Dev, Arvind A.

[["dc.bibliographiccitation.firstpage","2329"],["dc.bibliographiccitation.issue","10"],["dc.bibliographiccitation.journal","physica status solidi (b)"],["dc.bibliographiccitation.lastpage","2337"],["dc.bibliographiccitation.volume","247"],["dc.contributor.author","Ronning, Carsten"],["dc.contributor.author","Borschel, Christian"],["dc.contributor.author","Geburt, Sebastian"],["dc.contributor.author","Niepelt, R."],["dc.contributor.author","Mueller, S."],["dc.contributor.author","Stichtenoth, Daniel"],["dc.contributor.author","Richters, J.-P."],["dc.contributor.author","Dev, A."],["dc.contributor.author","Voss, Tobias"],["dc.contributor.author","Chen, L."],["dc.contributor.author","Heimbrodt, W."],["dc.contributor.author","Gutsche, C."],["dc.contributor.author","Prost, Werner"],["dc.date.accessioned","2018-11-07T08:38:54Z"],["dc.date.available","2018-11-07T08:38:54Z"],["dc.date.issued","2010"],["dc.description.abstract","This review demonstrates that ion irradiation is a very useful tool in order to tailor the properties of semiconductor nanowires. Besides optical and electrical doping provided by adequate ion species and ion energies, one can use ion beams also for the controlled shaping of the morphology of nanostructures. Here, one utilizes the commonly as 'negative' described characteristics of ion implantation: defect formation and sputtering. We show that ion beams can be even used for an alignment of the nanowires. Furthermore, we report here on several successful experiments in order to modify the electrical and optical properties in a controlled manner of ZnO semiconductor nanowires by the use of transition metals, rare earth elements and hydrogen ions. [GRAPHICS] Schematic illustration of ion beam doping of a single contacted nanowire. (C) 2010 WILEY-VCH Verlag GmbH & Co. KGaA, Weinheim"],["dc.identifier.doi","10.1002/pssb.201046192"],["dc.identifier.isi","000283673800004"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/18867"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Wiley-blackwell"],["dc.relation.issn","0370-1972"],["dc.title","Tailoring the properties of semiconductor nanowires using ion beams"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","125"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Developmental Cell"],["dc.bibliographiccitation.lastpage","132"],["dc.bibliographiccitation.volume","11"],["dc.contributor.author","Nayernia, Karim"],["dc.contributor.author","Nolte, Jessica"],["dc.contributor.author","Michelmann, Hans Wilhelm"],["dc.contributor.author","Lee, Jae Ho"],["dc.contributor.author","Rathsack, Kristina"],["dc.contributor.author","Drusenheimer, Nadja"],["dc.contributor.author","Dev, Arvind"],["dc.contributor.author","Wulf, Gerald"],["dc.contributor.author","Ehrmann, Ingrid E."],["dc.contributor.author","Elliott, David J."],["dc.contributor.author","Okpanyi, Vera"],["dc.contributor.author","Zechner, Ulrich"],["dc.contributor.author","Haaf, Thomas"],["dc.contributor.author","Meinhardt, Andreas"],["dc.contributor.author","Engel, Wolfgang"],["dc.date.accessioned","2018-11-07T09:35:07Z"],["dc.date.available","2018-11-07T09:35:07Z"],["dc.date.issued","2006"],["dc.description.abstract","Male gametes originate from a small population of spermatogonial stem cells (SSCs). These cells are believed to divide infinitely and to support spermatogenesis throughout life in the male. Here, we developed a strategy for the establishment of SSC lines from embryonic stem (ES) cells. These cells are able to undergo meiosis, are able to generate haploid male gametes in vitro, and are functional, as shown by fertilization after intracytoplasmic injection into mouse oocytes. Resulting two-cell embryos were transferred into oviducts, and live mice were born. Six of seven animals developed to adult mice. This is a clear indication that male gametes derived in vitro from ES cells by this strategy are able to induce normal fertilization and development. Our approach provides an accessible in vitro model system for studies of mammalian gametogenesis, as well as for the development of new strategies for the generation of transgenic mice and treatment of infertility."],["dc.identifier.doi","10.1016/j.devcel.2006.05.010"],["dc.identifier.isi","000239128300015"],["dc.identifier.pmid","16824959"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/32324"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Cell Press"],["dc.relation.issn","1534-5807"],["dc.title","In vitro-differentiated embryonic stem cells give rise to male gametes that can generate