Schulze, Thomas Gerd

[["dc.bibliographiccitation.artnumber","e165"],["dc.bibliographiccitation.journal","Translational Psychiatry"],["dc.bibliographiccitation.volume","2"],["dc.contributor.author","Meier, Sandra"],["dc.contributor.author","Mattheisen, Manuel"],["dc.contributor.author","Vassos, Evangelos"],["dc.contributor.author","Strohmaier, Jana"],["dc.contributor.author","Treutlein, Jens"],["dc.contributor.author","Josef, F."],["dc.contributor.author","Breuer, Rene"],["dc.contributor.author","Degenhardt, Franziska A."],["dc.contributor.author","Muehleisen, Thomas W."],["dc.contributor.author","Mueller-Myhsok, Bertram"],["dc.contributor.author","Steffens, Michael"],["dc.contributor.author","Schmael, C."],["dc.contributor.author","McMahon, Francis J."],["dc.contributor.author","Noethen, M. M."],["dc.contributor.author","Cichon, Sven"],["dc.contributor.author","Schulze, Thomas G."],["dc.contributor.author","Rietschel, Marcella"],["dc.date.accessioned","2018-11-07T09:06:00Z"],["dc.date.available","2018-11-07T09:06:00Z"],["dc.date.issued","2012"],["dc.description.abstract","Research suggests that clinical symptom dimensions may be more useful in delineating the genetics of bipolar disorder (BD) than standard diagnostic models. To date, no study has applied this concept to data from genome-wide association studies (GWAS). We performed a GWAS of factor dimensions in 927 clinically well-characterized BD patients of German ancestry. Rs9875793, which is located in an intergenic region of 3q26.1 and in the vicinity of the solute carrier family 2 (facilitated glucose transporter), member 2 gene (SLC2A2), was significantly associated with the factor analysis-derived dimension 'negative mood delusions' (n = 927; P = 4.65 x 10(-8), odds ratio (OR) = 2.66). This dimension was comprised of the symptoms delusions of poverty, delusions of guilt and nihilistic delusions. In case-control analyses, significant association with the G allele of rs9875793 was only observed in the subgroup of BD patients who displayed symptoms of 'negative mood delusions' (allelic chi(2) model: P-G = 0.0001, OR = 1.92; item present, n = 89). Further support for the hypothesis that rs9875793 is associated with BD in patients displaying 'negative mood delusions' symptom, such as delusions of guilt, was obtained from an European American sample (GAIN/TGEN), which included 1247 BD patients and 1434 controls (P-EA = 0.028, OR = 1.27). Translational Psychiatry (2012) 2, e165; doi:10.1038/tp.2012.81; published online 25 September 2012"],["dc.identifier.doi","10.1038/tp.2012.81"],["dc.identifier.isi","000312900000008"],["dc.identifier.pmid","23010768"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/25455"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Nature Publishing Group"],["dc.relation.issn","2158-3188"],["dc.title","Genome-wide significant association between a 'negative mood delusions' dimension in bipolar disorder and genetic variation on chromosome 3q26.1"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","384"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","International Archives of Occupational and Environmental Health"],["dc.bibliographiccitation.lastpage","388"],["dc.bibliographiccitation.volume","73"],["dc.contributor.author","Westphal, Goetz Alexander"],["dc.contributor.author","Schnuch, Axel"],["dc.contributor.author","Schulz, Thomas G."],["dc.contributor.author","Reich, Kristian"],["dc.contributor.author","Aberer, Werner"],["dc.contributor.author","Brasch, Jochen"],["dc.contributor.author","Koch, P."],["dc.contributor.author","Wessbecher, R."],["dc.contributor.author","Szliska, Christiane"],["dc.contributor.author","Bauer, A."],["dc.contributor.author","Hallier, Ernst"],["dc.date.accessioned","2018-11-07T10:37:14Z"],["dc.date.available","2018-11-07T10:37:14Z"],["dc.date.issued","2000"],["dc.description.abstract","Objective: Thimerosal is an important preservative in vaccines and ophthalmologic preparations. The substance