Gruber, Oliver

[["dc.bibliographiccitation.firstpage","1135"],["dc.bibliographiccitation.issue","5"],["dc.bibliographiccitation.journal","Schizophrenia Bulletin"],["dc.bibliographiccitation.lastpage","1148"],["dc.bibliographiccitation.volume","42"],["dc.contributor.author","Schilbach, Leonhard"],["dc.contributor.author","Derntl, Birgit"],["dc.contributor.author","Aleman, Andre"],["dc.contributor.author","Caspers, Svenja"],["dc.contributor.author","Clos, Mareike"],["dc.contributor.author","Diederen, Kelly M. J."],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Kogler, Lydia"],["dc.contributor.author","Liemburg, Edith J."],["dc.contributor.author","Sommer, Iris E."],["dc.contributor.author","Mueller, Veronika I."],["dc.contributor.author","Cieslik, Edna C."],["dc.contributor.author","Eickhoff, Simon B."],["dc.date.accessioned","2018-11-07T10:09:06Z"],["dc.date.available","2018-11-07T10:09:06Z"],["dc.date.issued","2016"],["dc.description.abstract","Impairments of social cognition are well documented in patients with schizophrenia (SCZ), but the neural basis remains poorly understood. In light of evidence that suggests that the \"mirror neuron system\" (MNS) and the \"mentalizing network\" (MENT) are key substrates of intersubjectivity and joint action, it has been suggested that dysfunction of these neural networks may underlie social difficulties in SCZ patients. Additionally, MNS and MENT might be associated differently with positive vs negative symptoms, given prior social cognitive and symptom associations. We assessed resting state functional connectivity (RSFC) in meta-analytically defined MNS and MENT networks in this patient group. Magnetic resonance imaging (MRI) scans were obtained from 116 patients and 133 age-, gender- and movement-matched healthy controls (HC) at 5 different MRI sites. Network connectivity was analyzed for group differences and correlations with clinical symptoms. Results demonstrated decreased connectivity within the MNS and also the MENT in patients compared to controls. Notably, dysconnectivity of the MNS was related to symptom severity, while no such relationship was observed for the MENT. In sum, these findings demonstrate that differential patterns of dysconnectivity exist in SCZ patients, which may contribute differently to the interpersonal difficulties commonly observed in the disorder."],["dc.identifier.doi","10.1093/schbul/sbw015"],["dc.identifier.isi","000386209900010"],["dc.identifier.pmid","26940699"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/39594"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Oxford Univ Press"],["dc.relation.issn","1745-1701"],["dc.relation.issn","0586-7614"],["dc.title","Differential Patterns of Dysconnectivity in Mirror Neuron and Mentalizing Networks in Schizophrenia"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","189"],["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","Cortex"],["dc.bibliographiccitation.lastpage","200"],["dc.bibliographiccitation.volume","45"],["dc.contributor.author","Melcher, Tobias"],["dc.contributor.author","Gruber, Oliver"],["dc.date.accessioned","2018-11-07T08:32:57Z"],["dc.date.available","2018-11-07T08:32:57Z"],["dc.date.issued","2009"],["dc.description.abstract","In the current event-related functional magnetic resonance imaging (fMRI) study, we sought to trace back Stroop-interference to circumscribed properties of task-irrelevant word information - response-incompatibility, semantic incongruency and task-reference that we conceive as conflict factors. Thereby, we particularly wanted to disentangle intermingled contributions of semantic conflict and response conflict to the overall Stroop-interference effect. To delineate neural substrates of single factors, we referred to the logics of cognitive subtraction and cognitive conjunction. Moreover, in a second step, we conducted correlation analyses to determine the relationship between neural activations and behavioral interference costs (i.e., conflict-related reaction time (RT) slowing) so as to further elucidate the functional role of the respective brain regions in conflict processing. Response-incompatibility was associated with activation in the left premotor cortex which can be interpreted as indicating motor competition or conflict, i.e., the presence of competing response tendencies. Accordingly, this activation was positively correlated with behavioral conflict costs. Semantic incongruency exhibited specific activation in the anterior cingulate cortex (ACC), the bilateral insula, and thalamus as well as in left somatosensory cortex. As supported by the consistent negative correlation with behavioral conflict costs, these activations most probably reflect strengthened control efforts to overcome