstract","PURPOSE: To determine total homocysteine levels in aqueous humor of pseudoexfoliation open-angle glaucoma patients. DESIGN: Case-control study. METHODS: Total homocysteine levels were measured by enzyme-linked immunosorbent assay in aqueous humor and plasma of 29 patients with pseudoexfoliation glaucoma and 31 control patients with cataract. Patients with factors affecting homocysteine levels were excluded. RESULTS: We observed significantly elevated (twofold) homocysteine levels in the aqueous humor of the glaucoma patients (Z = -5.11, P < .0001). Additionally, the calculated ratio (plasma:aqueous humor) was significantly lower in these patients (Z = -3.57, P < .001), and aqueous homocysteine was significantly correlated with their respective elevated plasma levels (r = .42, P = .02). CONCLUSIONS: Because homocysteine induces vascular injury and alterations of extracellular matrix, high aqueous homocysteine may trigger the abnormal matrix accumulation characteristic. It may reflect the proposed impairment of the blood-aqueous barrier of pseudoexfoliation open-angle glaucoma and be involved in its pathogenesis. (C) 2004 by Elsevier Inc. All rights reserved."],["dc.identifier.doi","10.1016/j.ajo.2004.02.027"],["dc.identifier.isi","000222568200033"],["dc.identifier.pmid","15234308"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/47955"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Elsevier Science Inc"],["dc.relation.issn","0002-9394"],["dc.title","Elevated homocysteine levels in aqueous humor of patients with pseudoexfoliation glaucoma"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","445"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","Journal of Neural Transmission"],["dc.bibliographiccitation.lastpage","450"],["dc.bibliographiccitation.volume","114"],["dc.contributor.author","Roedl, J. B."],["dc.contributor.author","Bleich, Stefan"],["dc.contributor.author","Reulbach, Udo"],["dc.contributor.author","von Ahsen, Nicolas"],["dc.contributor.author","Schloetzer-Schrehardt, Ursula"],["dc.contributor.author","Rejdak, R."],["dc.contributor.author","Naumann, G. O. H."],["dc.contributor.author","Kruse, F. E."],["dc.contributor.author","Kornhuber, Johannes"],["dc.contributor.author","Juenemann, A. G. M."],["dc.date.accessioned","2018-11-07T11:03:37Z"],["dc.date.available","2018-11-07T11:03:37Z"],["dc.date.issued","2007"],["dc.description.abstract","We determined homocysteine (Hcy) levels in aqueous humor (AH) and plasma and their association with B-vitamin levels in patients with primary open-angle glaucoma (POAG) and controls. Both AH Hcy and plasma Hcy levels were significantly increased in POAG, and elevation of AH Hcy and plasma Hcy was a significant risk factor for POAG. In contrast to controls, neither plasma nor AH Hcy of POAG patients demonstrated a significant association with important non-genetic determinants of elevated Hcy such as low B-vitamin levels, increasing age and caffeine consumption. Considering that Hcy is a neurotoxin that induces apoptotic retinal ganglion cell death via stimulation of the N-methyl-D-asparate (NMDA) receptor, increased Hcy concentrations in AH and plasma might contribute to the optic nerve damage in POAG."],["dc.identifier.doi","10.1007/s00702-006-0556-9"],["dc.identifier.isi","000245133400007"],["dc.identifier.pmid","16932990"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/51661"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.publisher.place","Wien"],["dc.relation.issn","0300-9564"],["dc.title","Homocysteine levels in aqueous humor and plasma of patients with primary open-angle glaucoma"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","151"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","Alcohol"],["dc.bibliographiccitation.lastpage","156"],["dc.bibliographiccitation.volume","37"],["dc.contributor.author","Wilhelm, Julia"],["dc.contributor.author","von Ahsen, Nicolas"],["dc.contributor.author","Frieling, Helge"],["dc.contributor.author","Hillemacher, Thomas"],["dc.contributor.author","Bayerlein, K."],["dc.contributor.author","Bonsch, D."],["dc.contributor.author","Ziegenbein, M."],["dc.contributor.author","Kornhuber, Johannes"],["dc.contributor.author","Bleich, Stefan"],["dc.date.accessioned","2018-11-07T10:54:26Z"],["dc.date.available","2018-11-07T10:54:26Z"],["dc.date.issued","2005"],["dc.description.abstract","Aim of this prospective study was to investigate a possible association between the apolipoprotein E4 (ApoE4) genotype and clinically well-known cognition deficits during alcohol withdrawal, We examined 172 patients with alcohol dependence (137 men, 35 women) during withdrawal treatment. The ApoE genotype was determined in all patients using polymerase chain reaction. Cognitive function was assessed applying the c.I.-Test on day 0 (admission) and on day 7 of withdrawal treatment. Using Pearson's chi(2) test we found no significant association between the ApoE4 genotype and cognition deficits for both dates (day 0: p = .463; day 7: p = .760). Moreover, multivariate logistic regression analyses revealed no significant association between presence of the ApoE4 allele and cognitive dysfunction. Even though ApoE4 plays an important role in alcoholism-related brain atrophy and cognition deficits in demented as well as in nondemented healthy elderly people, this study provides no evidence for an association with short-term cognition deficits during alcohol withdrawal. (c) 2005 Elsevier Inc. All rights reserved."],["dc.identifier.doi","10.1016/j.alcohol.2006.01.002"],["dc.identifier.isi","000237868400004"],["dc.identifier.pmid","16713503"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/49560"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Pergamon-elsevier Science Ltd"],["dc.relation.issn","0741-8329"],["dc.title","Apolipoprotein E4 genotype is not associated with short-term cognition deficits during alcohol withdrawal"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]