is known to be a type IV sensitizing agent. High sensitization rates were observed in contact-allergic patients and in health care workers who had been exposed to thimerosal-preserved vaccines. There is evidence for the involvement of the glutathione system in the metabolism of thimerosal or its decomposition products (organomercury alkyl compounds). Thus detoxification by polymorphically expressed glutathione S-transferases such as GSTT1 and GSTM1 might have a protective effect against sensitization by these substances. Methods: To address this question, a case control study was conducted, including 91 Central European individuals with a positive patch-test reaction to thimerosal. This population was compared with 169 healthy controls and additionally with 114 individuals affected by an allergy against para-substituted aryl compounds. The latter population was included in order to test whether possible associations were due to substance-specific effects, or were a general feature connected with type IV immunological diseases. Homozygous deletions of GSTT1 and GSTM1 were determined by polymerase chain reaction. Results: Glutathione S-transferase M1 deficiency was significantly more frequent among patients sensitized to thimerosal (65.9%, P = 0.013) compared with the healthy control group (49.1%) and the \"para-compound\" group (48%, P = 0.034). Glutathione S-transferase T1 deficiency in the thimerosal/mercury group (19.8%) was barely elevated versus healthy controls (16.0%) and the \"para-compound\" group (14.0%). The combined deletion (GSTT1-/GSTM1-) was markedly more frequent among thimerosal-sensitized patients than in healthy controls (17.6% vs. 6.5%, P = 0.0093) and in the \"para-compound\" group (17.6% vs. 6.1%; P = 0.014), revealing a synergistic effect of these enzyme deficiencies (healthy controls vs. thimerosal GSTM1 negative individuals, OR = 2.0 [CI = 1.2-3.4], GSTT1-, OR = 1.2 [CI = 0.70-2.1], GSTM1/T1-, OR = 3.1 [CI = 1.4-6.5]). Conclusions: Since the glutathione-dependent system was repeatedly shown to be involved in the metabolism of thimerosal decomposition products, the observed association may be of functional relevance."],["dc.identifier.doi","10.1007/s004200000159"],["dc.identifier.isi","000089088300005"],["dc.identifier.pmid","11007341"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/45516"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Springer"],["dc.relation.issn","0340-0131"],["dc.title","Homozygous gene deletions of the glutathione S-transferases M1, and T1 are associated with thimerosal sensitization"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","140"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Human Brain Mapping"],["dc.bibliographiccitation.lastpage","151"],["dc.bibliographiccitation.volume","35"],["dc.contributor.author","Esslinger, Christine"],["dc.contributor.author","Schueler, Nadja"],["dc.contributor.author","Sauer, Carina"],["dc.contributor.author","Gass, Dagmar"],["dc.contributor.author","Mier, Daniela"],["dc.contributor.author","Braun, Urs"],["dc.contributor.author","Ochs, Elisabeth"],["dc.contributor.author","Schulze, Thomas G."],["dc.contributor.author","Rietschel, Marcella"],["dc.contributor.author","Kirsch, Peter"],["dc.contributor.author","Meyer-Lindenberg, Andreas"],["dc.date.accessioned","2018-11-07T09:47:17Z"],["dc.date.available","2018-11-07T09:47:17Z"],["dc.date.issued","2014"],["dc.description.abstract","Neuronal plasticity is crucial for flexible interaction with a changing environment and its disruption is thought to contribute to psychiatric diseases like schizophrenia. High-frequency repetitive transcranial magnetic stimulation (rTMS) is a noninvasive tool to increase local excitability of neurons and induce short-time functional reorganization of cortical networks. While this has been shown for the motor system, little is known about the short-term plasticity of networks for executive cognition in humans. We examined 12 healthy control