Sander, C.

[["dc.bibliographiccitation.firstpage","39"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Wood Science and Technology"],["dc.bibliographiccitation.lastpage","46"],["dc.bibliographiccitation.volume","37"],["dc.contributor.author","Sander, C."],["dc.contributor.author","Beckers, EPJ"],["dc.contributor.author","Militz, Holger"],["dc.contributor.author","van Veenendaal, W."],["dc.date.accessioned","2018-11-07T10:38:51Z"],["dc.date.available","2018-11-07T10:38:51Z"],["dc.date.issued","2003"],["dc.description.abstract","The properties of acetylated solid wood were investigated earlier, in particular the anti-shrink efficiency and the resistance against decay. This study focuses on the possible changes and damage to the wood structure due to an acetylation process leading to weight per cent gains of up to 20%. Electron microscopy (SEM and TEM) was used to investigate the fine structure of acetylated beech, pine and spruce. Cell wall swelling was observed, but no evidence of damage could be seen as a result of the acetylation procedure. The fine structure of the wood tissue such as the pits and the thin parenchyma walls appeared untouched."],["dc.identifier.doi","10.1007/s00226-002-0160-6"],["dc.identifier.isi","000183774500004"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/45908"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.relation.issn","0043-7719"],["dc.title","Analysis of acetylated wood by electron microscopy"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","610"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","Cancer"],["dc.bibliographiccitation.lastpage","627"],["dc.bibliographiccitation.volume","97"],["dc.contributor.author","Santucci, M."],["dc.contributor.author","Pimpinelli, N."],["dc.contributor.author","Massi, D."],["dc.contributor.author","Kadin, Marshall E."],["dc.contributor.author","Meijer, CJLM"],["dc.contributor.author","Muller-Hermelink, H. K."],["dc.contributor.author","Paulli, M."],["dc.contributor.author","Wechsler, J."],["dc.contributor.author","Willemze, R."],["dc.contributor.author","Audring, H."],["dc.contributor.author","Bernengo, M. G."],["dc.contributor.author","Cerroni, L."],["dc.contributor.author","Chimenti, S."],["dc.contributor.author","Chott, A."],["dc.contributor.author","Diaz-Perez, J. L."],["dc.contributor.author","Dippel, Edgar"],["dc.contributor.author","Duncan, L. M."],["dc.contributor.author","Feller, A. C."],["dc.contributor.author","Geerts, M. L."],["dc.contributor.author","Hallermann, Christian"],["dc.contributor.author","Kempf, Werner"],["dc.contributor.author","Russell-Jones, R."],["dc.contributor.author","Sander, C."],["dc.contributor.author","Berti, E."],["dc.date.accessioned","2018-11-07T10:41:07Z"],["dc.date.available","2018-11-07T10:41:07Z"],["dc.date.issued","2003"],["dc.description.abstract","BACKGROUND. Cutaneous lymphomas expressing a cytotoxic or natural killer (NK) cell phenotype represent a group of lymphoproliferative disorders for which there is currently much confusion and little consensus regarding the best nomenclature and classification. METHODS. This study analyzes 48 cases of primary cutaneous lymphoma expressing cytotoxic proteins and/or the NK cell marker, CD56. These cases were collected for a workshop of the European Organization for Research and Treatment of Cancer Cutaneous Lymphoma Task Force, to better clarify the clinical, morphologic, and phenotypic features of these uncommon tumors. RESULTS. Several categories with different clinical and pathologic features were delineated: 1) aggressive, CD8+, epidermotropic, cytotoxic T-cell lymphoma; 2) mycosis fungoides, cytotoxic immunophenotype variant; 3) subcutaneous panniculitis-like T-cell lymphoma; 4) NK/T-cell lymphoma, nasal type; 5) CD4+, NK cell lymphoma; 6) blastoid NK cell lymphoma; (7) intravascular NK-like lymphoma; and 8) cytotoxic, peripheral T-cell lymphoma. CONCLUSIONS. Our data show that primary cutaneous cytotoxic/NK cell lymphomas include distinct groups of diseases, clinically, histologically, and biologically. Because the finding of a cytotoxic phenotype often has prognostic significance, the routine use of cytotoxic markers in the diagnosis and classification of cutaneous lymphomas should be expanded. Cancer 2003;97:610-27. (C) 2003 American Cancer Society."],["dc.identifier.doi","10.1002/cncr.11107"],["dc.identifier.isi","000180536400011"],["dc.identifier.pmid","12548603"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/46463"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","John Wiley & Sons