interference and to ensure adequate task performance. Finally, task-reference elicited activation in the left temporo-polar cortex (TPC) and the right medial superior as well as in left rostroventral prefrontal cortex (rvPFC, sub-threshold activation). As strongly supported by prior studies' findings, this neural activation pattern may underlie residual semantic processing of the task-irrelevant word information. (c) 2007 Elsevier Srl. All rights reserved."],["dc.description.sponsorship","German Research Foundation (DFG) [1950/1-3]"],["dc.identifier.doi","10.1016/j.cortex.2007.06.004"],["dc.identifier.isi","000263206400007"],["dc.identifier.pmid","19150520"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/17454"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Elsevier Masson"],["dc.relation.issn","0010-9452"],["dc.title","Decomposing interference during Stroop performance into different conflict factors: An event-related fMRI study"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1710"],["dc.bibliographiccitation.issue","12"],["dc.bibliographiccitation.journal","Molecular Psychiatry"],["dc.bibliographiccitation.lastpage","1716"],["dc.bibliographiccitation.volume","21"],["dc.contributor.author","Hibar, D. P."],["dc.contributor.author","Westlye, L. T."],["dc.contributor.author","van Erp, T. G. M."],["dc.contributor.author","Rasmussen, J."],["dc.contributor.author","Leonardo, C. D."],["dc.contributor.author","Faskowitz, J."],["dc.contributor.author","Haukvik, U. K."],["dc.contributor.author","Hartberg, C. B."],["dc.contributor.author","Doan, N. T."],["dc.contributor.author","Agartz, I."],["dc.contributor.author","Dale, A. M."],["dc.contributor.author","Gruber, O."],["dc.contributor.author","Krämer, B."],["dc.contributor.author","Trost, S."],["dc.contributor.author","Liberg, B."],["dc.contributor.author","Abé, C."],["dc.contributor.author","Ekman, C. J."],["dc.contributor.author","Ingvar, M."],["dc.contributor.author","Landén, M."],["dc.contributor.author","Fears, S. C."],["dc.contributor.author","Freimer, N. B."],["dc.contributor.author","Bearden, C. E."],["dc.contributor.author","Sprooten, E."],["dc.contributor.author","Glahn, D. C."],["dc.contributor.author","Pearlson, G. D."],["dc.contributor.author","Emsell, L."],["dc.contributor.author","Kenney, J."],["dc.contributor.author","Scanlon, C."],["dc.contributor.author","McDonald, C."],["dc.contributor.author","Cannon, D. M."],["dc.contributor.author","Almeida, J."],["dc.contributor.author","Versace, A."],["dc.contributor.author","Caseras, X."],["dc.contributor.author","Lawrence, N. S."],["dc.contributor.author","Phillips, M. L."],["dc.contributor.author","Dima, D."],["dc.contributor.author","Delvecchio, G."],["dc.contributor.author","Frangou, S."],["dc.contributor.author","Satterthwaite, T. D."],["dc.contributor.author","Wolf, D."],["dc.contributor.author","Houenou, J."],["dc.contributor.author","Henry, C"],["dc.contributor.author","Malt, U. F."],["dc.contributor.author","Bøen, E."],["dc.contributor.author","Elvsåshagen, T."],["dc.contributor.author","Young, A. H."],["dc.contributor.author","Lloyd, A. J."],["dc.contributor.author","Goodwin, G. M."],["dc.contributor.author","Mackay, C. E."],["dc.contributor.author","Bourne, C."],["dc.contributor.author","Bilderbeck, A."],["dc.contributor.author","Abramovic, L."],["dc.contributor.author","Boks, M. P."],["dc.contributor.author","van Haren, N. E. M."],["dc.contributor.author","Ophoff, R. A."],["dc.contributor.author","Kahn, R. S."],["dc.contributor.author","Bauer, M."],["dc.contributor.author","Pfennig, A."],["dc.contributor.author","Alda, M."],["dc.contributor.author","Hajek, T."],["dc.contributor.author","Mwangi, B."],["dc.contributor.author","Soares, J. C."],["dc.contributor.author","Nickson, T."],["dc.contributor.author","Dimitrova, R."],["dc.contributor.author","Sussmann, J. E."],["dc.contributor.author","Hagenaars, S."],["dc.contributor.author","Whalley, H. C."],["dc.contributor.author","McIntosh, A. M."],["dc.contributor.author","Thompson, P. M."],["dc.contributor.author","Andreassen, O. A."],["dc.date.accessioned","2019-07-09T11:42:55Z"],["dc.date.available","2019-07-09T11:42:55Z"],["dc.date.issued","2016"],["dc.description.abstract","Considerable uncertainty exists about the defining brain changes associated with bipolar disorder (BD). Understanding and quantifying the sources of uncertainty can help generate novel clinical hypotheses about etiology and assist in the development of biomarkers for indexing disease progression and prognosis. Here we were interested in quantifying case–control differences in intracranial volume (ICV) and each of eight subcortical brain measures: nucleus accumbens, amygdala, caudate, hippocampus, globus pallidus, putamen, thalamus, lateral