offspring mice"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","155"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","MHR Basic science of reproductive medicine"],["dc.bibliographiccitation.lastpage","163"],["dc.bibliographiccitation.volume","13"],["dc.contributor.author","Jaroszynski, L."],["dc.contributor.author","Dev, A."],["dc.contributor.author","Li, M."],["dc.contributor.author","Meinhardt, Andreas"],["dc.contributor.author","de Rooij, D. G."],["dc.contributor.author","Mueller, Christian"],["dc.contributor.author","Boehm, Detlef"],["dc.contributor.author","Wolf, S."],["dc.contributor.author","Adham, Ibrahim M."],["dc.contributor.author","Wulf, Gerald"],["dc.contributor.author","Engel, Wolfgang"],["dc.contributor.author","Nayernia, K."],["dc.date.accessioned","2018-11-07T11:04:40Z"],["dc.date.available","2018-11-07T11:04:40Z"],["dc.date.issued","2007"],["dc.description.abstract","Testis expressed gene 18 (Tex18) is a small gene with one exon of 240 bp, which is specifically expressed in male germ cells. The gene encodes for a protein of 80 amino acids with unknown domain. To investigate the function of (Tex18) gene, we generated mice with targeted disruption of the (Tex18) gene by homologous recombination. Homozygous mutant males on a mixed genetic background (C57BL/6J x 129/Sv) are fertile, while they are subfertile on the 129/Sv background, although mating is normal. We showed that Tex18(-/-) males are subfertile because of abnormal sperm morphology and reduced motility, which is called asthenoteratozoospermia, suggesting that (Tex18) affects sperm characteristics. Maturation of spermatids is unsynchronized and partially impaired in seminiferous tubules of Tex18(-/-) mice. Electron microscopical examination demonstrated abnormal structures of sperm head. In vivo experiments with sperm of Tex18(-/-) 129/Sv mice revealed that the migration of spermatozoa from the uterus into the oviduct is reduced. This result is supported by the observation that sperm motility, as determined by the computer-assisted semen analysis system, is significantly affected, compared to wild-type spermatozoa. Generation of transgenic mice containing Tex18-EGFP fusion construct revealed a high transcriptional activity of (Tex18) during spermiogenesis, a process with morphological changes of haploid germ cells and development to mature spermatozoa. These results indicate that (Tex18) is expressed predominantly during spermatid differentiation and subfertility of the male Tex18(-/-) mice on the 129/Sv background is due to the differentiation arrest, abnormal sperm morphology and reduced sperm motility."],["dc.identifier.doi","10.1093/molehr/gal107"],["dc.identifier.isi","000244662300003"],["dc.identifier.pmid","17208930"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/51894"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Oxford Univ Press"],["dc.relation.issn","1360-9947"],["dc.title","Asthenoteratozoospermia in mice lacking testis expressed gene 18 (Tex18)"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","432"],["dc.bibliographiccitation.issue","29-30"],["dc.bibliographiccitation.journal","Swiss Medical Weekly"],["dc.bibliographiccitation.lastpage","436"],["dc.bibliographiccitation.volume","138"],["dc.contributor.author","Argyriou, Loukas"],["dc.contributor.author","Wirbelauer, Johannes"],["dc.contributor.author","Dev, Arvind"],["dc.contributor.author","Panchulidze, Irakli"],["dc.contributor.author","Shoukier, Moneef"],["dc.contributor.author","Teske, Ute"],["dc.contributor.author","Nayernia, Karim"],["dc.date.accessioned","2018-11-07T11:12:53Z"],["dc.date.available","2018-11-07T11:12:53Z"],["dc.date.issued","2008"],["dc.description.abstract","Hereditary haemorrhagic telangiectasia (HHT), associated with arteriovenous malformations, is a genetic disease of the vascular system with a frequency of approx. 