Inc"],["dc.relation.issn","0008-543X"],["dc.title","Cytotoxic/natural killer cell cutaneous lymphomas - Report of the EORTC Cutaneous Lymphoma Task Force Workshop"],["dc.type","review"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1121"],["dc.bibliographiccitation.issue","5"],["dc.bibliographiccitation.journal","Clinical Oral Investigations"],["dc.bibliographiccitation.lastpage","1128"],["dc.bibliographiccitation.volume","19"],["dc.contributor.author","Böhrnsen, F."],["dc.contributor.author","Fricke, M."],["dc.contributor.author","Sander, C."],["dc.contributor.author","Leha, A."],["dc.contributor.author","Schliephake, H."],["dc.contributor.author","Kramer, F. J."],["dc.date.accessioned","2018-06-26T08:09:06Z"],["dc.date.available","2018-06-26T08:09:06Z"],["dc.date.issued","2015"],["dc.description.abstract","Cancer progression is influenced by tumor microenvironment and communication of stromal cells and tumor cells. Interactions may enhance epithelial-mesenchymal transition (EMT) of tumor cells through signaling proteins such as Wnt/beta-catenin and matrix metalloproteinases (MMP), as well as loss of cellular integrity, which affects invasion, progression, and metastasis of head and neck squamous cell carcinoma (HNSCC). In this study, we are testing the hypothesis that interactions of human mesenchymal stromal cells (MSCs) with HNSCC might influence the expression of markers of EMT and tumor progression by co-culturing human MSC with the PCI-13 HNSCC line."],["dc.identifier.doi","10.1007/s00784-014-1338-7"],["dc.identifier.pmid","25346374"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/15148"],["dc.language.iso","en"],["dc.notes.status","final"],["dc.relation.eissn","1436-3771"],["dc.title","Interactions of human MSC with head and neck squamous cell carcinoma cell line PCI-13 reduce markers of epithelia-mesenchymal transition"],["dc.type","journal_article"],["dc.type.internalPublication","unknown"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1591"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","The Journal of Lipid Research"],["dc.bibliographiccitation.lastpage","1598"],["dc.bibliographiccitation.volume","51"],["dc.contributor.author","Janzen, N."],["dc.contributor.author","Sander, S."],["dc.contributor.author","Terhardt, M."],["dc.contributor.author","Das, Anibh M."],["dc.contributor.author","Sass, Joern Oliver"],["dc.contributor.author","Kraetzner, Ralph"],["dc.contributor.author","Rosevich, H."],["dc.contributor.author","Peter, M."],["dc.contributor.author","Sander, Josemir W."],["dc.date.accessioned","2018-11-07T08:42:50Z"],["dc.date.available","2018-11-07T08:42:50Z"],["dc.date.issued","2010"],["dc.description.abstract","The aim of the study was to develop a method for fast and reliable diagnosis of peroxisomal diseases and to facilitate differential diagnosis of cholestatic hepatopathy. For the quantification of bile acids and their conjugates as well as C-27 precursors di- and trihydroxycholestanoic acid (DHCA, THCA), in small pediatric blood samples we combined HPLC separation on a reverse-phase C18 column with ESI-MS/MS analysis in the negative ion mode. Analysis was done with good precision (CV 3,7%-11.1%) and sufficient sensitivity (LOQ: 11-91 nmol/L) without derivatization. Complete analysis of 17 free and conjugated bile acids from dried blood spots and 10 mu L serum samples, respectively, was performed within 12 min. Measurement of conjugated primary bile acids plus DHCA and THCA as well as ursodeoxycholic acid was done in 4.5 min. In blood spots of healthy newborns, conjugated primary bile acids were found in the range of 0.01 to 2.01 mu mol/L. Concentrations of C-27 precursors were below the detection limit in normal controls. DHCA and THCA were specifically elevated in cases of peroxysomal defects and one Zellweger patient.-Janzen, N., S. Sander, M. Terhardt, A. M. Das, J. O. Sass, R. Kraetzner, H. Rosevich, M. Peter, and J. Sander. Rapid quantification of conjugated and unconjugated bile acids and C27 precursors in dried blood spots and small volumes of serum. J. Lipid Res. 2010. 51: 1591-1598."],["dc.identifier.doi","10.1194/jlr.D003814"],["dc.identifier.isi","000277564700035"],["dc.identifier.pmid","20093478"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/19796"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Amer Soc Biochemistry Molecular Biology Inc"],["dc.relation.issn","0022-2275"],["dc.title","Rapid quantification of conjugated and unconjugated bile acids and C-27 precursors in dried blood spots and small volumes of serum"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]