ventricles. In a large study of 1710 BD patients and 2594 healthy controls, we found consistent volumetric reductions in BD patients for mean hippocampus (Cohen’s d= − 0.232; P = 3.50 × 10− 7) and thalamus (d= − 0.148; P = 4.27 × 10− 3) and enlarged lateral ventricles (d= − 0.260; P = 3.93 × 10− 5) in patients. No significant effect of age at illness onset was detected. Stratifying patients based on clinical subtype (BD type I or type II) revealed that BDI patients had significantly larger lateral ventricles and smaller hippocampus and amygdala than controls. However, when comparing BDI and BDII patients directly, we did not detect any significant differences in brain volume. This likely represents similar etiology between BD subtype classifications. Exploratory analyses revealed significantly larger thalamic volumes in patients taking lithium compared with patients not taking lithium. We detected no significant differences between BDII patients and controls in the largest such comparison to date. Findings in this study should be interpreted with caution and with careful consideration of the limitations inherent to meta-analyzed neuroimaging comparisons."],["dc.identifier.doi","10.1038/mp.2015.227"],["dc.identifier.pmid","26857596"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/13998"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/58785"],["dc.language.iso","en"],["dc.notes.intern","Merged from goescholar"],["dc.relation","info:eu-repo/grantAgreement/EC/FP7/602450/EU/IMAging GEnetics for MENtal Disorders/IMAGEMEND"],["dc.relation.issn","1476-5578"],["dc.rights","CC BY-NC-ND 4.0"],["dc.rights.uri","https://creativecommons.org/licenses/by-nc-nd/4.0"],["dc.title","Subcortical volumetric abnormalities in bipolar disorder"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.issue","5"],["dc.bibliographiccitation.journal","Pharmacopsychiatry"],["dc.bibliographiccitation.volume","40"],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Meyer, J."],["dc.contributor.author","Zilles, David"],["dc.contributor.author","Gruber, Eva"],["dc.contributor.author","Falkai, Peter Gaston"],["dc.date.accessioned","2018-11-07T10:59:21Z"],["dc.date.available","2018-11-07T10:59:21Z"],["dc.date.issued","2007"],["dc.format.extent","221"],["dc.identifier.isi","000249873600085"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/50679"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Georg Thieme Verlag Kg"],["dc.publisher.place","Stuttgart"],["dc.relation.conference","25th Symposium of the Arbeitsgemeinschaft-Neuropsychopharmakologie-und-Pharmakopsychiatrie"],["dc.relation.eventlocation","Munich, GERMANY"],["dc.relation.issn","0176-3679"],["dc.title","Genetic polymorphisms of serotonergic and dopaminergic neurotransmission differentially affect neurofunctional subsystems of working memory"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","547"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","Molecular Psychiatry"],["dc.bibliographiccitation.lastpage","553"],["dc.bibliographiccitation.volume","21"],["dc.contributor.author","van Erp, Theo G. M."],["dc.contributor.author","Hibar, D. P."],["dc.contributor.author","Rasmussen, J. M."],["dc.contributor.author","Glahn, D. C."],["dc.contributor.author","Pearlson, G. D."],["dc.contributor.author","Andreassen, Ole A."],["dc.contributor.author","Agartz, Ingrid"],["dc.contributor.author","Westlye, L. T."],["dc.contributor.author","Haukvik, Unn K."],["dc.contributor.author","Dale, Anders M."],["dc.contributor.author","Melle, Ingrid"],["dc.contributor.author","Hartberg, C. B."],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Kraemer, Bernd"],["dc.contributor.author","Zilles, David"],["dc.contributor.author","Donohoe, Gary"],["dc.contributor.author","Kelly, S."],["dc.contributor.author","McDonald, Colm"],["dc.contributor.author","Morris, D. W."],["dc.contributor.author","Cannon, Dara M."],["dc.contributor.author","Corvin, Aiden"],["dc.contributor.author","Machielsen, M. W. J."],["dc.contributor.author","Koenders, L."],["dc.contributor.author","de Haan, Laurens"],["dc.contributor.author","Veltman, D. J."],["dc.contributor.author","Satterthwaite, T. D."],["dc.contributor.author","Wolf, D. H."],["dc.contributor.author","Gur, Ruben C."],["dc.contributor.author","Gur, R. E."],["dc.contributor.author","Potkin, Steven G."],["dc.contributor.author","Mathalon, D. H."],["dc.contributor.author","Mueller, B. A."],["dc.contributor.author","Preda, A."],["dc.contributor.author","Macciardi, F."],["dc.contributor.author","Ehrlich, S."],["dc.contributor.author","Walton, E."],["dc.contributor.author","Hass, J."],["dc.contributor.author","Calhoun, V. D."],["dc.contributor.author","Bockholt, H. J."],["dc.contributor.author","Sponheim, S. R."],["dc.contributor.author","Shoemaker, J. M."],["dc.contributor.author","van