subjects in a crossover study with fMRI after real and sham 5 Hz rTMS to the right dorsolateral prefrontal cortex (DLPFC). During scanning, subjects performed an n-back working memory (WM) task and a flanker task engaging cognitive control. Reaction times during the n-back task were significantly shorter after rTMS than after sham stimulation. RTMS compared with sham stimulation caused no activation changes at the stimulation site (right DLPFC) itself, but significantly increased connectivity within the WM network during n-back and reduced activation in the anterior cingulate cortex during the flanker task. Reduced reaction times after real stimulation support an excitatory effect of high-frequency rTMS. Our findings identified plastic changes in prefrontally connected networks downstream of the stimulation site as the substrate of this behavioral effect. Using a multimodal fMRI-rTMS approach, we could demonstrate changes in cortical plasticity in humans during executive cognition. In further studies this approach could be used to study pharmacological, genetic and disease-related alterations. Hum Brain Mapp 35:140-151, 2014. (c) 2012 Wiley Periodicals, Inc."],["dc.description.sponsorship","Deutsche Forschungsgemeinschaft (DFG) [Sonderforschungsbereich SFB 636]"],["dc.identifier.doi","10.1002/hbm.22165"],["dc.identifier.isi","000327859900011"],["dc.identifier.pmid","22965696"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/35075"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Wiley-blackwell"],["dc.relation.issn","1097-0193"],["dc.relation.issn","1065-9471"],["dc.title","Induction and Quantification of Prefrontal Cortical Network Plasticity Using 5 Hz rTMS and fMRI"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","150"],["dc.bibliographiccitation.issue","1-3"],["dc.bibliographiccitation.journal","Toxicology"],["dc.bibliographiccitation.lastpage","151"],["dc.bibliographiccitation.volume","164"],["dc.contributor.author","Schulz, Thomas G."],["dc.contributor.author","Ruhnau, P."],["dc.contributor.author","Mueller, M."],["dc.contributor.author","Bunger, J."],["dc.contributor.author","Ellie-hausen, H. J."],["dc.contributor.author","Hallier, Ernst"],["dc.date.accessioned","2018-11-07T08:52:57Z"],["dc.date.available","2018-11-07T08:52:57Z"],["dc.date.issued","2001"],["dc.identifier.isi","000169888500479"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/22289"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Elsevier Sci Ireland Ltd"],["dc.publisher.place","Clare"],["dc.relation.issn","0300-483X"],["dc.title","Lack of correlation between CYP2A6 genotype and smoking habits"],["dc.type","conference_abstract"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","32"],["dc.bibliographiccitation.journal","Schizophrenia Research"],["dc.bibliographiccitation.lastpage","39"],["dc.bibliographiccitation.volume","209"],["dc.contributor.author","Kalman, Janos L."],["dc.contributor.author","Bresnahan, Michaeline"],["dc.contributor.author","Schulze, Thomas G."],["dc.contributor.author","Susser, Ezra"],["dc.date.accessioned","2020-12-10T15:21:11Z"],["dc.date.available","2020-12-10T15:21:11Z"],["dc.date.issued","2019"],["dc.identifier.doi","10.1016/j.schres.2019.05.012"],["dc.identifier.issn","0920-9964"],["dc.identifier.purl","https://resolver.sub.uni-goettingen.de/purl?gs-1/16429"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/72940"],["dc.language.iso","en"],["dc.notes.intern","DOI Import GROB-354"],["dc.notes.intern","Merged from goescholar"],["dc.rights","CC BY-NC-ND 4.0"],["dc.rights.uri","https://creativecommons.org/licenses/by-nc-nd/4.0"],["dc.title","Predictors of persisting psychotic like experiences in children and adolescents: A scoping review"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.version","published_version"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","340"],["dc.bibliographiccitation.issue","4"],["dc.bibliographiccitation.journal","METHODS OF INFORMATION IN