Schroeter, Marco Robin

[["dc.bibliographiccitation.firstpage","423"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","International Journal of Stroke"],["dc.bibliographiccitation.lastpage","429"],["dc.bibliographiccitation.volume","14"],["dc.contributor.author","Schnieder, Marlena"],["dc.contributor.author","Siddiqui, Tariq"],["dc.contributor.author","Karch, André"],["dc.contributor.author","Bähr, Mathias"],["dc.contributor.author","Hasenfuß, Gerd"],["dc.contributor.author","Schroeter, Marco R"],["dc.contributor.author","Liman, Jan"],["dc.date.accessioned","2020-12-10T18:38:36Z"],["dc.date.available","2020-12-10T18:38:36Z"],["dc.date.issued","2018"],["dc.identifier.doi","10.1177/1747493018816511"],["dc.identifier.eissn","1747-4949"],["dc.identifier.issn","1747-4930"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/77384"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","Low flow in the left atrial appendage assessed by transesophageal echocardiography is associated with increased stroke severity—Results of a single-center cross-sectional study"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","2121"],["dc.bibliographiccitation.issue","12"],["dc.bibliographiccitation.journal","The FASEB Journal"],["dc.bibliographiccitation.lastpage","2130"],["dc.bibliographiccitation.volume","15"],["dc.contributor.author","Wagner, Andreas H."],["dc.contributor.author","Schroeter, M. R."],["dc.contributor.author","Hecker, M."],["dc.date.accessioned","2018-11-07T08:34:52Z"],["dc.date.available","2018-11-07T08:34:52Z"],["dc.date.issued","2001"],["dc.description.abstract","We investigated the hypothesis that the antiatherosclerotic effect of 17 beta -estradiol (E-2) is due to a shift in the nitric oxide (NO)/superoxide (O-2(-)) balance in the vessel wall, thereby increasing the bioavailability of NO. In human umbilical vein cultured endothelial cells, E-2 (1-100 nmol/l), but not 17 alpha -estradiol, caused a time- and concentration-dependent decrease in expression of the NADPH oxidase subunit gp91phox (up to 60% inhibition at both the mRNA and protein level). This effect was prevented by coincubation with the estrogen receptor antagonists tamoxifen and ICI 182,780 (1 mu mol/l each). Within the same concentration range, E-2 also up-regulated endothelial nitric oxide synthase expression (similar to twofold). Moreover, preincubation of the cells with E-2 or a gp91phox antisense oligonucleotide significantly decreased their capacity to generate O-2(-) on phorbol ester stimulation (i.e., assembly of the active NADPH oxidase complex). Blockade of NO synthase activity, on the other hand, had no effect on phorbol ester-stimulated O-2(-) formation. In addition, E-2 (100 nmol/l) inhibited the increase in adhesion molecule and chemokine expression in cells exposed to cyclic strain. Cyclic strain enhanced endothelial O-2(-) formation, thereby offsetting the inhibitory effect of NO on the expression of these gene products. E-2 thus seems to act as an antioxidant at the genomic level which by improving the NO/O-2(-) balance normalizes expression of proatherosclerotic gene products in endothelial cells."],["dc.identifier.doi","10.1096/fj.01-0123com"],["dc.identifier.isi","000171920400023"],["dc.identifier.pmid","11641238"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/17925"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Federation Amer Soc Exp Biol"],["dc.relation.issn","0892-6638"],["dc.title","17 beta-estradiol inhibition of NADPH oxidase expression in human endothelial cells"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.issue","18"],["dc.bibliographiccitation.journal","Circulation"],["dc.bibliographiccitation.volume","120"],["dc.contributor.author","Leifheit, Maren"],["dc.contributor.author","Conrad, Gaby"],["dc.contributor.author","Heide, Nana-Maria"],["dc.contributor.author","Schroeter, Marco"],["dc.contributor.author","Hasenfuß, Gerd"],["dc.contributor.author","Konstantinides, Stavros V."],["dc.contributor.author","Schaefer, Katrin"],["dc.date.accessioned","2018-11-07T11:22:15Z"],["dc.date.available","2018-11-07T11:22:15Z"],["dc.date.issued","2009"],["dc.format.extent","S1151"],["dc.identifier.isi","000271831504246"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/55953"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Lippincott Williams & Wilkins"],["dc.publisher.place","Philadelphia"],["dc.relation.conference","82nd Scientific Session of the American-Heart-Association"],["dc.relation.eventlocation","Orlando, FL"],["dc.relation.issn","0009-7322"],["dc.title","Overexpression