1:10,000. Genetic diagnosis serves to identify individuals at risk of developing the disease and is a useful tool for genetic counselling purposes. Questions under study: Here we report on a child presenting severe arteriovenous malformations leading to heart failure. Her mother and grandmother present fewer symptoms of hereditary haemorrhagic telangiectasia. In this study we identify the cause of HHT in the family. Methods: Clinical examination, PCR, DNA sequencing, quantitative PCR, Southern blot, x-ray, ultrasound, cardiac catheterisation and angiocardiography. Results: Initially the sequence variant in c.392C > T in the endoglin gene was detected in the grandmother, but not in other affected family members. Further analyses revealed a deletion of exon 1 of endoglin, segregating with the phenotype. Conclusions: This report points out the need for careful evaluation of molecular genetic findings, particularly in diseases with highly variable phenotype."],["dc.identifier.isi","000257973200004"],["dc.identifier.pmid","18654869"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/53767"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","E M H Swiss Medical Publishers Ltd"],["dc.relation.issn","1424-7860"],["dc.title","A newborn with hereditary haemorrhagic telangiectasia and an unusually severe phenotype"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","166"],["dc.bibliographiccitation.issue","5"],["dc.bibliographiccitation.journal","physica status solidi (RRL) - Rapid Research Letters"],["dc.bibliographiccitation.lastpage","168"],["dc.bibliographiccitation.volume","3"],["dc.contributor.author","Richters, J.-P."],["dc.contributor.author","Dev, A."],["dc.contributor.author","Mueller, S."],["dc.contributor.author","Niepelt, R."],["dc.contributor.author","Borschel, Christian"],["dc.contributor.author","Ronning, Carsten"],["dc.contributor.author","Voss, Tobias"],["dc.date.accessioned","2018-11-07T08:28:30Z"],["dc.date.available","2018-11-07T08:28:30Z"],["dc.date.issued","2009"],["dc.description.abstract","We report on low-temperature photoluminescence studies of ZnO nanowires coated with thin metallic films. For all analyzed metals (Al, In, Au, Ni, Cu), we find an increased relative intensity of the green deep-level emission. This is accompanied by a significant reduction of the relative intensity of the surface exciton band. The observed effects are most likely related to the formation of metal induced gap states in the surface region of the ZnO nanowires. A model for the band structure in the surface region of the metal-coated nanowires is proposed that successfully explains the changes in the photoluminescence spectra after the coating process. (C) 2009 WILEY-VCH Verlag GmbH & Co. KGaA, Weinheim"],["dc.description.sponsorship","German Research Foundation (DFG) [Vo1265/4-2, Ro 1198/7-3]"],["dc.identifier.doi","10.1002/pssr.200903176"],["dc.identifier.isi","000268708800020"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/16437"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Wiley-v C H Verlag Gmbh"],["dc.relation.issn","1862-6254"],["dc.title","Influence of metallic coatings on the photoluminescence properties of ZnO nanowires"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1456"],["dc.bibliographiccitation.issue","11"],["dc.bibliographiccitation.journal","Molecular Reproduction and Development"],["dc.bibliographiccitation.lastpage","1464"],["dc.bibliographiccitation.volume","74"],["dc.contributor.author","Dev, Arvind"],["dc.contributor.author","Nayernia, Karim"],["dc.contributor.author","Meins, Moritz"],["dc.contributor.author","Adham, Ibrahim M."],["dc.contributor.author","Lacone, Franco"],["dc.contributor.author","Engel, Wolfgang"],["dc.date.accessioned","2018-11-07T10:57:16Z"],["dc.date.available","2018-11-07T10:57:16Z"],["dc.date.issued","2007"],["dc.description.abstract","RNA-binding proteins are involved in post-transcriptional processes like mRNA stabilization, alternative splicing, and transport. Brunol1 is a novel mouse gene related to elav/Bruno family of genes encoding for RNA-binding proteins. We report here the expression and functional analysis of murine Brunol1. Expression analysis of Brunol1 during embryogenesis by RT-PCR showed that Brunol1 expression starts at 9.5 dpc and continues to the later stages of embryonic development. In adult mice, the Brunol1 expression is restricted to brain and testis. We also analyzed the Brunol1 expression in testes of different mutants with spermatogenesis defects: W/W-V, Tfm/y, Leyl(-/-), olt/olt, and qk/qk. Brunol1 transcript was detectable in Leyl-/-, olt/olt, and qk/qk mutant but not in W/W-V and Tfm/y mutants. We also showed by transfection of a fusion protein of green fluorescent protein and Brunol1 protein into NIH3T3 cells, that Brunol1 is localized in cytoplasm and nucleus. In order to elucidate the function of the Brunol1 protein in spermatogenesis, we disrupted the Brunol1 locus in mouse by homologous recombination, which resulted in a complete loss of the Brunol1 transcript. Male and female Brunol1(-/-) and Brunol1(-/-) mice from genetic backgrounds C57BU6J x 129/Sv hybrid and 129X1/SvJ when inbred exhibited normal phenotype and are fertile, although the number