MEDICINE"],["dc.bibliographiccitation.lastpage","350"],["dc.bibliographiccitation.volume","52"],["dc.contributor.author","Schwanke, J."],["dc.contributor.author","Rienhoff, Otto"],["dc.contributor.author","Schulze, Thomas G."],["dc.contributor.author","Nussbeck, S. Y."],["dc.date.accessioned","2018-11-07T09:29:44Z"],["dc.date.available","2018-11-07T09:29:44Z"],["dc.date.issued","2013"],["dc.description.abstract","Background: Longitudinal biomedical research projects study patients or participants over a course of time. No IT solution is known that can manage study participants, enhance quality of data, support re-contacting of participants, plan study visits, and keep track of informed consent procedures and recruitments that may be subject to change over time.. In business settings management of personal is one of the major aspects of customer relationship management systems (CRMS). Objectives: To evaluate whether CRMS are suitable IT solutions for study participant management in biomedical research. Methods: Three boards of experts in the field of biomedical research were consulted to get an insight into recent IT developments regarding study participant management systems (SPMS). Subsequently, a requirements analysis was performed with stake-holders of a major biomedical research project. The successive suitability evaluation was based on the comparison of the identified requirements with the features of six CRMS. Results: Independently of each other, the interviewed expert boards confirmed that there is no generic IT solution for the management of participants. Sixty-four requirements were identified and prioritized in a requirements analysis. The best CRMS was able to fulfill forty-two of these requirements. The non-fulfilled requirements demand an adaption of the CRMS, consuming time and resources, reducing the update compatibility, the system's suitability, and the security of the CRMS. Conclusions: A specific solution for the SPMS is favored instead of a generic and commercially-oriented CRMS. Therefore, the development of a small and specific SPMS solution was commenced and is currently on the way to completion."],["dc.identifier.doi","10.3414/ME12-02-0012"],["dc.identifier.isi","000323468700008"],["dc.identifier.pmid","23877579"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/31115"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Schattauer Gmbh-verlag Medizin Naturwissenschaften"],["dc.relation.issn","0026-1270"],["dc.title","Suitability of Customer Relationship Management Systems for the Management of Study Participants in Biomedical Research"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","4864"],["dc.bibliographiccitation.issue","32"],["dc.bibliographiccitation.journal","European Journal of Inorganic Chemistry"],["dc.bibliographiccitation.lastpage","4869"],["dc.contributor.author","Zhang, Z."],["dc.contributor.author","Roesky, Herbert W."],["dc.contributor.author","Schulz, Thomas G."],["dc.contributor.author","Stalke, Dietmar"],["dc.contributor.author","Doering, Alexander"],["dc.date.accessioned","2018-11-07T11:22:33Z"],["dc.date.available","2018-11-07T11:22:33Z"],["dc.date.issued","2009"],["dc.description.abstract","A unique (guanidinato)samarium dichloride cluster [(Me3Si)(2)-NC(NCy)(2)SmCl2](5)(thf)(2) was synthesized from anhydrous SmCl3 and (Me3Si)(2)NC(NCy)(2)Li in thf/toluene solution. This cluster exhibits a novel structure containing a chlorine atom with four coordinated samarium atoms. The X-ray crystal structure was investigated and compared with that of the heavier ytterbium congener [(Me3Si)(2)NC(NCy)(2)YbCl2](2-)(LiCl)(2)(thf)4, ((C) Wiley-VCH Verlag GmbH & Co. KGaA, 69451 Weinheim, Germany, 2009)"],["dc.description.sponsorship","Deutsche Forschungsgemeinschaft of the Schwerpunktprogramm [RO224/51]"],["dc.identifier.doi","10.1002/ejic.200900790"],["dc.identifier.isi","000272330800013"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/56018"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Wiley-blackwell"],["dc.relation.issn","1434-1948"],["dc.title","A