of Integrin Beta5 Enhances the Paracrine Angiogenic Properties of Early Outgrowth Endothelial Progenitor Cells via Sire Kinase-Mediated Activation of STAT3"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","107"],["dc.bibliographiccitation.journal","International Journal of Cardiology"],["dc.bibliographiccitation.lastpage","111"],["dc.bibliographiccitation.volume","220"],["dc.contributor.author","Tichelbäcker, Tobias"],["dc.contributor.author","Puls, Miriam"],["dc.contributor.author","Jacobshagen, Claudius"],["dc.contributor.author","Hasenfuß, Gerd"],["dc.contributor.author","Schillinger, Wolfgang"],["dc.contributor.author","Huenlich, Mark"],["dc.contributor.author","Schroeter, Marco Robin"],["dc.date.accessioned","2017-09-07T11:44:37Z"],["dc.date.available","2017-09-07T11:44:37Z"],["dc.date.issued","2016"],["dc.description.abstract","Background: Percutaneous mitral valve repair using MitraClip (R) (MC) is a well-established method for a subset of patients with severe mitral regurgitation (MR) and high risk for surgical intervention. Amplatzer (R) Cardiac Plug (ACP) occludes left atrial appendage and allows the discontinuation of oral anticoagulation and prevention of thromboembolic stroke. Due to the need for femoral and transseptal access in both procedures, a single approach could lead to minor risk of further complications and shorter cumulative intervention time. Methods: We systematically analysed all four patients who underwent a combined procedure with MC and ACP in our heart-centre. All procedures were performed under fluoroscopic as well as echocardiographic guidance, and follow-up controls in a midterm period were carried out. Results: In all patients (2 male/female; age 73-88 years), MC (1-2 Clips) and ACP (size 18-28mm) were successfully implanted in one procedure (mean total time: 114 +/- 17 min). At least moderate MR was achieved and two patients had no complications and therefore were discharged early. In a third patient, a dislocation of ACP occurred 2 h after the implantation. The oldest patient developed a respiratory insufficiency due to cardiac decompensation and further complications. Conclusion: A combination of MC and ACP in a single procedure was feasible in this first case series of patients without a significant extension of procedure time. However, it might be important to select patients carefully. The location of optimal transseptal puncture may be challenging in regard to ACP placement, even in experienced hands and subsequent complications can occur. (C) 2016 Elsevier Ireland Ltd. All rights reserved."],["dc.identifier.doi","10.1016/j.ijcard.2016.06.170"],["dc.identifier.gro","3141615"],["dc.identifier.isi","000381582000019"],["dc.identifier.pmid","27389439"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/1456"],["dc.notes.intern","WoS Import 2017-03-10 / Funder: Abbott Vascular; St. Jude Medical"],["dc.notes.status","final"],["dc.notes.submitter","PUB_WoS_Import"],["dc.publisher","Elsevier Ireland Ltd"],["dc.relation.eissn","1874-1754"],["dc.relation.issn","0167-5273"],["dc.title","MitraClip (R) and Amplatzer (R) cardiac plug implantation in a single procedure: A reasonable approach?"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.subtype","original"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","204"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","Cardiovascular Research"],["dc.bibliographiccitation.lastpage","216"],["dc.bibliographiccitation.volume","111"],["dc.contributor.author","Gogiraju, Rajinikanth"],["dc.contributor.author","Schroeter, Marco R."],["dc.contributor.author","Bochenek, Magdalena L."],["dc.contributor.author","Hubert, Astrid"],["dc.contributor.author","Muenzel, Thomas"],["dc.contributor.author","Hasenfuß, Gerd"],["dc.contributor.author","Schaefer, Katrin"],["dc.date.accessioned","2017-09-07T11:44:47Z"],["dc.date.available","2017-09-07T11:44:47Z"],["dc.date.issued","2016"],["dc.description.abstract","Cardiac angiogenesis is an important determinant of heart failure. We examined the hypothesis that protein tyrosine phosphatase (PTP)-1B, a negative regulator of vascular endothelial growth factor (VEGF) receptor-2 activation, is causally involved in the cardiac microvasculature rarefaction during hypertrophy and that deletion of PTP1B in endothelial cells prevents the development of heart failure. Cardiac hypertrophy was induced by transverse aortic constriction (TAC) in mice with endothelial-specific deletion of PTP1B (End.PTP1B-KO) and controls (End.PTP1B-WT). Survival