and motility of sperms are significantly reduced. An intensive phenotypic analysis showed no gross abnormalities in testis morphology. Collectively our results demonstrate that Brunol1 might be nonessential protein for mouse embryonic development and sperm atogenesis."],["dc.identifier.doi","10.1002/mrd.20742"],["dc.identifier.isi","000249677800012"],["dc.identifier.pmid","17393433"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/50201"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Wiley-liss"],["dc.relation.issn","1040-452X"],["dc.title","Mice deficient for RNA-Binding protein brunoll show reduction of sperniatogenesis but are fertile"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","2760"],["dc.bibliographiccitation.issue","22"],["dc.bibliographiccitation.journal","Human Molecular Genetics"],["dc.bibliographiccitation.lastpage","2769"],["dc.bibliographiccitation.volume","16"],["dc.contributor.author","Chapple, J. Paul"],["dc.contributor.author","Anthony, Karen"],["dc.contributor.author","Martin, Teresa Rodriguez"],["dc.contributor.author","Dev, Arvind"],["dc.contributor.author","Cooper, Thomas A."],["dc.contributor.author","Gallo, Jean-Marc"],["dc.date.accessioned","2018-11-07T10:53:31Z"],["dc.date.available","2018-11-07T10:53:31Z"],["dc.date.issued","2007"],["dc.description.abstract","Elucidating the mechanisms of alternative splicing in the brain is a prerequisite to the understanding of the pathogenesis of major neurological diseases linked to impairment of pre-mRNA alternative splicing. The gene trinucleotide repeat-containing 4 (TNRC4) is predicted to encode a member of the CELF (CUG-BP-and ETR-3-like factors) family of RNA-binding proteins containing a 15 - 18-residue polyglutamine sequence. The TNRC4 transcript is selectively expressed in the brain. Using an anti-peptide antibody against the predicted sequence, we establish the presence of TNRC4 as a similar to 50 kDa protein in the brain. Full-length TNRC4 displays nuclear and cytoplasmic localizations in transfected cells, whereas a C-terminally truncated mutant is essentially confined to the cytoplasm. TNRC4 is not recruited into inclusions formed by polyglutamine-expanded ataxin-1 or huntingtin. TNRC4 activates tau exon 10 (E10) inclusion at high efficiency in transfected cells. TNRC4 contains two consecutive N-terminal RNA recognition motifs (RRMs) separated from the C-terminal RRM. Deletion and point mutant analysis show that the activity of TNRC4 on tau E10 splicing is mainly mediated by the RNA-binding activity of the second RRM and involves an intronic element of the tau pre-mRNA. The polyglutamine sequence has no effect on the activity of TNRC4 on tau E10 splicing. This study represents the first characterization of TNRC4 and provides further insight into the mechanisms of brain-specific alternative splicing and their possible pathological implications."],["dc.description.sponsorship","NIAMS NIH HHS [R01AR45653]"],["dc.identifier.doi","10.1093/hmg/ddm233"],["dc.identifier.isi","000250679400012"],["dc.identifier.pmid","17725984"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/49367"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Oxford Univ Press"],["dc.relation.issn","0964-6906"],["dc.title","Expression, localization and tau exon 10 splicing activity of the brain RNA-binding protein TNRC4"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","273"],["dc.bibliographiccitation.issue","3"],["dc.bibliographiccitation.journal","Molecular Reproduction and Development"],["dc.bibliographiccitation.lastpage","279"],["dc.bibliographiccitation.volume","74"],["dc.contributor.author","Tseden, Khailun"],["dc.contributor.author","Topaloglu, Ozlem"],["dc.contributor.author","Meinhardt, Andreas"],["dc.contributor.author","Dev, Arvind"],["dc.contributor.author","Adham, Ibrahim M."],["dc.contributor.author","Mueller, Christian"],["dc.contributor.author","Wolf, Stephan"],["dc.contributor.author","Boehm, Detlef"],["dc.contributor.author","Schlueter, Gregor"],["dc.contributor.author","Engel, Wolfgang"],["dc.contributor.author","Nayernia, Karim"],["dc.date.accessioned","2018-11-07T11:04:41Z"],["dc.date.available","2018-11-07T11:04:41Z"],["dc.date.issued","2007"],["dc.description.abstract","During mammalian spermiogenesis somatic histones are replaced at first by transition proteins, which are in turn replaced by the protamines, forming the sperm nucleoprotamines. It is believed that transition protein 2 (Tnp2) is necessary for maintaining the normal processing of protamines and, consequently, the completion