Chlorine-Centered Cluster of Composition [(Me3Si)(2)NC(NCY)(2)SMCl2](5)(thf)(2) and a Comparison with the Heavier Ytterbium Congener [(Me3Si)(2)NC(NCy)(2)YbCl2](2)(LiCl)(2)(thf)(4)"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","65"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Biological Psychiatry"],["dc.bibliographiccitation.lastpage","71"],["dc.bibliographiccitation.volume","63"],["dc.contributor.author","Wobrock, Thomas"],["dc.contributor.author","Kamer, Thomas"],["dc.contributor.author","Roy, Anand"],["dc.contributor.author","Vogeley, Kai"],["dc.contributor.author","Schneider-Axmann, Thomas"],["dc.contributor.author","Wagner, Michael"],["dc.contributor.author","Maier, Wolfgang"],["dc.contributor.author","Rietschel, Marcella"],["dc.contributor.author","Schulze, Thomas G."],["dc.contributor.author","Scherk, Harald"],["dc.contributor.author","Schild, Hans H."],["dc.contributor.author","Block, Wolfgang"],["dc.contributor.author","Traeber, Frank"],["dc.contributor.author","Tepest, Ralf"],["dc.contributor.author","Honer, William G."],["dc.contributor.author","Falkai, Peter"],["dc.date.accessioned","2018-11-07T11:19:33Z"],["dc.date.available","2018-11-07T11:19:33Z"],["dc.date.issued","2008"],["dc.description.abstract","Background: The anterior limb of the internal capsule (ALIC), connecting cortical and subcortical structures, is involved in functional important circuits. To detect volumetric changes in ALIC, including the influence of genetic factors, a magnetic resonance imaging (MRI) study of families affected with schizophrenia was performed. Methods: The study sample comprised 22 family members with schizophrenia (FM-SZ), 34 family members without schizophrenia (FM-NSZ), and 43 control subjects. In addition to manual tracing of ALIC, subjects underwent proton magnetic resonance spectroscopy in the left prefrontal cortex, psychopathological rating, and neuropsychological assessment of frontal lobe function. Results: Compared with controls, a significant reduction of right ALIC volume was seen in all family members (12%-16% reduction, p <.01) and a reduction of left ALIC volume in FM-NSZ (10% reduction, p =.028) was also observed. Both groups of family members showed a bilateral reduction in maximal cross sectional area of the ALIC. FM-SZ performed significantly worse on neurocognitive measures (Subject Ordered Pointing Task [SOPT] and Wisconsin Card Sorting Test), and performance correlated negatively with the ALIC volume (SOPT, r = -.6, p =.03). Conclusions: A reduced volume of ALIC in affected families supports the hypothesis of disturbed frontothalamic connectivity in schizophrenia and demonstrates functional relevance by an association with reduced neurocognitive performance."],["dc.identifier.doi","10.1016/j.biopsych.2007.02.026"],["dc.identifier.isi","000251864000011"],["dc.identifier.pmid","17574215"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/55310"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Elsevier Science Inc"],["dc.relation.issn","0006-3223"],["dc.title","Reduction of the internal capsule in families affected with schizophrenia"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","951"],["dc.bibliographiccitation.issue","6"],["dc.bibliographiccitation.journal","Neuron"],["dc.bibliographiccitation.lastpage","963"],["dc.bibliographiccitation.volume","72"],["dc.contributor.author","Malhotra, Dheeraj"],["dc.contributor.author","McCarthy, Shane A."],["dc.contributor.author","Michaelson, Jacob J."],["dc.contributor.author","Vacic, Vladimir"],["dc.contributor.author","Burdick, Katherine E."],["dc.contributor.author","Yoon, Seungtai"],["dc.contributor.author","Cichon, Sven"],["dc.contributor.author","Corvin, Aiden"],["dc.contributor.author","Gary, Sydney"],["dc.contributor.author","Gershon, Elliot S."],["dc.contributor.author","Gill, Michael"],["dc.contributor.author","Karayiorgou, Maria"],["dc.contributor.author","Kelsoe, John