up to 20 weeks after TAC was significantly improved in mice lacking endothelial PTP1B. Serial echocardiography revealed a better systolic pump function, less pronounced cardiac hypertrophy, and left ventricular dilation compared with End.PTP1B-WT controls. Histologically, banded hearts from End.PTP1B-KO mice exhibited increased numbers of PCNA-positive, proliferating endothelial cells resulting in preserved cardiac capillary density and improved perfusion as well as reduced hypoxia, apoptotic cell death, and fibrosis. Increased relative VEGFR2 and ERK1/2 phosphorylation and greater eNOS expression were present in the hearts of End.PTP1B-KO mice. The absence of PTP1B in endothelial cells also promoted neovascularization following peripheral ischaemia, and bone marrow transplantation excluded a major contribution of Tie2-positive haematopoietic cells to the improved angiogenesis in End.PTP1B-KO mice. Increased expression of caveolin-1 as well as reduced NADPH oxidase-4 expression, ROS generation and TGF beta signalling were observed and may have mediated the cardioprotective effects of endothelial PTP1B deletion. Endothelial PTP1B deletion improves cardiac VEGF signalling and angiogenesis and protects against chronic afterload-induced heart failure. PTP1B may represent a useful target to preserve cardiac function during hypertrophy."],["dc.identifier.doi","10.1093/cvr/cvw101"],["dc.identifier.gro","3141642"],["dc.identifier.isi","000383197000008"],["dc.identifier.pmid","27207947"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/4455"],["dc.identifier.url","https://sfb1002.med.uni-goettingen.de/production/literature/publications/296"],["dc.notes.intern","WoS Import 2017-03-10"],["dc.notes.status","final"],["dc.notes.submitter","PUB_WoS_Import"],["dc.relation","SFB 1002: Modulatorische Einheiten bei Herzinsuffizienz"],["dc.relation","SFB 1002 | C06: Mechanismen und Regulation der koronaren Gefäßneubildung"],["dc.relation.eissn","1755-3245"],["dc.relation.issn","0008-6363"],["dc.relation.workinggroup","RG Hasenfuß (Transition zur Herzinsuffizienz)"],["dc.relation.workinggroup","RG Schäfer (Translationale Vaskuläre Biologie)"],["dc.title","Endothelial deletion of protein tyrosine phosphatase-1B protects against pressure overload-induced heart failure in mice"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.subtype","original_ja"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","European Heart Journal - Case Reports"],["dc.bibliographiccitation.volume","6"],["dc.contributor.author","Schroeter, Marco R"],["dc.contributor.author","Klingel, Karin"],["dc.contributor.author","Korsten, Peter"],["dc.contributor.author","Hasenfuß, Gerd"],["dc.contributor.editor","De Potter, Tom"],["dc.contributor.editor","Vidal-Perez, Rafael"],["dc.contributor.editor","Nistor, Dan Octavian"],["dc.contributor.editor","Agarwal, Megha"],["dc.contributor.editor","Sunjaya, Antony Paulo"],["dc.date.accessioned","2022-04-01T10:01:50Z"],["dc.date.available","2022-04-01T10:01:50Z"],["dc.date.issued","2022"],["dc.description.abstract","Abstract Background Lyme disease is a tick-borne multisystem infection. The most common cardiac manifestation is an acute presentation of Lyme carditis, which often manifests as conduction disorder and rarely as myocarditis. Case summary We report the case of a 37-year-old male with a history of microscopic polyangiitis receiving immunosuppressive therapy. He was admitted for severe dyspnoea secondary to acute heart failure. Echocardiography and cardiac magnetic resonance imaging indicated a severely reduced left ventricular ejection fraction (LVEF) with global hypokinesia. Coronary heart disease was excluded, and endomyocardial biopsies (EMB) were performed. The left ventricular EMB revealed a rare case of fulminant Lyme carditis with evidence of typical lymphocytic myocarditis. Borrelia afzelii-DNA was detected without any relevant atrioventricular blockage or systemic signs of Lyme disease. The patient had no clinically apparent tick-borne infection or self-reported history of a tick bite. Immunological testing revealed a positive ELISA and Immunoblot for anti-Borrelia immunoglobulin G antibodies. After specific intravenous antibiotic therapy and optimized medical therapy for heart failure, the LVEF recovered, and the patient could be discharged in an improved condition. Repeat EMB a few months later revealed a dramatic regression of the cardiac inflammation and absence of Borrelia DNA in the myocardium. Discussion A severely reduced LVEF can be the primary manifestation