of chromatin condensation. The transition protein mRNAs are stored in translationally inert messenger ribonucleoprotein particles for up to 7 days until translational activation in elongated spermatids. Substantial evidence suggests an involvement of 3'untranslated region (UTR) in the translational regulation of the Tnp2 mRNAs. In order to determine the role of Tnp2 3'UTR in translational regulation and to study whether the translational repression of Tnp2 mRNA is necessary for normal spermatid differentiation in mice, we generated transgenic mice that carry a Tnp2-hGH transgene. In this transgene, 3'UTR of Thp2 gene was replaced by 3' 3'UTR of human growth hormone gene. In these transgenic animals, transcription and translation of Tnp2 occur simultaneously in round spermatids which is an evidence for involvement of Tnp2 3'UTR in its translation repression. Premature translation of Tnp2 mRNA caused abnormal head morphogenesis, reduced sperm motility and male infertility. These results show clearly that a strict temporal and stage-specific Tnp2 translation is necessary for the correct differentiation of round spermatids into mature spermatozoa and for male fertility."],["dc.identifier.doi","10.1002/mrd.20570"],["dc.identifier.isi","000243567900002"],["dc.identifier.pmid","16967499"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/51896"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Wiley-liss"],["dc.relation.issn","1040-452X"],["dc.title","Premature translation of transition protein 2 mRNA causes sperm abnormalities and male infertility"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","41"],["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","IRANIAN JOURNAL OF REPRODUCTIVE MEDICINE"],["dc.bibliographiccitation.lastpage","44"],["dc.bibliographiccitation.volume","5"],["dc.contributor.author","Nayernia, Karim"],["dc.contributor.author","Lee, Jae Ho"],["dc.contributor.author","Engel, Wolfgang"],["dc.contributor.author","Nolte, Jessica"],["dc.contributor.author","Drusenheimer, Nadja"],["dc.contributor.author","Rathsack, Kristina"],["dc.contributor.author","Dev, Arvind"],["dc.contributor.author","Wulf, Gerald"],["dc.contributor.author","Ehrmann, Ingrid E."],["dc.contributor.author","Elliott, David J."],["dc.contributor.author","Zechner, Ulrich"],["dc.contributor.author","Haaf, Thomas"],["dc.contributor.author","Meinhardt, Andreas"],["dc.contributor.author","Michelmann, Hans Wilhelm"],["dc.contributor.author","Hasenfuss, Gerlad"],["dc.contributor.author","Guan, Kaomei"],["dc.date.accessioned","2018-11-07T11:04:31Z"],["dc.date.available","2018-11-07T11:04:31Z"],["dc.date.issued","2007"],["dc.description.abstract","Germline and somatic stem cells are distinct types of stem cells that are dedicated to reproduction and somatic tissue regeneration, respectively. Germline stem cells (GSCs), which can self-renew and generate gametes, are unique stem cells in that they are solely dedicated to transmit genetic information from generation to generation. We developed a strategy for the establishment of germline stem cell lines from embryonic stem cells (ES). These cells are able to undergo meiosis, generate haploid male gametes in vitro and are functional, as shown by fertilization after intra-cytoplasmic injection into mouse oocytes. In other approach, we show that bone marrow stem (BMS) cells are able to trans-differentiate into male germ cells. BMS cell-derived germ cells expressed the known molecular markers of primordial germ cells. The ability to derive male germ cells from ES and BMS cells reveals novel aspects of germ cell development and opens the possibilities for use of these cells in reproductive medicine. Conversely, we showed that adult male germline stem cells, spermatogonial stem cells (SSCs), can be converted into embryonic stem cell like cells which can differentiate into the somatic stem cells of three germ layers. Understanding how SSC can give rise to pluripotent stem cells and how somatic stem cells differentiate into germ cells could give significant insights into the regulation of developmental totipotency as well as having important implications for male fertility and regenerative medicine."],["dc.identifier.isi","000254385400001"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/51863"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.relation.issn","1680-6433"],["dc.title","From stem cells to germ cells and from germ cells to stem cells"],["dc.type","review"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dspace.entity.type","Publication"]]