R."],["dc.contributor.author","Krastoshevsky, Olga"],["dc.contributor.author","Krause, Verena"],["dc.contributor.author","Leibenluft, Ellen"],["dc.contributor.author","Levy, Deborah L."],["dc.contributor.author","Makarov, Vladimir"],["dc.contributor.author","Bhandari, Abhishek"],["dc.contributor.author","Malhotra, Anil K."],["dc.contributor.author","McMahon, Francis J."],["dc.contributor.author","Noethen, Markus M."],["dc.contributor.author","Potash, James B."],["dc.contributor.author","Rietschel, Marcella"],["dc.contributor.author","Schulze, Thomas G."],["dc.contributor.author","Sebat, Jonathan"],["dc.date.accessioned","2018-11-07T08:48:47Z"],["dc.date.available","2018-11-07T08:48:47Z"],["dc.date.issued","2011"],["dc.description.abstract","While it is known that rare copy-number variants (CNVs) contribute to risk for some neuropsychiatric disorders, the role of CNVs in bipolar disorder is unclear. Here, we reasoned that a contribution of CNVs to mood disorders might be most evident for de novo mutations. We performed a genome-wide analysis of de novo CNVs in a cohort of 788 trios. Diagnoses of offspring included bipolar disorder (n = 185), schizophrenia (n = 177), and healthy controls (n = 426). Frequencies of de novo CNVs were significantly higher in bipolar disorder as compared with controls (OR = 4.8 [1.4,16.0], p = 0.009). De novo CNVs were particularly enriched among cases with an age at onset younger than 18 (OR = 6.3 [1.7,22.6], p = 0.006). We also confirmed a significant enrichment of de novo CNVs in schizophrenia (OR = 5.0 [1.5,16.8], p = 0.007). Our results suggest that rare spontaneous mutations are an important contributor to risk for bipolar disorder and other major neuropsychiatric diseases."],["dc.identifier.doi","10.1016/j.neuron.2011.11.007"],["dc.identifier.isi","000298771000010"],["dc.identifier.pmid","22196331"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/21306"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Cell Press"],["dc.relation.issn","0896-6273"],["dc.title","High Frequencies of De Novo CNVs in Bipolar Disorder and Schizophrenia"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dspace.entity.type","Publication"]]

[["dc.bibliographiccitation.firstpage","4"],["dc.bibliographiccitation.issue","1"],["dc.bibliographiccitation.journal","Molecular Psychiatry"],["dc.bibliographiccitation.lastpage","9"],["dc.bibliographiccitation.volume","21"],["dc.contributor.author","Liu, C."],["dc.contributor.author","Saffen, D."],["dc.contributor.author","Schulze, Thomas G."],["dc.contributor.author","Burmeister, M. A."],["dc.contributor.author","Sham, Pak C."],["dc.contributor.author","Yao, Y-g"],["dc.contributor.author","Kuo, P-H"],["dc.contributor.author","Chen, C."],["dc.contributor.author","An, Y."],["dc.contributor.author","Dai, J."],["dc.contributor.author","Yue, W."],["dc.contributor.author","Li, M. X."],["dc.contributor.author","Xue, H."],["dc.contributor.author","Su, B."],["dc.contributor.author","Chen, L."],["dc.contributor.author","Shi, Y."],["dc.contributor.author","Qiao, M."],["dc.contributor.author","Liu, T."],["dc.contributor.author","Xia, K."],["dc.contributor.author","Chan, R. C. K."],["dc.date.accessioned","2018-11-07T10:21:42Z"],["dc.date.available","2018-11-07T10:21:42Z"],["dc.date.issued","2016"],["dc.identifier.doi","10.1038/mp.2015.95"],["dc.identifier.isi","000367096900003"],["dc.identifier.pmid","26481319"],["dc.identifier.uri","https://resolver.sub.uni-goettingen.de/purl?gro-2/42141"],["dc.notes.status","zu prüfen"],["dc.notes.submitter","Najko"],["dc.publisher","Nature Publishing Group"],["dc.relation.issn","1476-5578"],["dc.relation.issn","1359-4184"],["dc.title","Psychiatric genetics in China: achievements and challenges"],["dc.type","journal_article"],["dc.type.internalPublication","yes"],["dc.type.peerReviewed","yes"],["dc.type.status","published"],["dc.type.subtype","letter_note"],["dspace.entity.type","Publication"]]