of Lyme disease even without typical systemic findings and can have a favourable prognosis with antibiotic treatment. A thorough workup for Lyme carditis is required in patients with unexplained heart failure, particularly with EMB, especially in immunosuppressed patients."],["dc.identifier.doi","10.1093/ehjcr/ytac062"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/105759"],["dc.language.iso","en"],["dc.notes.intern","DOI-Import GROB-530"],["dc.relation.eissn","2514-2119"],["dc.title","Fulminant Lyme myocarditis without any other signs of Lyme disease in a 37-year-old male patient with microscopic polyangiitis—a case report"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","333"],["dc.bibliographiccitation.issue","05"],["dc.bibliographiccitation.journal","Aktuelle Kardiologie"],["dc.bibliographiccitation.lastpage","338"],["dc.bibliographiccitation.volume","5"],["dc.contributor.author","Schroeter, M."],["dc.date.accessioned","2020-12-10T18:12:21Z"],["dc.date.available","2020-12-10T18:12:21Z"],["dc.date.issued","2016"],["dc.identifier.doi","10.1055/s-0042-114033"],["dc.identifier.eissn","2193-5211"],["dc.identifier.issn","2193-5203"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/74334"],["dc.language.iso","de"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","Primär- und Sekundärprävention der KHK – bekannte und neue Aspekte"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","473"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","Clinical Research in Cardiology"],["dc.bibliographiccitation.lastpage","476"],["dc.bibliographiccitation.volume","102"],["dc.contributor.author","Schroeter, Marco R."],["dc.contributor.author","Hasenfuß, Gerd"],["dc.contributor.author","Zabel, Markus"],["dc.contributor.author","Vollmann, Dirk"],["dc.date.accessioned","2017-09-07T11:47:41Z"],["dc.date.available","2017-09-07T11:47:41Z"],["dc.date.issued","2013"],["dc.identifier.doi","10.1007/s00392-013-0557-2"],["dc.identifier.gro","3142348"],["dc.identifier.isi","000319081700008"],["dc.identifier.pmid","23529655"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/7286"],["dc.notes.intern","WoS Import 2017-03-10"],["dc.notes.status","final"],["dc.notes.submitter","PUB_WoS_Import"],["dc.relation.issn","1861-0684"],["dc.title","Atrial standstill in a patient with progressive severe heart failure"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.subtype","letter_note"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.issue","16"],["dc.bibliographiccitation.journal","Circulation"],["dc.bibliographiccitation.volume","116"],["dc.contributor.author","Heida, Nana-Maria"],["dc.contributor.author","Schroeter, M. R."],["dc.contributor.author","Cheng, I-Fen"],["dc.contributor.author","Mueller, Jan-Peter"],["dc.contributor.author","Hasenfuß, Gerd"],["dc.contributor.author","Konstantindes, Stavros"],["dc.contributor.author","Schaefer, Katrin"],["dc.date.accessioned","2018-11-07T10:57:45Z"],["dc.date.available","2018-11-07T10:57:45Z"],["dc.date.issued","2007"],["dc.format.extent","839"],["dc.identifier.isi","000250394303788"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/50323"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Lippincott Williams & Wilkins"],["dc.publisher.place","Philadelphia"],["dc.relation.conference","80th Annual Scientific Session of the American-Heart-Association"],["dc.relation.eventlocation","Orlando, FL"],["dc.relation.issn","0009-7322"],["dc.title","Effects of obesity and weight loss on the functional properties of endothelial progenitor cells"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","79"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Clinical Research in Cardiology"],["dc.bibliographiccitation.lastpage","81"],["dc.bibliographiccitation.volume","103"],["dc.contributor.author","Schroeter, M. R."],["dc.contributor.author","Koziolek, Michael"],["dc.date.accessioned","2018-11-07T09:46:41Z"],["dc.date.available","2018-11-07T09:46:41Z"],["dc.date.issued","2014"],["dc.identifier.doi","10.1007/s00392-013-0634-6"],["dc.identifier.isi","000330809000011"],["dc.identifier.pmid","24249314"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/34939"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.publisher.place","Heidelberg"],["dc.relation.issn","1861-0692"],["dc.relation.issn","1861-0684"],["dc.title","Early reduction of therapy-resistant hypertension in a patient after single-sided renal denervation approach"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.subtype","letter_note"],["dspace.entity.type","Publication"]]