Haren, Neeltje E. M."],["dc.contributor.author","Pol, H. E. H."],["dc.contributor.author","Ophoff, Roel A."],["dc.contributor.author","Kahn, Rene S."],["dc.contributor.author","Roiz-Santianez, R."],["dc.contributor.author","Crespo-Facorro, B."],["dc.contributor.author","Wang, L."],["dc.contributor.author","Alpert, K. I."],["dc.contributor.author","Jonsson, E. G."],["dc.contributor.author","Dimitrova, R."],["dc.contributor.author","Bois, C."],["dc.contributor.author","Whalley, H. C."],["dc.contributor.author","McIntosh, Andrew M."],["dc.contributor.author","Lawrie, Stephen M."],["dc.contributor.author","Hashimoto, Ryota"],["dc.contributor.author","Thompson, Paul M."],["dc.contributor.author","Turner, Jessica A."],["dc.date.accessioned","2018-11-07T10:16:34Z"],["dc.date.available","2018-11-07T10:16:34Z"],["dc.date.issued","2016"],["dc.description.abstract","The profile of brain structural abnormalities in schizophrenia is still not fully understood, despite decades of research using brain scans. To validate a prospective meta-analysis approach to analyzing multicenter neuroimaging data, we analyzed brain MRI scans from 2028 schizophrenia patients and 2540 healthy controls, assessed with standardized methods at 15 centers worldwide. We identified subcortical brain volumes that differentiated patients from controls, and ranked them according to their effect sizes. Compared with healthy controls, patients with schizophrenia had smaller hippocampus (Cohen's d = -0.46), amygdala (d = -0.31), thalamus (d = -0.31), accumbens (d = -0.25) and intracranial volumes (d = -0.12), as well as larger pallidum (d = 0.21) and lateral ventricle volumes (d = 0.37). Putamen and pallidum volume augmentations were positively associated with duration of illness and hippocampal deficits scaled with the proportion of unmedicated patients. Worldwide cooperative analyses of brain imaging data support a profile of subcortical abnormalities in schizophrenia, which is consistent with that based on traditional meta-analytic approaches. This first ENIGMA Schizophrenia Working Group study validates that collaborative data analyses can readily be used across brain phenotypes and disorders and encourages analysis and data sharing efforts to further our understanding of severe mental illness."],["dc.identifier.doi","10.1038/mp.2015.63"],["dc.identifier.isi","000372421500014"],["dc.identifier.pmid","26033243"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/13363"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/41060"],["dc.notes.intern","Merged from goescholar"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.relation.issn","1476-5578"],["dc.relation.issn","1359-4184"],["dc.rights","CC BY-NC-ND 4.0"],["dc.rights.uri","https://creativecommons.org/licenses/by-nc-nd/4.0"],["dc.title","Subcortical brain volume abnormalities in 2028 individuals with schizophrenia and 2540 healthy controls via the ENIGMA consortium"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","1004"],["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","Human Brain Mapping"],["dc.bibliographiccitation.lastpage","1014"],["dc.bibliographiccitation.volume","39"],["dc.contributor.author","Richter, Anja"],["dc.contributor.author","Gruber, Oliver"],["dc.date.accessioned","2020-12-10T14:06:34Z"],["dc.date.available","2020-12-10T14:06:34Z"],["dc.date.issued","2017"],["dc.identifier.doi","10.1002/hbm.v39.2"],["dc.identifier.eissn","1097-0193"],["dc.identifier.issn","1065-9471"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/69947"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.title","Influence of ventral tegmental area input on cortico‐subcortical networks underlying action control and decision making"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","229"],["dc.bibliographiccitation.issue","5"],["dc.bibliographiccitation.journal","Pharmacopsychiatry"],["dc.bibliographiccitation.lastpage","230"],["dc.bibliographiccitation.volume","40"],["dc.contributor.author","Zilles, David"],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Burke, Sarah"],["dc.contributor.author","Falkai, Peter Gaston"],["dc.date.accessioned","2018-11-07T10:59:22Z"],["dc.date.available","2018-11-07T10:59:22Z"],["dc.date.issued","2007"],["dc.identifier.isi","000249873600125"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/50683"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Georg Thieme Verlag Kg"],["dc.publisher.place","Stuttgart"],["dc.relation.conference","25th Symposium of the Arbeitsgemeinschaft-Neuropsychopharmakologie-und-Pharmakopsychiatrie"],["dc.relation.eventlocation","Munich, GERMANY"],["dc.relation.issn","0176-3679"],["dc.title","The influence of genetic loading on working memory performance in schizophrenia"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","287"],["dc.bibliographiccitation.journal","Der Nervenarzt"],["dc.bibliographiccitation.lastpage","288"],["dc.bibliographiccitation.volume","78"],["dc.contributor.author","Gruber, Oliver"],["dc.date.accessioned","2018-11-07T10:57:23Z"],["dc.date.available","2018-11-07T10:57:23Z"],["dc.date.issued","2007"],["dc.identifier.isi","000253318800738"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/50232"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.publisher.place","New york"],["dc.relation.issn","0028-2804"],["dc.title","Physiology and pathophysiology of the work thought system"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","100"],["dc.bibliographiccitation.issue","2"],["dc.bibliographiccitation.journal","European Neuropsychopharmacology"],["dc.bibliographiccitation.lastpage","113"],["dc.bibliographiccitation.volume","22"],["dc.contributor.author","Delvecchio, Giuseppe"],["dc.contributor.author","Fossati, Philippe"],["dc.contributor.author","Boyer, Patrice"],["dc.contributor.author","Brambilla, Paolo"],["dc.contributor.author","Falkai, Peter"],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Hietala, Jarmo"],["dc.contributor.author","Lawrie, Stephen M."],["dc.contributor.author","Martinot, Jean-Luc"],["dc.contributor.author","McIntosh, Andrew M."],["dc.contributor.author","Meisenzahl, Eva"],["dc.contributor.author","Frangou, Sophia"],["dc.date.accessioned","2018-11-07T09:13:58Z"],["dc.date.available","2018-11-07T09:13:58Z"],["dc.date.issued","2012"],["dc.description.abstract","Neuroimaging studies have consistently shown functional brain abnormalities in patients with Bipolar Disorder (BD) and Major Depressive Disorder (MDD). However, the extent to which these two disorders are associated with similar or distinct neural changes remains unclear. We conducted a systematic review of functional magnetic resonance imaging studies comparing BD and MDD patients to healthy participants using facial affect processing paradigms. Relevant spatial coordinates from twenty original studies were subjected to quantitative Activation Likelihood Estimation meta-analyses based on 168 BD and 189 MDD patients and 344 healthy controls. We identified common and distinct patterns of neural engagement for BD and MDD within the facial affect processing network. Both disorders were associated with increased engagement of limbic regions. Diagnosis-specific differences were observed in cortical, thalamic and striatal regions. Decreased ventrolateral prefrontal cortical engagement was associated with BD while relative hypoactivation of the sensorimotor cortices was seen in MDD. Increased responsiveness in the thalamus and basal ganglia were associated with BD. These findings were modulated by stimulus valence. These data suggest that whereas limbic overactivation is reported consistently in patients with mood disorders, future research should consider the relevance of a wider network of regions in formulating conceptual models of BD and MDD. (c) 2011 Elsevier B.V. and ECNP. All rights reserved."],["dc.description.sponsorship","ECNP networks Initiative"],["dc.identifier.doi","10.1016/j.euroneuro.2011.07.003"],["dc.identifier.isi","000299720700003"],["dc.identifier.pmid","21820878"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/27291"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Elsevier Science Bv"],["dc.relation.issn","0924-977X"],["dc.title","Common and distinct neural correlates of emotional processing in Bipolar Disorder and Major Depressive Disorder: A voxel-based meta-analysis of functional magnetic resonance imaging studies"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.journal","The International Journal of Neuropsychopharmacology"],["dc.bibliographiccitation.volume","11"],["dc.contributor.author","Gruber, Oliver"],["dc.contributor.author","Burkhardt, S."],["dc.contributor.author","Gruber, Eva"],["dc.contributor.author","Falkai, Peter Gaston"],["dc.date.accessioned","2018-11-07T11:13:33Z"],["dc.date.available","2018-11-07T11:13:33Z"],["dc.date.issued","2008"],["dc.format.extent","271"],["dc.identifier.isi","000258855501400"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/53922"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Cambridge Univ Press"],["dc.publisher.place","New york"],["dc.relation.conference","26th Collegium Internationale Neuro-Psychopharmacologicum Congress (CINP)"],["dc.relation.eventlocation","Munich, GERMANY"],["dc.relation.issn","1461-1457"],["dc.title","Common and disease-specific dysfunctions of brain systems underlying attentional and executive control in schizophrenia and bipolar disorder"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]